Return to Basics. Normal Intervals & Axes. ECG Rate and Rhythm

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Return to Basics Management of the Hospitalized Patient October 15, 2010 ECG Refresher and Update 2010 Determine rate and rhythm Determine intervals and axes Define morphology of P-QRS-T-U Compare with normal values Integrate with clinical presentation into meaningful conclusion ECG Rate and Rhythm Normal rate for adults 60 to 100 bpm Speed Reading Normal, Slow, Fast Normal rhythm for adults Sinus with less than 10% variability of beat to beat Remember hiding premature atrial or ectopic beats can affect rhythm regularity Review p wave morphologies to determine if the rhythm is truly sinus Normal Intervals & Axes PR 0.12 to 0.20 sec QRS - < 0.10; 0.11 to 0.12 incomplete IVCD; abnormal > 0.12 sec QT c < 0.44 sec (QT c = QT/ RR) P axis: 0-60º QRS axis: -30 to 90º T axis generally follows the QRS axis except in the precordial leads where T waves should be upright from V2-6 1

Speed Reading Intervals & Axes PR < one large box QRS < 3 small boxes QT < 50% of RR interval (not reliable in AFib) Axes: P upright in I & II QRS positive in Leads I & II = normal LAD QRS isoelectric or negative in II RAD QRS isoelectric or negative in I T waves Same direction as major QRS vector in limb leads Upright in V2-6 Orderly Sequencing of Frontal Leads Cabrera Format 55 yo man awaken by chest pain Question #1 Which is the correct answer? 1. This ECG excludes an Acute MI 2. This ECG can be seen in STEMI 3. This ECG is a Right-Sided ECG 4. This ECG represents Arm Lead Reversal 2

Electrically silent MI s Right Ventricular MI Right sided ECG High Lateral MI V7, V8, V9 Small Apical MI 78 yo man with intracerebral hemorrhage 3

Einthoven s Triangle - ECG RA RL Cable Reversal (-) (+) RA-RL rev II Far Field Signal in II No structural abnormality can explain this finding The Technician s Test 55 yo man admitted for abdominal pain Sinus Arrhythmia 1.0 sec 0.8 sec 4

43 yo obese man admitted with chest pain. Sinus Rhythm Variations Normal Sinus Rhythm HR 60 100 bpm Sinus Bradycardia HR < 60 bpm Sinus Tachycardia HR > 100 bpm Sinus Arrhythmia >10% change from longest to shortest P P interval Irregular sinus rhythm (Sinus arrhythmia of the elderly) LVH LBBB PCard K+ AMI RB+AMI Brugada 5

STEMI - ACS If the patient s history and clinical presentation is consistent with ACS IT DOESN T MATTER WHAT THE ECG LOOKS LIKE! THE WORRISOME CLINICAL PRESENTATION OF CONCERN IS THIS PATIENT LOOKS LIKE!! ST Elevation Common Causes Acute myocardial injury pattern Acute pericarditis Early Repolarization / Normal Variant + 0.1mV in any lead Up to 0.3mV in young men in early precordial leads Myocardial aneurysm 6

Acute Pericarditis 45 yo woman with nausea and vomiting RBBB Which of the following is not true about RBBB? A. RBBB occurs in up to 29% of AMI B. The most common cause in children is post open heart surgery for Tetralogy of Fallot repair C. Post MI patients with persistent RBBB are at the same risk for cardiac mortality D. Transient RBBB is a complication of right heart catheterization Differential Dx of Tall R wave in V1 Definition R/S ratio > 1 in V1 or V2 RBBB RVH True Posterior MI WPW Brugada s Abnormality 7

65 yo Asian man who presents with recurrent syncope. Echo was normal. Brugada Syndrome Brugada Abnormality is defined as RBBB with ST elevation and occurrence of sudden cardiac death or syncope due to polymorphous VT (1990) Hereditary bundle branch defect Autosomal dominant trait with variable expression Diagnosis by EPS with procainamide stimulation TX: AICD 36 yo woman presenting to the ED with acute dyspnea. Brugada Pattern Patterns of ST segment elevation in brugada syndrome RVH 8

79 yo man admitted with CVA Diffuse ( Global ) T Wave Inversion Myocardial Ischemia (Evolving MI is focal) CNS event Apical HCM Pericarditis Myocarditis Takatsubo s Cardiomyopathy Cardiac metastases Carotid endarterectomy Cocaine abuse Pheochromocytoma Acute illness in women 84 yo woman with pneumonia Which of the following answers about LBBB is not true? A. LBBB can be caused by toxic, inflammatory changes, hyperkalemia, or digitalis toxicity B. In most patients with LBBB, septal wall motion abnormalities can exist without coronary artery disease C. In patients presenting to the ED with chest pain and a LBBB, the management is observe with frequent ECGs, treat pain and await enzymes D. The QRS duration is at least 0.12 seconds 9

32 yo woman from Viet Nam presents with dyspnea on exertion. What is the diagnosis? The most likely diagnosis is: Biatrial Abnormality A. Primary Pulmonary Hypertension B. Atrial Septal Defect C. Ventricular Septal Defect D. Mitral Stenosis E. Aortic Stenosis Biatrial Abnormality Non Conducted PAC s o A large diphasic P in V 1 with the positive component > 1.5 mm and the terminal negative component reaching 1mm in amplitude, > 0.04 sec in duration or both o Tall peaked P wave (>1.5mm) in the right precordial lead and a wide notched P wave in the limb leads or lateral leads (V 5-6 ) o Increase in both the amplitude (>2.5mm) and duration(>0.12 sec) of the P wave in the limb leads 10

56 yo man presents with flu This ECG represents: Rate-Related RBBB 1. Acute Myocardial Infarction with alternating bundles 2. Sinus rhythm with PVC s 3. Ashman s Phenomenon 4. He s got more going on than a flu 5. A cardiac emergency requiring pacemaker implantation 70 yo woman admitted for weakness 24 yo woman with FHX of SCD presents with syncope Giant U waves Prolonged QTc = 498 msec 11

78 yo man with hx of HF presents with nausea Afib with AV Dissociation What is the most appropriate next test? 1. Serial Troponins 2. Serial CK with iso s 3. Digoxin level 4. Serum potassium level 5. I don t know AFib with AV Dissociation AFib or AFib/Aflutter or Aflutter? 58 58 58 58 58 58 58 58 58 AFib 12

What is the rhythm? What is the treatment? What is the rhythm? What is the treatment? p-p 640 640 640 2 nd AVB Mobitz 1 200 240 200 pr 240 2 nd AVB Mobitz 1 What is the rhythm? What is the treatment? 2 nd AVB Mobitz2 AVNRT 13

Supraventricular Tachycardias: Which statement is true? Slow-fast form of AVNRT 1. If hemodynamically stable, acute management includes Adenosine by slow IV push 2. The majority of SVT is AVNRT 3. AVRT does not involve accessory bypass tracts 4. If the patient after adenosine converts from a narrow complex tachycardia to a wide complex tachycardia, the patient has most likely degenerated to ventricular tachycardia Generation of ECG in Common form of AVNRT Narrow QRS Tachy response to Adenosine 14

Rx of Hemodynamically stable tachycardia 34 yo woman with idiopathic cardiomyopathy became dizzy and came to the ED. Wide QRS-complex tachycardia (QRS > 120 ms) Regular or irregular? Wide QRS-complex tachycardia (QRS > 120 ms) Regular Is QRS identical to that during SR? If yes, consider: Vagal maneuvers SVT and BBB or Antidromic AVRT Adenosine Previous MI or structural heart disease? If yes, VT is likely 1:1 AV relationship? Irregular Atrial Fibrillation Atrial Flutter/AT with variable conduction and a) BBB or b) Antegrade conduction via AP Regular Previous MI or structural heart disease? If yes, VT is likely 1:1 AV relationship? Yes or unknown QRS morphology in precordial leads Yes or unknown QRS morphology in precordial leads Next slide V rate faster than A rate VT No A rate faster than V rate Atrial Tachycardia Typical RBBB or LBBB = SVT Precordial leads VT Concordant No R/S pattern Onset of R to nadir > 100 ms RBBB pattern VT qr, Rs or Rr in V1 Frontal plane axis range from +90 degrees to 90 degrees LBBB pattern VT R in V1 > 30 ms R to nadir of S in V1 > 60 ms qr or qs in V6 Atrial Flutter Wide QRS-complex tachycardia I Wide QRS-complex tachycardia II 15

A fib with WPW 82 yo man presents with chronic fatigue. Why is he on your service? CHB The correct diagnosis is: 82 yo man presents with chronic fatigue. Why is he on your service? 1. Acute anterior myocardial infarction 2. Congestive Heart Failure 3. Sick Sinus Syndrome 4. Complete Heart Block with an artifact 5. Wandering Atrial Pacemaker CHB with Answer Artifact 16

75 yo AA male presents with prolonged CP Prognostic Value of Lead avr in Patients with a First Non-ST segment Elevation Acute Myocardial Infarction nonstemi & avr 70.00% 60.00% 50.00% 40.00% 30.00% 20.00% In-house Mort LM/3v CAD 10.00% 0.00% without 0.05-0.1 mv > 0.1 mv Odds ratio for death in groups 2 and 3 4.2 and 6.6 Barrabes, JA et al. Circ 2003; 108: 814-819 First Degree AV Block TriFasicular Block Generally benign Bad prognostic sign in: Bifascicular Block RBBB + LAFB RBBB + LPFB LBBB Infectious endocarditis 17

Q: Had this patient suffered a myocardial infarction? 1. No! 2. Yes. Inferior 3. Yes. Posterior 4. Yes. Septal 5. Yes. Localization not possible due to RBBB 75 yo man presents with palpitations Blocked PAC s 18

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