MYOFASCIAL PAIN. Dr. Janet Travell ( ) credited with bringing MTrPs to the attention of healthcare providers.

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Myofascial Trigger Points background info Laurie Edge-Hughes BScPT, MAnimSt (Animal Physio), CAFCI, CCRT History lesson Dr. Janet Travell (1901 1997) credited with bringing MTrPs to the attention of healthcare providers. Cardiologist & Medical researcher Interest in muscle pain after reading several article on referred pain Reports of myofascial pain go back as far as the 16 th century however! 1

History lesson Dr. Janet Travell 1940 s developed and published injection techniques of MTrPs 1952 described the myofascial genesis of pain with detailed referred pain patters for 32 muscles (Note other clinicians had work that paralleled in describing the characteristics of MTrPs & effective manual therapies) History lesson Dr. Janet Travell 1966 Travell + Dr. John Mennell founded the North American Academy of Manipulative Medicine. She often promoted integrating myofascial treatment with articular treatments 1960 s collaboration with Dr. David Simons, which led to the Trigger Point Manuals (1983 & 1992 1 st eds) 2

What is myofascial pain? What are trigger points? Myofascial Pain Syndrome A a regional pain syndrome characterized by muscle pain caused by MTrPs. Myofascial Trigger Point A tender point located in the endplate zone, and characterized by a taut band of muscle, referred pain, & a local twitch response. 3

Sensory Component: Local pain, referred pain, and local twitch response when the locus is mechanically stimulate with pressure Motor Component: Spontaneous electrical activity & Endplate noise Physical Component TOGETHER = the taut band Myofascial Trigger Point Region Thoughts: Endplate Noise is the result of excessive ACh leakage. Combined with sarcomere shortening & release of sensitizing substances can lead to an energy crisis = local ischemia & hypoxia 4

Myofascial Trigger Point region Etiology: Low-level continual muscle contraction (i.e. poor posture) Direct trauma Unaccustomed eccentric contractions Over-use of unconditioned muscles Arthritis / pain in an adjacent joint Stress / Pain Spinal DJD & nerve root compression Spinal Cord Mechanisms A strong noxious stimulus can send an impose to the corresponding dorsal horn neurons causing release of substance P & calcitonin gene-related peptide. Which increases pain signaling of other receptors supplied by the same dorsal horn neurons = Central Sensitization 5

Central Sensitization: Increased responsiveness to nociceptive neurons in the central nervous system to their normal or sub-threshold afferent input Altered Nerve Conduction (NR compression) Decreased sympathetic outflow to muscles Motor Signs Increase in ACTH receptors in a zone of degeneration & supersensitivity Alignment & Postural Affects (banding or tightening) Muscle tissue changes Hypertonicity, Muscle fasciculation, Tight bands of muscle, Trigger points ROM deficits (due to tight bands / shortened muscles) Altered Reflexes BRISK (not necessarily hyper-reflexic) 6

Altered Nerve Conduction (NR compression) Autonomic Nervous System Changes Vasomotor (cold & clammy skin) Sudomotor (increased sweating) Cutaneous circulatory changes Pilomotor effect (goosebumps along dermatomes) Tropic changes Skin dystrophy & edema Skin rolls, denting, peau d orange Match stick sign, reduced skin rolling Increased skin creases, Dermatomal hair loss Neural Mechanosensitization (Peripheral origin) Peripheral neuritis occurs with minimal peripheral nerve injury, with no axonal loss or changes in nerve conduction Nerve sheath inflammation can cause pain behaviours, hyperalgesia, and allodynia on sensory testing The lesion site shows an increase in mechanosensitization of Aβ fibres, C-fibres, and deep nociceptor axons. = PERIPHERAL Sensitization 7

Peripheral Sensitization: Increased responsiveness and reduced threshold of nociceptors to stimulation of their receptive fields Neural Mechanosensitization (Peripheral origin) The inflammation and resultant mechanosensitization may cause spontaneous firing of the Aβ fibres & C-fibres Subsequently, can result in spontaneous firing from the associated dorsal root ganglia Spontaneous firing triggers a cascade of events in the CNS that can lead to chronic pain (and Central Sensitization ) 8

Diagnosis Myofascial Trigger Points Diagnosis Manual Palpation & Clinical Judgment Spot tenderness, taut band, pain recognition Confirmed by referred pain and local twitch response 9

Myofascial Trigger Points Diagnosis Interrater reliability? Most reliable was referred pain sensation & Jump sign on testing Least reliable were finding a nodule in a taut band, and eliciting a local twitch response Where to look next in research? Biochemical measurements Sonography MRI EMG Myofascial Trigger points clinically Test on yourself Trapezius Extensor muscles of your forearm Lateral or medial head of gastrocs Lateral side of your thigh 10

Myofascial Trigger points clinically Test on someone else Trapezius Infraspinatus 11

Myofascial Trigger points clinically Next Time Palpation on a dog & Dry needling on a dog See ya next week! 12