Differentiating Incontinence Associated Dermatitis from Category/Stage II Pressure Ulcers

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Differentiating Incontinence Associated Dermatitis from Category/Stage II Pressure Ulcers Suzanne Collins, MS BSN RN CWOCN Mid Atlantic Region Clinical Specialist Mölnlycke Health Care 1

Pre-Test: 1. What is the most important intervention in pressure ulcer reduction? 2. What does IAD stand for? 3. Who is responsible for reducing pressure ulcer incidence? 4. Is there a link between IAD and pressure ulcers?

Why is This Important? Hospital-acquired pressure ulcers are never events in acute care: Stage/Category III and IV wounds are not reimbursed at the higher diagnostic-related group for the costs of their care, and affect the hospital s standing in nursing-sensitive quality indicators. These wounds can be serious injuries that lead to death! 1 Incontinence-associated dermatitis can lead to inflammation, erosion, and secondary infection! 2

And Let s Not Forget Patients with incontinence-associated dermatitis can develop a pressure ulcer because excessive moisture reduces the skin s tolerance to excessive pressure! 3

Pressure Ulcer Snap Review: Remember that pressure ulcers usually occur over bony prominences. Can you identify these? Drawing: courtesy of NDNQI

Snap Review: Definition of a Pressure Ulcer-- A pressure ulcer is localized injury to the skin and/or underlying tissue usually over a bony prominence, as a result of pressure, or pressure in combination with shear and/or friction. A number of contributing or confounding factors are also associated with pressure ulcers; the significance of these factors is yet to be elucidated (National Pressure Ulcer Advisory and European Pressure Ulcer Advisory Panel, 2009 Pressure compresses tissue and blood vessels, hindering oxygen and nutrient delivery, leading to tissue death Shear deforms adipose and muscle tissue, and disrupts blood flow when one layer of tissue slides horizontally over another layer 4

Snap Review: Category/Stage I Intact skin with non-blanchable redness of a localized area usually over a bony prominence. Darkly pigmented skin may not have visible blanching; it s color may differ from the surrounding area Photos courtesy of Dot Weir Photo: Logicalimages 5

Snap Review: A Bit More on Category/Stage I and furthermore, the area may be painful, firm, soft, warmer, or cooler as compared to adjacent tissue. May indicate at risk persons. Stage I pressure ulcers may be difficult to detect in individuals with dark skin tones. Photos: Dot Weir

Snap Review: Category/Stage II Partial-thickness loss of dermis presenting as a shallow, open ulcer with a red pink wound bed, without slough. Stage II pressure ulcers may also present as an intact or open/ruptured serum-filled or serosanguineous-filled blister. Courtesy: Dot Weir Courtesy: Dot Weir Courtesy: NDNQI with permission

Snap Review: A Bit More on Category/Stage II Stage II presents as a shiny or dry shallow ulcer without slough or bruising. This stage should not be used to describe skin tears, tape burns, perineal (incontinence-associated) dermatitis, maceration, or excoriation. 6 Pop Quiz: What s going on here? Photo: WOCN image gallery

Snap Review: Category/Stage III Full thickness tissue loss. Subcutaneous fat may be visible, but bone, muscle or tendon are not exposed. Some slough may be present. Category/Stage III pressure ulcers may include undermining and tunneling Photo: NPUAP with permission

Snap Review: Category/Stage IV Full thickness tissue loss with exposed bone, tendon, or muscle. These ulcers often include undermining and tunneling Photo: NPUAP with permission

So, What is Incontinence- Associated Dermatitis? Simply put, IAD is a skin irritation that results from urinary and/or fecal incontinence A more complete definition is Inflammation and skin erosion associated with exposure to urine and/or stool, the use of absorptive containment devices, in which secondary cutaneous infection (usually fungal) is common 7 WOCN image gallery

How Tissue Loss Happens in IAD Exposure to urine: keratinocytes (skin cells) absorb urine, swell, and can no longer provide a barrier Ammonia in urine raises skin s ph, harming acid mantle, decreasing skin s resistance to external forces Exposure to stool: digestive enzymes denude the skin they literally DIGEST it! Exposure to pressure and shear: drag when moving patient, HOB elevated, inadequate turn intervals all create tissue ischemia, worsening skin integrity 8,9

What are the Risk Factors for IAD? Neuro-related incontinence History of incontinence Use of briefs or pads that increase exposure to moisture Look for: Skin that looks denuded in many areas Red flaring on the edges of the area of breakdown Skin may smell of ammonia, even after cleansing Photo: WOCN image gallery

More IAD Risk Factors Intensity of irritant: liquid stool with or without urine Duration of contact: linen and/or mattress pad wet every two hours Perineal excoriation, denuded state, or dermatitis Low serum albumin, tube feedings, presence of C. difficile, or antibiotic therapy 10 Photo: nih.gov

What Does IAD Look Like? Location: buttocks, perineum and upper thighs Look: Diffuse area of light to dark erythema Deepening skin color in dark-skinned patients Scaling of the skin Papule and vesicle formation Tissue weeping Secondary yeast or bacterial infection: red plaque with pinpoint satellite lesions that burns, or bright red, very itchy areas

Some Examples of IAD: Photo: WOCN Image Library Photo: WOCN image gallery Photo: WOCN image gallery

IAD Complicated by Pressure Hamburger Butt is one way to describe this complicated wound Photo: WOCN Image Library, accessed July, 2011

IAD Complicated by Pressure, Fungal, and Bacterial Infection Well, what would YOU call it? Photo: Google Image, accessed July, 2011

Wound Management Challenge What do you think this is? How would you manage it? 66 year old Caucasian female, immune suppressed secondary to chemotherapy for ovarian CA. Fecal and urinary incontinence. Foley to manage the urine. Moisture barrier ointment with dimethicone applied heavily to the affected area gave pain relief because the nerve endings were covered, and protected the lesions from fecal incontinence.

Risk factors: What About Yeast (Candidiasis)? Warm body areas Moist skin folds Hard-to-clean skin folds Obesity Antibiotic therapy History of incontinence Remember: skin may have bacterial AND yeast infection! Photos: WOCN image library

So, What s the Difference Between IAD and Pressure Ulcers? 11 IAD Pressure Ulcers Location: Diffusely distributed, skin folds Oval or round, over pressure areas/bony prominences Color: Red or bright red Red or purple Depth: Partial thickness Partial or full thickness Necrotic Tissue: Symptoms: None Pain or itching, with history of incontinence Slough or eschar if full thickness Pain or itching, not necessarily with a history of incontinence

So, This: IAD: Diffusely distributed, in skin folds, red or bright red, without slough or necrotic tissue, causing pain and/or itching. There is always history of incontinence!

Or This Photos: Dot Weir Category/Stage II pressure ulcer: usually oval or round, over a bony prominence, red or purplish, and partial thickness. What if there s slough? What if it s purple?

Hey, Wait A Minute: What About That 3 rd Photo? Special point: When a patient has been supine for an extended period, pressure ulcers can occur on the cheeks of buttocks This happens when the patient was positioned flat These can be Deep Tissue Injuries Show as single wounds with distinct edges Photo: Courtesy of Elliott Douglas, VUMC

Deep Tissue Injuries? Yes! Remember that DTI is a pressure ulcer Category/Stage. DTI leads to epidermal loss at about 48 hours from injury 12 The bed of the ulcer is dark and usually purple There is usually no history of incontinence! 13 Photo: NPUAP with permission

Pressure Ulcers That Are Associated With Moisture: Usually due to pressure on moisture-damaged skin Usually found on the pelvis Necrotic tissue IS often present Exudate is common, but may be hidden by stool or urine if patient is incontinent May become infected Associated with malnutrition (especially low protein) Photo: NPUAP with permission

So, What Do We Do? Determine the cause of the incontinence! Acute urinary incontinence: often from a UTI post-menopausal women history of instrumentation, Catheterization women recently sexually active-

Determine the Cause, contd. Chronic urinary incontinence: colonized urine elders with cognitive problems patients with mobility problems Photo: WOCN Image Library

And, Of Course-- Keep skin clean and dry, and use skin protectant Watch for infectious forms of diarrhea Consider side-effects of medications or tube feedings as cause of diarrhea Immediately treat fungal skin infection Consider Foley catheter if skin denudement severe Use containment products carefully

And Also Consider fecal collector for frequent diarrhea Monitor for dehydration, which can worsen incontinence Consult MD/WOCN/NP for evaluation for fecal management system if diarrhea is high-volume and greater than 6 stool episodes per day

Protect the Skin! Avoid plastic or rubberized drawsheets on low airloss beds Keep incontinence care products at the bedside Keep skin moisturized Avoid use of abrasive wash cloths Use effective barrier cream after each exposure Avoid zinc oxide, as it can damage skin to remove Consider bowel management system if diarrhea meets criteria for use and patient care setting 13

Protect the Wound! Isolate any wound from contact with incontinence Use a waterproof dressing if possible to prevent contamination of wound and wound breakdown from excessive moisture If necessary, seal edges of dressing with waterproof liquid skin barrier to reduce chance of leaking into dressing Wipe stool or urine off waterproof dressing rather than change each time patient is incontinent

Absorbent Bordered Soft Silicone Foam Sacral Dressing Intended Use Designed for a wide range of exuding wounds such as sacral pressure ulcers. Can also be used on dry or necrotic wounds in combination with gels

Manufactured in Silver-Ion Releasing Form to Provide Antimicrobial Effect Several other versions of sacral foam dressings available to address complex sacral wound management challenges

Absorbent Bordered Soft Silicone Dressings Photos: Dunbar, A. et al. SAWC Poster, 2006 and MHC, data on file

Post-Test 1. Name two differences between IAD and a Category/Stage II pressure ulcer 2. What is the Number One most important thing to do to prevent a pressure ulcer? 3. Name two effects that incontinence has on the skin, either urinary or fecal 4. Urinary incontinence is often a sign or signal of:

Answer Key 1. IAD skin breakdown is ALWAYS found when the patient is incontinent; pressure ulcers are not necessarily 2. Reduce pressure 3. Swells keratinocytes, increases friction and shear between skin and surface, can expose skin to enzymes, etc 4. Urinary tract infection

Thank you! Any questions?

References: 1. Armstrong, D; Ayello, E. et al: New opportunities to improve pressure ulcer prevention and treatment. JWOCN; 2008: 35(5). Pg.485-6. 2. NDNQI pressure ulcer tutorial: https://www.nursingquality.org/ndnqipressureulcertraining/module1/pr essureulcerdefinition_1.aspx. Accessed Jan 3, 2010. 3. Ibid. 4. Ibid. 5. Logicalimages:http://www.logicalimages.com/ publichealthresources/pressureulcer.htm. Accessed Jan 3, 2010 6. Ibid. #2. 7. Black, Joyce. Incontinence-associated dermatitis and pressure ulcers: what is the connection presented at SER WOCN, Oct 24, 2009.

References, Contd. 8. Junkin, J. and Selekov, J. Prevalence of incontinence and associated skin injury in the acute care inpatient. JWOCN, 34, (3). Pg. 260-269. 9. Ibid., No. 7. 10. Bryant, R. and Nix, D. Acute and chronic wounds: current management concepts, 3 rd ed. St Louis: Mosby Elsevier, 2007. Pg. 211-230. 11. Gray, M. et. Al., Incontinence-associated dermatitis: A consensus. JWOCN, 2007; 34 (1). Pg.45-54. 12. Black, J. Deep tissue injury: an evolving science. Ostomy Wound Management, 2009,VOLUME: 55 (2) 13. Ibid. #11