Degenerative arthritis of Hip Bone Bangalore. Prof Sharath Rao Head, Dept. of Orthopaedics KMC Manipal

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Transcription:

Degenerative arthritis of Hip Prof Sharath Rao Head, Dept. of Orthopaedics KMC Manipal

Hip joint Classical Synovial joint

Biomechanics of hip Force coincides with trabecular pattern

Hip joint

Acetabulum

Hip joint Acetabulum

Acetabulum

Hip joint Acetabulum

Neck shaft angle

Neck shaft angle

Neck shaft angle

Neck shaft angle

Neck shaft angle

Neck shaft angle

Neck shaft angle

Anteversion of neck

Anteversion of neck

Anteversion of neck

Anteversion of neck

Definition Chronic joint disorder in which there is progressive softening and disintegration of articular cartilage and bone at the joint margin (osteophytes) and capsular fibrosis

Etiology Increase frequency with age Reduced proteoglycan Loss of elasticity Decrease in breaking strength Cartilage matrix changes Crystal deposition disease Ochronosis Genetic defect in type II collagen

Etiology Inflammatory disorders Enzymatic leaching of proteoglycans Il-i mediated suppression in synthesis Increased mechanical stress Increased load Reduced articular contact area Idiopathic

Types Primary osteoarthrosis No obvious antecedent cause Secondary osteoarthrosis Following a demonstrable abnormality

Pathogeneisis Dysplasia instability spectrum Femoro acetabular impingement Both

Pathogeneisis Structural damage to Articular cartilage Chondro labral complex

Pathogeneisis Actabular dysplasia Femoral head is inadequately covered Instability Shearing forces on the edge of acetabulum & head

Femoro acetabular impingement - FAI Cam type Pincer type Mixed

Cam type of impingement

Cam type of impingement

Pincer type of impingement

pincer type of impingement

Cam & pincer type of impingement

Pathogenesis Progressive cartilage destruction Subarticular cyst formation Sclerosis of surrounding bone Osteophyte formation Capsular fibrosis

Prevalence Commonest of all joint diseases Equal in both sexes Hip is second most involved in Indian population. Opposite in the European countries

Risk factors Joint dysplasia Trauma Occupation Bone density Obesity Family history

Causes ABNORMAL STRESS SUBLUXATION COXA MAGNA COXA VARA MINOR DEFORMITY PROTRUSIO DEFECTIVE CARTILAGE INFECTION RHEUMATOID CALCINOSIS ABNORMAL BONE FRACTURE NECROSIS PAGETS OTHERS

Clinical features Pain Groin Radiation to knee Stiffness Limp Increased after rest Early manifestation Shortening

Pain Pain worsening after activity Trochanteric pain abd muscle fatigue Pain on prolonged sitting & groin pain FAI

Clinical features Passive hip extension, adduction & external rotation Hip 90 degrees flexion adduction & intenal rotation

Clinical features Hip is flexed, Abducted & externally rotated and then extended and internally rotated There is a snap

Clinical features Look Feel Shortening Scars Sinuses Temp Joint line tenderness Move Deformity

Deformity Primary osteoarthrosis Flexion Abduction External rotation Secondary osteoarhtrosis Depending on the condition

Investigation Radiograph Pelvis with both hips AP Frog or translateral Special veiws

Investigation Reading of x-ray A. Alingnment B. Bone quality C. Cartilage / joint space

Investigation Complete blood work Bone scan Ct scan MRI

Treatment Early Maintain movement Muscle strenghtening Protect joint from overload Pain relief Activity modification

Treatment

Treatment Joint preserving Acetabular osteotomy Femoral osteotomy Safe surgical dislocation Arthroscopy

Acetabular osteotomy

Acetabular osteotomy

Acetabular osteotomy

Femoral osteotomy

Safe surgical dislocation

Arthroscopy labral tear

Arthroscopy FAI

Treatment Late / operative treatment indication Progressive increase in pain Rest pain Restriction of activity Marked deformity Progressive loss of movement Radiological evidence of joint destruction

Operative management Joint debridment Excision arthroplasty Arthrodesis Replacement Arthroplasty