NASAL OBSTRUCTION. Andy Whyte PERTH RADIOLOGICAL CLINIC UNIVERSITY OF MELBOURNE UNIVERSITY OF WA

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Transcription:

NASAL OBSTRUCTION Andy Whyte PERTH RADIOLOGICAL CLINIC UNIVERSITY OF MELBOURNE UNIVERSITY OF WA

INTRODUCTION sinonasal imaging focuses on structural abnormalities of the POSTERIOR (BONY 3/4 ) of the nose : septal deviation (SD), turbinate morphology + size, spurs, variants SD is associated with an risk of CRS but is a normal finding - 90% adults, M>>F NASAL OBSTRUCTION (NO) is a common symptom and a FREQUENT indication for imaging the ANTERIOR NOSE (SOFT TISSUE 1/4) is of critical importance in nasal physiology including REGULATION of airflow ANTERIOR NASAL PATHOLOGY is the MAJOR cause of nasal obstruction: a small deviation of the anterior nasal septum can lead to significant NO whereas a large deviation of the posterior septum has no effect on airflow resistance

FUNCTIONS OF THE NASAL CAVITY HEAT EXCHANGE HUMIDIFY FILTER inspired air OLFACTORY sensation REGULATE air flow and respiration MUCOCILIARY CLEARANCE IMMUNOLOGICAL DEFENSE with remainder of the upper and lower respiratory tract: UNIFIED AIRWAY PLEASURE

FUNCTION AND TYPE OF EPITHELIUM olfactory area B A C D E A: skin B : modified skin thermoreceptors C: transitional epithelium D: pseudostratified columnar with few cilia E: pseudostratified columnar with many cilia

NASAL AIRFLOW Within the INTERNAL NASAL VALVE (INV), turbulent inspired air is converted to a high velocity jet of LAMINAR flow 70% of air Passes POSTERIORLY and HORIZONTALLY between the middle (MT) and inferior turbinates (IT). POST POST ANT ANT

70% airflow passes between the middle and inferior turbinates NASAL AIRFLOW HORIZONTAL

NASAL BREATHING NASAL BREATHING occurs in the inferior half of the nose IT and MT act like shutters deflecting airflow in a stable + orderly pattern: LAMINAR flow

NASAL AIRFLOW - VALVES 50-70% of resistance to airflow in the upper airway is ANTERIOR: NOSTRIL ( EXTERNAL NASAL VALVE) VESTIBULE INTERNAL NASAL VALVE: KEY INTERNAL NASAL VALVE INTERNAL NASAL VALVE MT IT V V NOSTRIL (EXTERNAL NASAL VALVE)

NASAL AIRFLOW IN VALVE Internal nasal valve valve angle anterior nose head IT

NASAL AIRFLOW - VALVES resistance to airflow in the anterior (external) nose is regulated by: thermoreceptors in the vestibule monitor air temperature reflex contraction or relaxation of the dilator muscles

CT OF THE NASAL VALVES ENV INV INV V ENV INV, VESTIBULE and ENV are assessed by a dynamic (stress) manoeuvre: modified Cottle procedure performed in inspiration CT can be used for the same purpose but provides static assessment only

CT OF THE INTERNAL NASAL VALVE grade as normal OR mild - moderate - severe narrowing or measure the valve angle + area ( see refs) average angle:10degrees Narrow R INV Narrow INV L>R average area = 84mm2

CASE 1: 45M post septoplasty + partial inferior turbinectomies c/o snoring/ R nasal obstruction cartilaginous septum ant bony septum mid bony septum CB BE oblique for valves RIGHT LEFT CB BE CB BE INT NV

CASE 1: 45M nose, age, sex, habitus + narrowed retropalatal OP hyoid and OP length normal

CASE 2: 42M previous septoplasty, RIGHT nasal obstruction ant bony septum cartilaginous septum oblique for INV

CASE 3 : 37F LEFT nasal obstruction normal INVs and constricted L ENV Coronal: piriform aperture Coronal: ant cart septum External Oblique: Vestibule NVINV

CASE 4: 44M bilateral NO no deviation of the bony septum INV vestibule ENV

NASAL OBSTRUCTION: NO KEY FACTS during sleep, upper airway (UA) resistance is 2.5 x lower with nasal rather than oral breathing. Normally, less than 5% of breathing is oral in type with NO, there is a switch to inefficient oral breathing and reduction in airway dimensions exacerbated by the supine position the nasal-ventilatory reflex is absent during oral breathing; airflow through the anterior nose stimulates breathing and this protects against apnoeas reversible NO, most commonly due to allergic rhinitis and non-allergic rhinitis (NAR) closely correlates (83%) with SDB rather than structural abnormalities: septal deviation, spurs, CB that we carefully report on CT. NO is not a major factor in the aetiology of OSA but it can exacerbate this condition ; it is a major factor causing habitual snoring (SDB) following a septoplasty, narrowing of the INV is the major cause of failure to improve NO

NASAL OBSTRUCTION: posterior 3/4 structural abnormalities? importance surgery is NOT benign: resection of IT + septal perforations disrupt laminar flow most severe manifestation of radical surgery is the paradoxical sensation of NO: empty nose syndrome now grudgingly accepted as a significant reality!

INCIDENCE NASAL OBSTRUCTION - SUMMARY treatment of NO improves OSA + can cure snoring SOFT TISSUE rhinitis polyps ANT NOSE cart. septal deviation narrowed INV POST NOSE bony septal deviation spur + CB SPECTRUM OF BENIGN NASAL OBSTRUCTION NIL empty nose S

REFERENCES Computed Tomography Technique for Evaluation of the Nasal Valve. Arch Facial Plast Surg. 2004;6: 240-243 The role of the nose in snoring and obstructive sleep apnoea: an update. Eur Arch Otorhinolaryngol 2011; 268(8): 1365 1373 Anatomy and Physiology of the Upper Airway. Proc Am Thorac Soc; 8: 31 39 The role of 3 dimensional CT in the evaluation of nasal structures and anomalies. Eur Arch Otorhinolaryngol 2011;268(8):1163-1167 Value of CT scan measures of the nasal valve for predicting clinical nasal obstruction. ECR exhibit. 2011 Reformatted Computed Tomography to Assess the Internal Nasal Valve and Association With Physical Examination. Arch Facial Plast Surg. 2012;14(5):331-335