Celiac plexus block. Dr.Kasturi Bhagawati Asst.professor Dept. of Emergency Medicine & Critical care.

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Celiac plexus block Dr.Kasturi Bhagawati Asst.professor Dept. of Emergency Medicine & Critical care.

Introduction A celiac plexus block is an injection of local anesthetic into or around the celiac plexus of nerves that surrounds the aorta. Involves blocking of sympathetic chain. Prevents transmission of pain impulses from the target organs to the brain

Celiac plexus block refers to temporary disruption of pain transmission via the celiac plexus and is accomplished by injecting corticosteroids or long-acting local anesthetics. celiac plexus neurolysis, refers to permanent destruction of the celiac plexus with ethanol or phenol. Imaging guidance for celiac plexus neurolysis is most often performed with multidetector computed tomography (CT), which has superseded the use of fluoroscopy- or ultrasonography (US) guided techniques

COMMON NEUROLYTIC BLOCKS Stellate ganglion block Thoracic sympathetic chain block Coeliac plexus block Lumbar sympathetic block Superior hypogastric block Ganglion impar block

Celiac plexus block

HISTORY 1914 KAPPIS first block in lateral position 1920 WELDING anterior approach. 1927 LABAT now followed retrocrural approach in prone position. 1947 _GAGE &FLOYED _ use in pancreatitis 1971 _GORBITZ _use of plain x ray 1979 _HEGEDEUS _fluroscopic guidance. 1982 SINGLERS CT guided transcrural approach 1983 ISCHIA posterior transaortic approach

LOCATION

Relation

INDICATIONS Chronic malignant & non malignant visceral pain 1. Upper g.i. malignancy 2. Chronic pancreatitis 3. Acute pancreatitis 4. Repeated abdominal surgeries 5. HIV related sclerosing cholangitis 6. Diagnostic purposes 7. Abdominal angina

Pathophysiological causes of pain in pancreatitis 1.Edematous distention of the pancreatic capsule 2.Local peritonitis resulting from enzyme release, causing a chemical burn of the peritoneum, 3.Ductal spasm from obstruction of the bile duct Radiation of pain to the back d/t anatomical location of pancreas retroperitoneally

CONTRAINDICATIONS ABSOLUTE Anti coagulant therapy Coagulopathy Anti-blastic cancer therapy Bowel obstruction Patient on disulfuram therapy

CONTRAINDICATION RELATIVE Drug seeking behaviour to pain Patient on CNS depressant drugs Liver abnormalities

Chronic Pain (Non-Cancer-Related) -1) systemic medications: - (NSAIDs), opioid analgesics, anticonvulsants, antidepressants -2) spinal and epidural analgesia -3) peripheral nerve blocks -4) sympathetic nerve blocks -5) other techniques: TENS, spinal cord stimulation, neuroablation - (surgical and chemical neurolysis)

Positioning Prone with pillow placed under the abdomen _posterior approach Right lateral decubitus with pillow under the flank_ one needle technique Supine position_ anterior approach

contd Hydration & electrolyte balance Analgesic Monitoring _EKG,pulse oximetry NIBP Oxygen

TECHNIQUE Posterior approach Retrocrural Antecrural transaortic transcrural Anterior approach

Other approaches Transintervertebral disc Endoscopic dennervation Intraoperative injection Flouroscopic guided Ct scan guided Ultrasound Contineous block via catheter

RETROCRURAL APPROACH

RETROCRURAL APPROACH Bilateral Posterior approach Splanchnic block Drug deposited behind the crus of diaphragm

MARKINGS

ANTECRURAL APPROACH Unilateral approach Right sided only Needle placed anterior to crus of diaphragm.

MARKINGS

Anterior approach

CONTINUOUS PLEXUS BLOCK

CT guided

COMPLICATIONS CHEMICAL ALCOHOL FACIAL FLUSHING, PALPITATIONS, DIAPHORESIS PHENOL TRANSIENT TINNITUS, FLUSHING,MALAISE CNS STIMULATION, MYOCLONUS, SEIZURES,HYPERTENSION,ARRYTHMIAS,HEPAT IC &RENAL INSUFFICIENCY

ADVANTAGE OF COELIAC PLEXUS NEUROLYSIS Better long term pain relief Decrease drug dose for maintainance Better quality of life Improved performance status Overcomes the G.I.T effects of opioids In weight and survival rate

FAILURE DUE TO Delayed application Tumour extension Poor technique

LOCAL ANAESTHETICS Xylocaine_1% soultion preservative free 15 to 20ml. Prevents pain by blockage of condution. T1/2 2hrs Avoid avoided if hypersentivity to amide Causes malignent hypothermia

LOCAL ANAESTHETICS 0.25% BUPIVACAINE PREFFERED FOR INTERMITTENT ADMINISTRATION 6-8 ml/hr 0F 0.1% BUPIVACAINE PREFFERED FOR CONTINUOUS ADMINISTRATION KEPT FOR MAXIMUM OF 7 DAYS