UNIVERSITY HOSPITALS OF LEICESTER NHS TRUST CARDIAC INVESTIGATIONS PAEDIATRIC & CONGENITAL ELECTROCARDIOGRAPHY Guideline

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UNIVERSITY HOSPITALS OF LEICESTER NHS TRUST CARDIAC INVESTIGATIONS PAEDIATRIC & CONGENITAL ELECTROCARDIOGRAPHY Guideline Authors: Suhair Shebani, Claire Sansome Contact Name: Deborah Ip, Carla Blunt Approved by: CPG 18.5.2016 Last Reviewed Date: 13/6/16 Next Review Date: 13/6/19 I. PAEDIATRIC ELECTROCARDIOGRAPHY PROTOCOL (patients under the age of 16 years) Claire Sansome Patient Approach Note the patient s name, consultant, date of birth and hospital unit number before commencing the test. Check with the parents/guardians/patient whether they are correct. Using the correct information available, input information into ECG machine along with the date and if they are an inpatient or an outpatient. Explain the ECG procedure to the parents/guardians ( a record of the heart beat which is painless ), and where possible, explain the ECG to the child in simple terms (depending on the age of the child). Make sure the patient is as comfortable as possible. For children under the age of 4/5 years depending on how wriggly they are, flat is a desirable position, but obviously if the child is too wriggly or uncooperative, sitting the child on mum/dads knee is acceptable. Where appropriate, chat to the child and use distraction toys to keep the child interested and relaxed and hopefully as still as possible. Attach the electrodes to the limb and chest positions as follows:

Arm electrodes should be placed on the upper or lower arms, and SHOULD NOT be applied to the shoulders or clavicles. For total accuracy, arm electrodes should be attached to the wrists and leg electrodes to the ankles. However, this may not be possible if the child is wriggly and/or unco-operative in which case upper and lower limbs are satisfactory. Application of electrodes no skin preparation is usually required for paediatric patients. However, the contact of electrodes may be reduced on newborn babies in which case very gentle wiping of the electrode area with a Mediswab for example, may help the contact. In babies/small children, the standard self-adhesive electrodes may be too large in which case use paediatric electrodes. Chest Electrode Positions V1 (red electrode) to be placed in V4R position V2 (yellow electrode) to be placed in V1 position V3 (green electrode) to be placed in V2 position. No standard V3 to be taken V4, V5 and V6 as normal Once electrodes are in place, attach the ECG leads and ensure the cable is in a secure position. When ready to record the ECG, if possible encourage the child to keep still whilst recording. If your patient is to wriggley, use distraction techniques with toys to keep the child occupied while recording. A standard 12 lead ECG is recorded with a lead II rhythm strip. For children with supraventricluar tachycardia of atrial fibrillation/flutter, then a V1 rhythm strip is recorded. Once complete remove electrodes and check that the recording is labelled correctly. Ensure ECG is shown to medical staff.

Chest Positions For Patients With Dextrocardia Follow the ECG protocol as usual recording with normal chest positions. LIMB LEADS REMAIN THE SAME. Then repeat the ECG using the following positions on the right side of the chest. V1R- fourth intercostal rib space to the left sternal edge V2R fourth intercostal rib space to the right sternal edge V3R (over age of 16 years) midway between V2R and V4R V4R fifth intercostal space on the midclavicular line V5R at the same level as V4R on the anterior axillary line. V6R at the same level as V4R and V5R in the mid-axillary line. II. Paediatric Electrocardiography (ECG), Background information; Suhair Shebani Indications: assessment of every cardiac patient and in paediatric practice on suspicion of a structural cardiac, rhythm problem The Standard ECG Electrode positions See figure 1 Standard (I,II,III) and unipolar leads (avr, avf, avl) same as adults Precordial leads V1-V6, with V4R up to 12 years In dextrocardia V6R to V1 A rhythm strip with 3 leads or lead II if assessing rhythm disturbances If leads are very abnormal consider lead mal-placement Recording Speed 25mm /sec ( 1small square = 0.04sec) At full standardisation amplitude of 10mm= 1mV Recording at 50mm/sec helps in interval measurement

Deflections and Intervals: figure2

Order of ECG interpretation II. Rate and rhythm III. AV-conduction IV. Atrial de/repolarization V. Ventricular depolarization VI. Ventricular repolarization VII. Hypertrophy, enlargement Make sure the ECG corresponds to the patient in question with date on and patient DOB. I. Rate and Rhythm Heart Rate: Influenced by body temp, autonomic nervous system, activity, age. HR=300/No of large squares each 0.2 sec or 1500/No of small squares each 0.04 sec Normal age variations Premature and LBW: 77-211 bpm 3-33% of neonates have APCs or VPCs FT neonate: min 55-75 to 220 bpm Older children min 44-68, max 132-196, 15-40% APCs, VPCs 3% non-sustained VT Fit adolescents especially athletes 31-55 bpm Sinus pauses <3sec, 1st degree and 2nd degree type 1 heart block can be a normal variation. Rhythm Regular, irregular, or regular with intermittent predictable irregularity Relationship of P waves to the QRS complexes Sinus P wave = atrial depolarisation from top to bottom and from right to left +ve in I, II, avf, -ve in avr Reversal of P wave vector = situs inversus. II. A-V Conduction 1st degree Heart Block = prolonged PR interval 2nd degree Heart Block Type 1 Type 2 3rd degree Heart Block = CHB No conduction from A to V = no relation between P and QRS complexes

III. Atrial depolarization The P wave Initial RA, terminal LA components Duration: infancy: 0.04-0.07 s, adolescence: 0.06-0.1 s Tallest in leads II, V4R, V1 upper limit 2.5 mm Abnormal pattern: anomalous pacemaker Atrial hypertrophy RAH: spiked P-wave > 2.5 mm in II, V4R, V1. severe PS, PA, TA, PHT LAH : notched P-wave or large ve component in V4R, V1 MV disease, LVOTO, 1ry LV cardiomyopathy IV. Ventricular depolarization The QRS complex Duration: 0-8y <80 msec = 2 small squares >8 y < 90 msec Duration prolonged Rt or Lt BBB Ventricular pre-excitation Ventricular pace-maker Ventricular hypertrophy Intraventricular conduction delay: CMP, ARVD Q wave R and S Ventricular depolarisation: variation of R/S voltages with age Table 2.1: Normal voltages in mv in precordial leads: V 4 R V 1 V 3 V 4 R R S R R S Birth 8 (4-12) 6 months 5 (2-7) 1 year 4 (0-7) 10 years 2.5 (0-6) 12 (5-20) 11 (3-17) 9 (2-16) 5 (1-12) 10 (0-20) 10 (1-25) 10 (1-12) 10 (1-25) 9 (2-20) 20 (10-28) 20 (5-30) 20 (5-40) 5 (1-13) 14 (5-25) 14 (5-25) 16 (5-30) 6 (0-15) 3 (0-10) 3 (0-7) 2 (0-5) Q wave = depolarization of IVS from L to R Leads: II, III, avf, V5, V6

Measures 2-3 mm >4 mm is abnormal, duration <40 msec Prominent Q waves in LVH, BVH Q in other leads is rare; seen in HOCM, ALCAPA, CCTGA In CCTGA Q is seen in V4R, V1 and absent from V5,V6 R and S: size is determined by ventricular wall thickness, it varies with age QRS axis = Direction of maximal electrical force during depolarization Method 1: using leads I,II Plot R-S Drop perpendiculars from those axes Line from origin through intersection is the electrical axis Method 2: using leads I, avf Result of R-S in avf should be X 1.3 Method 3 Choose any limb lead with R=S Electrical axis will be at right angles to the axis of that lead QRS axis = Direction of maximal electrical force during depolarization Method 1: using leads I,II Plot R-S Drop perpendiculars from those axes Line from origin through intersection is the electrical axis Method 2: using leads I, avf Result of R-S in avf should be X 1.3 Method 3 Choose any limb lead with R=S Electrical axis will be at right angles to the axis of that lead Calculation of QRS

V. Ventricular Repolarization ST-T- wave abnormalities ST segment elevation > 0.1 mv, depression <-0.05 mv Affected by Electrolyte disturbance, myocarditis, pericarditis, MI, drugs QT interval: from start of QRS to end of T-wave HR and age dependent QTc = QT measured/sqrrt of previous RR interval in lead II (Bazett s formula) 0-6mo <0.490 sec, >6mo <0.425 sec QT: Familial: Romano Ward, Jervell &Lange Neilsen syndromes and electrolytes, drugs, metabolic causes. T wave Should measure ¼ -1/3 of R-wave Upright in V4R, V1 +/- V2, V3 then inverted after 3rd day of life Remains inverted in Rt chest leads as we approach adolescence it becomes upright in V2, V3 (5-15y) Normally inverted in avr, +/- avf, III T wave is always inverted in V1 unless there is a pathology Inverted T in other leads: ventricular hypertrophy, myocardial disease, pericarditis and severe hypothyroidism U wave VI. Ventricular hypertrophy Criteria for LVH 1. Mild LVH: Sum of S in V1 + R in V5 or V6 > 30mm under 1 yr or > 40 mm > 1 yr 2. Moderate LVH (1) + prolonged QRS or flat T-wave in V5, V6

3. Severe LVH (1) + (2) plus T-wave inversion in V5, V6 Examples: Aortic valve disease, moderate size VSD, large PDA MR, systemic hypertension, congestive and obstructive Cardiomyopathy Criteria for RVH 1. Mild RVH: RAD, R above normal limits in V4R, V1 R>S in V1 after age of 12 mo S in V6 > normal of age: 15mm in 1st week, 10 mm 1-24 wk, 7 mm 6-12 mo, 5 mm >1yr 2. Moderate RVH: (1) + prolonged QRS and upright T wave in V4R, V1 after 3 days of age 3. Severe RVH: tall R waves in V4R, V1 QRS>90 msec and deep T-wave inversion in V4R, V1 Examples PS, PHT, Fallot D/D RBBB, WPW Biventricular hypertrophy 1. Tall R and deep S in V3, V4 ( R+S > 50 mm at any age) 2. LVH + wide or bifid R in V4R, V1 > 8 mm 3. RVH + T wave Inversion V5-6 ( T upright in V1-2) or q wave 3 mm in V5-6 4. Criteria of RVH, LVH Example: 3 year old with large VSD and dynamic pulmonary hypertension R+S in V4 =70mV Specific Electrocardiographic Diagnosis P-R interval : Ebstein s AVSD CHB: Congenital, CCTGA, post op for AVSD, TOF RBBB: ASD, Ebsteins post ventriculotomy: VSD, TOF LAD:0 to -90 in pink baby : AVSD, in blue baby: TA. -90 to 180: CAVSD and DORV

RAH with absence of RV dominance in a cyanosed baby = Hypoplastic RV QRS axis 0 to +90: PA, intact septum 0 to 90 TA Cyanosed baby with PHT but no RVH: HLHS Deep Q consider HOCM or ALCAPA Presence of q in V1, V4R and absence in V5,6 = CCTGA Deep T wave inversion in V5-6 with no murmur: Cardiomyopathy or EFE