Module II
MEDICAL DISCLAIMER The information in this program is for educational purposes only. It is meant to as a guide towards health and does not replace the evaluation by and advice of a qualified licensed health care professional. For detailed interpretation of your health and specific conditions, consult with your physician.
OUTLINE & NOTES, MODULE II / page 1 COURSE OUTLINE - Module I I. Introduction & Summary of Module I II. III. IV. What to Do to Naturally Increase Bone Density and Have a Bone Friendly Lifestyle Sitting less Exercising regularly Avoiding bone damaging foods Having an appropriate acid/alkaline balance Taking supplements Balancing your hormones Getting tested Supplements Minerals: Calcium, Magnesium, Phosphorous Trace minerals: Boron, Manganese, Silicon, Strontium Vitamin D3 (Cholacalciferol) Vitamin K1 (Phytonadione) Herbs Nettles Horsetail Others? V. Diet Greens Less meat VI. Exercise
page 2 / THE COMPLETE BONE HEALTH SOLUTION COURSE NOTES - Module I INTRODUCTION Osteoporosis is a complex degenerative disease of the bone and bone forming mechanisms. Modern lifestyle and nutritional deficiencies, stress factors, and hormone imbalances play roles in cause osteoporosis. Healthy bones are resilient, hard to break, and last a lifetime. However, a lot can go wrong with you bones resulting in a vast array of diseases. The primary causes of bone diseases are nutrient deficiencies and the modern Western diet. Sedentary lifestyle also plays a major role. However, osteoporosis is not simply a nutrient deficiency disease. It is an imbalance of bone homeostasis that involves bone remodeling, immunological factors, and hormones. To restore bone health, all aspects of the condition have to be addressed where new bone replaces old bone and microdamage repair mechanisms are restored. DIETARY CONSIDERATIONS AVOID THESE NEGATIVE DIETARY INFLUENCES: Acidic Diet High Salt Excessive caffeine (more than 3 cups of coffee or strong tea daily) Cola drinks (high caffeine, sugar, and phosphorous) Excessive alcohol drinking Dietary deficiencies associated with the modern Western diet low in trace minerals and key vitamins Low calcium Low magnesium, high phosphorous High oxalate (oxalic acid) foods (spinach, beet greens, chard, tea, and coffee) High phytates (phytic acid) foods (Brazil nuts, wheat bran, soy, and other dried legumes) CALCIUM Osteoporosis is influenced by diet, especially calcium intake. Dairy products are a good source of calcium, but plant calcium may also be important in populations that do not consume a large amount of milk. High dietary intake of calcium, especially plant calcium, reduces the risk of osteoporosis and increased bone mineral density. Vegetables are rich in antioxidants and may be an important source of calcium, and provide vitamins and minerals that exert beneficial effects on the bone. There are several plant sources of calcium: Collard greens Soy products
OUTLINE & NOTES, MODULE II / page 3 (CALCIUM SOURCES, cont...) Almonds Kale Okra Mustard greens Bak Choy Broccoli Sesame Seeds Dried Legumes LOW RED MEAT, HIGH FISH DIET Seafood, especially fish rich in calcium like sardines, has a positive impact on BMD. FRUIT he high plant-based diet that includes vegetables and fruits is associated with stronger BMD and lower incidence of osteoporotic fractures. However, high glucose is a negative influence on bone health, so select from a variety of seasonal, locally grown organic fruits. Avoid fruit juice, as they are high in fructose. Don t over consume fruits. FOUNDATIONAL SUPPLEMENTS NUTRIENT Calcium Citrate Magnesium Citrate Manganese Potassium Bicarbonate Vitamin D3 Vitamin K1 Boron DAILY DOSAGE 500-750 mg (1,000 mg) 250-750 mg (1,000 mg) 5-10 mg 100 upwards to 200-300 mg 800-2,000 IU (5,000-10,000 IU) 1-10 mg 3-9 mg ADDITIONAL CONSIDERATIONS Probiotics Antioxidants (vitamins C and E) B vitamins
page 4 / THE COMPLETE BONE HEALTH SOLUTION CELIAC DISEASE, FOOD SENSITIVITIES & GUT INFLAMMATION Get tested Avoid allergenic foods Improve GI health CUTTING EDGE NUTRACEUTICALS LYCOPENE Lycopene is an antioxidant nutrient from tomato that is associated with a protective effect on bone, resulting in decreased bone turnover markers, thus reducing the risk of osteoporosis in postmenopausal women. BONE MORPHOGENETIC PROTEINS (BMPS) BMPs are a family of proteins that stimulates the cells responsible for growing bone (osteoblasts) and cartilage (chondrocytes). Ostinol Ostinol is a bone derived protein/collagen complex, which contains: BMPs TGF-Beta (Transforming Growth Factor Beta) PDGF (Platelet Derived Growth Factor) IGF (Insulin-like Growth Factor) bfgf (Basic Fibroblastic Growth Factor) Ostinol bone and cartilage stimulating proteins: Naturally activate bone & cartilage growth Are functionally involved in bone & cartilage formation Help maintain natural bone & joint homeostasis SOY ISOFLAVONES Impressive data from the many studies on cultured bone cells and rat models of postmenopausal osteoporosis support a significant bone-sparing effect of the soy isoflavones genistein and daidzein. Collective data suggest that diets rich in phytoestrogens have bone-sparing effects in the long term. Ipriflavone Ipriflavone is a synthetic isoflavone which may be used to inhibit bone resorption,[1] maintain bone density and to
OUTLINE & NOTES, MODULE II / page 5 prevent osteoporosis in postmenopausal women. It is not used to treat osteoporosis. It slows down the action of the osteoclasts (bone-eroding cells), possibly allowing the osteoblasts (bone-building cells) to build up bone mass. Studies in 1999 found ipriflavone effective in preventing bone loss associated with chronic steroid use, immobility, ovariectomy, renal osteodystro- phy, and gonadotrophin hormone-releasing hormone agonists. In addition, it holds prom- ise for the treatment of other metabolic diseases affecting the bones, including Paget s disease of the bone, hyperparathyroidism, and tinnitus caused by otosclerosis. A MEDSCAPE review by Jean-Yves Reginster, MD, PhD, reports that preliminary studies in 2001, mainly performed in Italy and Japan, suggested that ipriflavone (typical dosage 600 mg/day) is able to prevent bone loss, and some data even suggested that ipriflavone may increase bone mass in postmenopausal women. However, lymphocytopenia occurred in a significant number of women treated with ipriflavone. Recent studies suggest that there is sufficient evidence suggesting soy isoflavones may have potential benefits for bone. Soy protein with naturally occurring phytoestrogens, mainly isoflavones protect against bone loss and synthetic soy ipriflavone in some studies has been shown to favorably affect, but a cause and effect relationship has not been established between the consumption of ipriflavone and maintenance of bone mineral density in postmenopausal women. NUTRACEUTICAL Ipriflavone Lycopene Ostinol Soy Isoflavones DAILY DOSAGE 600-900 mg 10-25 mg 150-300 mg 120-200 mg HORMONES ESTROGEN Estrogen is the key hormone governing bone homeostasis. Estradiol is used to prevent osteoporosis in women after menopause. It may also be used to slow bone loss in women who have osteoporosis. TESTOSTERONE AND DHEA These are androgens (male hormones) that support bone health. Androgen-deficient men are at increased risk of osteoporosis. Testosterone low-dose replacement has been shown to increase BMD. HGH (HUMAN GROWTH HORMONE) HGH treatment shows increases in bone turnover, bone size, bone mineral density (BMD), and improved bone mineral content.