The European Journal of Orthodontics

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1 The ejo.oxfordjournals.org 2013 Media Kit Advertising & Sales Contacts Steve Simmonds Head of Advertising Sales t: +44 (0) Caroline Bracken Supplements Development Manager t:+44 (0) e: ISSN (PRINT) For reprints, eprints or tailored products: e: An official journal of the European Orthodontic Society, making it the ideal place to reach orthodontists throughout Europe. One of the leading periodicals in its field, The publishes scientific papers aimed at all orthodontists. The journal provides a forum for orthodontists in Europe where many developments are taking place, but also accepts papers from all parts of the world. Useful Information Print Circulation: 3,700 Geographic Breakdown: UK 9% - Europe 75% - North America 3% - Rest of World 13% Average Monthly Page Views: 90,000 Average Monthly Unique IPs: 31,720 Average Available Ad Impressions: 155,230* *Combined monthly leaderboard and skyscraper positions No. Of etoc Subscribers: 1,229 Impact Factor: Target Audience: Orthodontists Frequency: 6 Peer Reviewed: Yes Editor-In-Chief: David Rice Society Affiliation: The European Orthodontic Society

2 2013 Schedule Volume Issue Cover Month Ad artwork due Mailout Date Bonus Conference Distribution 35/1 February 03 January February /2 April 05 March April 2013 EOS June Reykjavik 35/3 June 02 May June /4 August 04 July August /5 October 06 September October /6 December 06 November December 2013 Print Advertising Options & Rates Colour 1 insertion Special Position Premiums Full Page $ ½ Page $ ¼ Page $ Outside Back Cover - 15% extra Inside Front Cover - 15% extra Inside Back Cover - 10% extra Facing Leading Article 10% extra Facing Contents 10% extra Black & White 1 insertion 3 6 Full Page $ ½ Page $ ¼ Page $ Incentives Publisher s Discount 10% Agency Commission 10% Double Page Spread = 2 x Full page rate

3 ISSN (PRINT) ISSN (PRINT) The Advance Access published August 21, 2012 doi: /ejo/cjs045 The Author Published by Oxford University Press on behalf of the European Orthodontic Society. All rights reserved. For permissions, please Anne Marie Renkema*, Piotr S. Fudalej**, Alianne Renkema***, Ewald Bronkhorst**** and Christos Katsaros***** *Department of and Craniofacial Biology, Radboud University Nijmegen Medical Centre, The Netherlands, **Department of, Palacky University, Olomouc, Czech Republic, ***Department of, University Medical Centre Groningen, University of Groningen, ****Department of Community and Restorative Dentistry, Radboud University Nijmegen Medical Centre, The Netherlands and *****Department of and Dentofacial Orthopedics, University of Bern, Switzerland Correspondence to: P. S. Fudalej, Department of, Palacky University, Palackého 12, Olomouc, Czech Republic. Introduction A gingival recession (Figure 1a and 1b) is defined as the displacement of the marginal tissue apical to the cementoenamel junction (Camargo et al., 2001). Recessions are relatively common in Caucasian populations and their development is age-dependent they are more prevalent in older than in younger persons. Furthermore, they are more frequently observed in mandibular than in maxillary teeth. The gingival recessions negatively affect the appearance of dentition and may cause tooth hypersensitivity and lead to root caries (Löe et al., 1992; Susin et al., 2004). Orthodontic treatment may promote development of recessions (Bollen et al., 2008; Slutzkey and Levin, 2008). Slutzkey and Levin (2008) observed that the prevalence and extent of recessions correlated with past orthodontic treatment. For example, young adults (18 22 years old) who had been treated orthodontically many years before showed twice as high risk of developing gingival recessions than their untreated peers (22.9 versus 11.4 per cent, respectively). Also, Bollen et al. (2008) concluded in their review that the evidence suggested a small mean worsening of periodontal status after orthodontic therapy. The precise mechanism by which orthodontic treatment influences the occurrence of recessions remains unclear. Nonetheless, it has been assumed that the presence of bony dehiscence is a prerequisite for the development of gingival recession (Wennström, 1996). Because a bony dehiscence does not always lead to recession (Thilander et al., 1983), other factors such as thin gingival biotype, prolonged gingivitis, or mechanical trauma during tooth brushing must coincide (Wennström, 1996). From the orthodontic perspective, however, a possibility of formation of alveolar bone dehiscences during treatment and the presence of gingivitis during and after therapy is most important. Animal experiments with labial movement of lower incisors in monkeys (Batenhorst et al., 1974; Steiner et al., 1981) demonstrated the development of bone dehiscences and subsequent loss of periodontal attachment. More options and solutions in partnership with Loose and Bound Inserts available Gingival recessions and the change of inclination of mandibular incisors during orthodontic treatment SUMMARY A recent systematic review demonstrated that, overall, orthodontic treatment might result in a small worsening of periodontal status. The aim of this retrospective study was to test the hypothesis that a change of mandibular incisor inclination promotes development of labial gingival recessions. One hundred and seventy-nine subjects who met the following inclusion criteria were selected: age years at start of orthodontic treatment (T S ), bonded retainer placed immediately after treatment (T 0 ), dental casts and lateral cephalograms available pre-treatment (T S ), post-treatment (T 0 ), 2 years posttreatment (T 2 ), and 5 years post-treatment (T 5 ). Depending on the change of lower incisor inclination during treatment (ΔInc_Incl), the sample was divided into three groups: Retro (N 34; ΔInc_Incl 1 degree), Stable (N 22; ΔInc_Incl > 1 degree and 1 degree), and Pro (N 123; ΔInc_Incl > 1 degree). Clinical crown heights of mandibular incisors and the presence of gingival recessions in this region were assessed on plaster models. Fisher s exact tests, one-way analysis of variance, and regression models were used for analysis of inter-group differences. The mean increase of clinical crown heights (T 0 to T 5 ) of mandibular incisors ranged from 0.6 to 0.91 mm in the Retro, Stable, and Pro groups, respectively; the difference was not significant (P 0.534). At T 5, gingival recessions were present in 8.8, 4.5, and 16.3 per cent patients from the Retro, Stable, and Pro groups, respectively. The difference was not significant (P 0.265). The change of lower incisors inclination during treatment did not affect development of labial gingival recessions in this patient group. 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