DPP-4 inhibitor. The new class drug for Diabetes

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DPP-4 inhibitor The new class drug for Diabetes 1

Cause of Death in Korea 1 st ; Neoplasm 2 nd ; Cardiovascular Disease 3 rd ; Cerebrovascular Disease Diabetes 2

Incidence of Fatal or Nonfatal MI During a 7-Year Follow-up in Relation to History of MI in Non-diabetic vs Diabetic Subjects: East-West Study Incidence During Follow-up (%) 5 4 3 2 1 Nondiabetics + OMI Nondiabetics Diabetics + OMI Diabetics 18.8 3.5 45. 2.2 (n=69) (n=134) (n=169) (n=89) Events per 1 person-yr: 3..5 7.8 3.2 3 Haffner SM et al. N Engl J Med 1998;339:229-234.

Stepwise Selection of Risk Factors in 2693 White Patients with Type 2 Diabetes with Dependent Variable as Time to First Event: UKPDS Coronary Artery Disease (n=28) Position in Model First Second Third Fourth Fifth Variable Low-Density Lipoprotein Cholesterol High-Density Lipoprotein Cholesterol Hemoglobin A 1c Systolic Blood Pressure Smoking P Value <.1.1.22.65.56 Adjusted for age and sex. Turner RC et al. BMJ 1998;316:823-828. 4

Abnormal Pancreatic Islet Function Determines the Development of IGT and T2DM in the Setting of Insulin Resistance Age, lifestyle, environmental factors Insulin resistance Intact islet function Decreased islet function NGT IGT / T2DM T2DM = type 2 diabetes mellitus; NGT = normal glucose tolerance; IGT = impaired glucose tolerance Adapted 5 from Ahren B, Pacini G. Diabetes Obes Metab. 25;7(1):2 8.

L1 Deterioration of Islet Function Drives Disease Progression Diagnosis Glucose Insulin Postprandial glucose Fasting glucose Insulin resistance Insulin secretion Inadequate β-cell function Decreasing β-cell function Microvascular changes Macrovascular changes NGT Prediabetes (IFG/IGT) Diabetes NGT = normal glucose tolerance; IFG = impaired fasting glucose; IGT = impaired glucose tolerance Adapted 6 from Type 2 Diabetes BASICS. International Diabetes Center; 2.

슬라이드 6 L1 Animation builds to be added during formatting LatiD6IT, 26-4-5

Pancreatic Islet Hormones are Critical for Normal Glucose Tolerance + Glucagon HGO α-cells Glucose β-cells Insulin HGO= Hepatic Glucose Output Adapted from Unger RH. Metabolism. 1974;23:581. 7 + Glucose uptake

Pancreatic Islet Dysfunction leads to Hyperglycemia in T2DM α-cell Hypertrophy 2 Fewer β-cells Excessive Glucagon + HGO Glucose Insufficient Insulin + Glucose uptake HGO= Hepatic Glucose Output Adapted from Ohneda A, et al. J Clin Endocrinol Metab. 1978;46:54 51; and Gomis R, et al. Diabetes Res Clin Pract. 1989;6:191 198. 8

Insufficient Insulin and Elevated Glucagon in T2DM ( Insulin/Glucagon Ratio) 4 CHO meal mg/dl 3 2 1 15 Glucose NGT T2DM mu/ml 1 5 Insulin 15 pg/ml 125 1 Glucagon T2DM = Type 2 Diabetes Mellitus; NGT = Normal Glucose Tolerance; CHO = Carbohydrate Adapted from Muller WA, et al. N Engl J Med. 197;283:19 115. 9 75-6 6 12 18 24 Time (min)

Proof of a Gastrointestinal Incretin Effect : Different Responses to Oral vs IV Glucose Oral Glucose Tolerance Test and Matched IV Infusion 2 4 5 g glucose Plasma Glucose (mg/dl) 15 1 5 Plasma Insulin (pmol/l) 3 2 1 3 3 6 9 12 15 18 21 Time (min) 3 3 6 9 12 15 18 21 Time (min) N = 6 IV = intravenous Adapted from Nauck MA, et al. J Clin Endocrinol Metab. 1986;63:492 498. 1 Oral IV

Pancreatic Islet Cells Are Targets for Incretin Hormones α-cell β-cell Food intake GLP 1 Incretin Response Pancreatic Islet GLP-1 = glucagon-like peptide-1 Adapted from Drucker D. Diabetes Care. 23;26:2929 294. Wang Q, et al. Diabetologia. 24;47:478 487. 11

GLP-1 Secretion and Inactivation Meal Intestinal GLP-1 release GLP-1 t ½ = 1 to 2 min Active GLP-1 DPP-4 GLP-1 inactive (>8% of pool) GLP-1 = glucagon-like peptide 1; DPP-4 = dipeptidyl-peptidase 4 Adapted 12 from Deacon CF, et al. Diabetes. 1995;44:1126-1131.

What Is DPP-4? A serine protease widely distributed throughout the body Cleaves N-terminal amino acids of a number of biologically active peptides, including the glucostatic incretins GLP-1 and GIP, for inactivation DPP-4 effects on GLP-1 and GIP proven to play a key role in incretin activity and glucose homeostasis Inactivates GLP-1 >5% in ~1 2 min Inactivates GIP >5% in ~7 min DPP-4 = dipeptidyl peptidase-4; GIP = glucose-dependent insulinotropic peptide; GLP-1 = glucagon-like peptide-1; t½ = half-life Adapted from Ahrén B. Curr Enzyme Inhib. 25;1:65 73. 13

Inhibition of DPP-4 Increases Active GLP-1 Meal Intestinal GLP-1 release GLP-1 t ½ = 1 2 min Active GLP-1 DPP-4 inhibitor DPP-4 GLP-1 inactive (>8% of pool) GLP-1 = glucagon-like peptide-1; DPP-4 = dipeptidyl peptidase-4 Adapted from Rothenberg Deacon CF, P, et et al. al. Diabetes. 1995;44:1126-1131. 2;49(suppl 1):A39. Abstract 16-OR. 14

Blocking DPP-4 Can Improve Incretin Activity and Correct the Insulin : Glucagon Ratio in T2DM T2DM Incretin Response Diminished Insulin Further impaired islet function Glucagon HYPERGLYCEMIA DPP-4 Inhibitor Incretin Activity Prolonged Insulin Improved islet function IMPROVED GLYCEMIC CONTROL DPP-4 = dipeptidyl peptidase-4; T2DM = type 2 diabetes mellitus Adapted from Unger RH. Metabolism. 1974;23:581 593. Ahrén B. Curr Enzyme Inhib. 25;1:65 73. 15 Glucagon

GLP-1 Restores Islet Glucose Sensing in Patients with T2DM Glucose (mg/dl) C-peptide (nmol/l) Glucagon (pmol/l) 3 GLP-1 infusion 3. GLP-1 infusion 3 GLP-1 infusion 25 2.5 25 2 15 2. 1.5 2 15 1 1. 1 5 GLP-1 Saline 3 3 6 9 12 15 18 21 24 Time (min). 3 3 6 9 12 15 18 21 24 P <.5 GLP-1 = glucagon-like peptide 1; T2DM= type 2 diabetes mellitus Adapted 16 from Nauck MA, et al. Diabetologia. 1993;36:741 744..5 Time (min) 5 3 3 6 9 12 15 18 21 24 Time (min)

Vildagliptin (LAF237) Glycylpyrrolidine H O N HO N N Highly selective DPP-4 inhibitor Has a high affinity for the human enzyme Reversible inhibition X-ray crystallographic structure of LAF237 (green) bound to the active site (yellow) of human DPP-4 17

Plasma DPP-4 Activity after Single Oral Doses of Vildagliptin (1 to 4 mg) in Patients with T2DM 125 DPP-4 activity (% baseline) 1 75 5 25 Placebo (16) 1 mg (16) 25 mg (16) 5 mg (16) 1 mg (15) 2 mg (16) 4 mg (16) -2 2 4 6 8 1 12 14 16 18 2 22 24 Time (h) Data 18 on file, Study LAF2215, Novartis

Plasma Levels of Intact (Active) GLP-1 after a Single Oral Dose of Vildagliptin (1 mg) or Placebo in Patients with T2DM meal 16. dose Placebo (16) Vildagliptin (16) Active GLP-1 (pmol/l) 12. 8. 4.. -12-6 6 12 18 24 3 36 42 48 54 6 66 72 78 84 Study 19 LAF2346, data on file Time (min)

Change from Baseline (pre-meal) in Plasma Glucagon after Single Oral Dose of Vildagliptin (1 mg) or Placebo meal 2 1 dose Placebo (16) Vildagliptin (16) Delta Glucagon (ng/l) -1-2 -3-4 -5-6 -6 6 12 18 24 3 36 42 48 54 6 66 72 78 84 Time (min) Study 2 LAF2346, data on file

Vildagliptin increases Insulin, and decreases Glucagon concentrations Resulting in lower Glucose levels in Patients with T2DM OGTT 3 min after single oral dose of Vildagliptin (5 mg) Insulin (pmol/l) 6 4 2 25. Placebo (16) Vilda 5 mg (16) Insulin AUC -5 (mnh) 1.8 1.6 1.4 1.2 1..8.6 Placebo Vildagliptin Glucose (mmol/l) Glucagon (ng/l) 2. 15. 1. 5. 125 1 75 5-9 -6-3 3 6 9 12 15 18 21 24 27 3 P<.1 Time (min) Data on file, Study LAF2215, Novartis 21 Glucagon AUC -4 (ng/lh) Glucose AUC -5 (mnh) 8 75 7 65 6 38 36 34 32 3 Placebo Placebo Vildagliptin Vildagliptin

Meal-related β-cell Function and Insulin Sensitivity after Vildagliptin vs. Placebo in Patients with T2DM Patients on Stable Metformin Therapy Insulin Secretion Insulin Sensitivity Adaptation Index pmol/l 3min/(mmol/L).5.45.4.35.3.25 12 24 52 ml min -1 m -2 3 275 25 225 2 12 24 52 14 12 1 8 6 12 24 52 Time (wk) Time (wk) Time (wk) Vildagliptin/metformin Placebo/metformin P<.5 vs. placebo; P<.1 vs. placebo Adapted 22 from Ahren B, et al. Diabetes Care. 25;28:1936-194

Vildagliptin Therapy Significantly Lowers HbA 1c over the course of 1 Year 8.4 Vildagliptin/MET (extension, ITT n=42) PBO/MET (extension, ITT n=29) Vildagliptin/MET (core, ITT n=56) PBO/MET (core, ITT n=51) 8. HbA 1c (%) 7.6 P <.1 P <.1 1.1 ±.2% 7.2 6.8 4 4 8 12 16 2 24 28 32 36 4 44 48 52 Week Met=Metformin; PBO=Placebo; ITT=Intent-To-Treat Adapted 23 from Ahrén B, et al. Diabetes Care. 24; 27(12):2874 288

New Approaches to Harnessing Incretins for Improved Glucose Control Two Approaches to Prolonging Incretin Activity DPP-4 Inhibitors Significant HbA 1c reduction Weight neutral Oral administration Almost no GI side effects Very low rate of hypoglycemia Multiple targets (GLP-1, GIP) Drug overdose not toxic Incretin Mimetics Significant HbA 1c reduction Weight loss Injection Higher rate of GI side effects Higher rate of hypoglycemia Single target (GLP-1) Drug overdose problematic DPP-4 = dipeptidyl peptidase-4; GI = gastrointestinal; GIP = glucose-dependent insulinotropic peptide; GLP-1 = glucagon-like peptide-1; HbA 1c = hemoglobin A 1c When administered with sulfonylureas Adapted from Ahrén B. Curr Enzyme Inhib. 25;1:65 73. Drucker D. Diabetes Care. 23;26:2929 294. 24

Summary: Potential of DPP-4 Inhibition in the Treatment of T2DM DPP-4 is a ubiquitous enzyme that rapidly inactivates more than 5% of the glucostatic incretins GLP-1 and GIP Two approaches to prolonging incretin activity are: DPP-4 inhibitors (oral agents): inhibit the degradation of active incretins Incretin mimetics (injectable agents): degradation-resistant GLP-1 analogues Inhibiting DPP-4 results in: Improved islet cell function through increased levels of intact incretin hormones (GLP-1, GIP) Improved glycemic control DPP-4 = dipeptidyl peptidase-4; GIP = glucose-dependent insulinotropic peptide; GLP-1 = glucagon-like peptide-1; T2DM = type 2 diabetes mellitus 25

Summary Vildagliptin inhibits DPP-4 resulting in Fasting and post prandial GLP-1 and GIP levels Enhanced islet function sensitivity of glucagon secretion to glucose sensitivity of insulin secretion to glucose 1st phase insulin secretion capacity to secrete insulin Post prandial triglycerides Insulin resistance No effect on gastric emptying 26

Vildagliptin in Monotherapy

Overview of Phase III Studies 235 SU add-on 234 TZD add-on 2355 Initial Combo with TZD 233 Met add-on Prediabetes Diet & Exercise OAD Mono OAD Combo Insulin Mono Insulin Combo Outcome 231 Dose finding 2311 Insulin add-on 239 Metformin mono 2327 Rosiglitazone mono 28

Demographics: Key Monotherapy Studies Placebo-controlled monotherapy study Study 231 Vilda 5 mg qd Vilda 5 mg bid Vilda 1 mg qd PBO N 14 9 92 94 Gender (%) F 58.7 53.3 46.7 52.1 M 41.3 46.7 53.3 47.9 Age (y) Mean 55.3 52.8 53.6 52.2 Wh 73.1 73.3 76.1 69.1 Race/Ethnicity (%) His 13.5 13.3 15.2 11.7 As 2. 1.1 4.4 5.3 Bl 9.6 1. 4.3 12.8 Active-controlled monotherapy studies Study 239 Vilda 5 mg bid Met 1 mg bid 526 254 47.1 42.5 52.9 57.5 52.8 53.6 67.9 69.7 19.8 21.7 2.1 2.4 8. 5.1 Study 2327 Vilda 5 mg bid Rosi 8 mg qd 459 238 42.5 42.4 57.5 57.6 54.5 54.2 79.5 79.8 11.1 12.2 2.4 1.6 5.9 4.6 F = female, M = Male; Wh = white/caucasian, His = Hispanic/Latino, As = Indian/non-Indian Asians, Bl = black/african origin Primary efficacy ITT patients Data 29 on file, Novartis

Disease Features at Baseline: Key Monotherapy Studies N BMI (kg/m 2 ) HbA 1c (%) FPG (mmol/l) Duration (y) Mean Mean Mean Mean Placebo-controlled monotherapy study Study 231 Vilda 5 mg qd Vilda 5 mg bid Vilda 1 mg qd PBO 14 9 92 94 32.9 33.3 32.4 32.6 8.2 8.6 8.4 8.4 9.8 1.1 9.9 9.9 2.1 2.1 2.4 1.6 Active-controlled monotherapy studies Study 239 Vilda 5 mg bid Met 1 mg bid 526 254 32.4 32.5 8.7 8.7 1.5 1.5 2.4 2.2 Study 2327 Vilda 5 mg bid Rosi 8 mg qd 459 238 32.2 32.9 8.7 8.7 1.3 1.3 2.3 2.7 Primary efficacy ITT patients Data on file, Novartis 3

Monotherapy: HbA 1c Reduction PBO-controlled 231 Baseline ~ 8.4 vs. Met 239 (24wks) Baseline ~ 8.7 vs. Rosi 2327 Baseline ~ 8.7 Adjusted mean HbA 1c (%). -.5-1. -1.5-2. Vilda 5 qd Vilda 5 bid -.78 -.79 Vilda 1 qd -.88 P<.1 vs. PBO for all doses PBO -.3 Vilda 5 bid -1.14 Met 1 bid -1.45 Not NI to Met Vilda 5 bid -1.13 Rosi 8 qd -1.32 NI to Rosi ITT=intention to treat; NI=non-inferiority Primary 31 efficacy ITT population Data on file Novartis

Monotherapy: Time Course of HbA 1c Reduction 9. PBO-controlled 231 (24 weeks) Placebo (n=94) Vildagliptin (5 mg qd) Mean HbA 1c (%) 8.5 8. Vildagliptin(5 mg bid) Vildagliptin(1 mg qd) 7.5 7. -4 4 8 12 16 2 24 Time (wk) Data 32 on file, Novartis

Study 231: Efficacy of Monotherapy in Stable vs. Newly Diagnosed Patients Time since diagnosis > 2 weeks (84% of pts) BL 8.3% 8.5% 8.4% 8.4% Time since diagnosis 2 weeks (16% of pts) 8.2% 8.7% 8.5% 8.6% Adjusted mean HbA 1c (%) -.5-1. -1.5-2. Vilda 5 qd -.64 Vilda 5 bid Vilda 1 qd PBO -.68 -.79 -.11 P=.5; P=.3; P <.1 vs PBO Vilda 5 qd -1.9 Vilda 5 bid Vilda 1 qd PBO -1.5-1.53-1.3 Vilda=vildagliptin; PBO=Placebo Primary ITT population Data 33 on file, Novartis

Study 231: Efficacy of Monotherapy in Stable vs. Newly Diagnosed Patients Diabetes diagnosis 3 months (61% of pts) Diabetes diagnosis <3 months (39% of pts).5 BL 8.3% 8.4% 8.4% 8.3% 8.1% 8.7% 8.5% 8.5% Adjusted mean HbA 1c (%). -.5-1. Vilda 5 qd -.7 Vilda 5 bid -.5 -.5 Vilda 1 qd -.7 -.7 -.7.2.2 PBO Vilda 5 qd -.9 -.9 Vilda 5 bid -1.3 Vilda 1 qd -1.3 PBO -1. -1. -1.5 Primary efficacy ITT population Data 34 on file, Novartis P<.1, P=.3 vs. PBO -1.3-1.3

Efficacy by BL HbA 1c Subgroups: Pooled Monotherapy Data (Vildagliptin 1 mg/day) HbA 1c 8 Baseline: 7.6% HbA 1c >8 Baseline: 9.3% HbA 1c >9 Baseline: 9.9% Change in HbA 1c (%). -.2 -.4 -.6 -.8-1. -1.2-1.4-1.6-1.8 n=375 n=684 n=373 -.63-1.35-1.71 Vildagliptin 5 mg bid Pooled analysis from LAF237A231, 239, 2327 primary efficacy ITT population Data 35 on file, Novartis Pharmaceuticals

No Weight Gain Associated with Vildagliptin Monotherapy over 52 weeks Vildagliptin vs. Metformin (52 weeks) Body weight (kg) 1 95 9 85 Vildagliptin 5 mg bid Metformin 1 mg bid 8-4 4 12 2 28 36 44 52 Time (wk) Vilda=Vildagliptin; Met=Metformin ITT population Data 36 on file, Novartis Pharmaceuticals, LAF237A239

Monotherapy: Fasting Lipids vs. Placebo 231 vs. Metformin 239 (Wk 24) Adj. mean change (%) 15. 1. 5.. -5. -1. -15. Vildagliptin 5 mg qd Vildagliptin 5 mg bid Vildagliptin 1 mg qd Placebo TG Total-C LDL-C HDL-C Non HDL-C Adj. mean change (%) 15. 1. 5.. -5. -1. -15. Vildagliptin 5 mg bid Metformin 1 mg bid TG Total-C LDL-C HDL-C Non HDL-C vs. Rosiglitazone 2327 Vildagliptin 5 mg bid Rosiglitazone 8 mg qd Adj. Mean Change (%) 15. 1. 5.. -5. -1. -15. TG Total-C LDL-C HDL-C Non HDL-C Data 37 on file, Novartis

Favorable Safety and Tolerability of Vildagliptin in Monotherapy Overall incidence of AEs comparable to Placebo No relationship to dose in the frequency of AEs Severity of AEs comparable in all vildagliptin dose groups No relationship between duration of exposure and onset of AEs Discontinuation rate due to AEs less than Placebo No increased risk of hypoglycemia at any dose Superior GI tolerability vs metformin (diarrhea: 6% vs 26%) No increased rates of notable changes in labs, vital signs, and clinically relevant prolongations of ECG conduction intervals during chronic use or at C max compared with placebo 38

Vildagliptin Safety in Monotherapy: AEs 5% Incidence in Any Group Preferred term, % Vilda 5 mg qd (N=323) Vilda 5 mg bid (N=1292) Vilda 1 mg qd (N=238) Met up to 1 g bid (N=252) Rosi 8 mg qd (N=267) Pio 3 mg qd (N=55) PBO (N=255) Any 56.3 63.5 64.3 75.4 64. 45.5 6. Nasopharyngitis 6.2 7.7 7.6 9.5 7.5 1.8 7.1 Headache 4.6 7.1 7.1 7.1 5.2 5.5 5.9 Dizziness 4.6 5.7 6.3 6. 4.1 3.6 4.3 Upper respiratory tract infection 1.2 4.8 3.8 6. 3. 1.8 2.7 Diarrhea.9 3.5.8 26.2 2.6 3.6 3.1 Nausea 1.5 3.2 1.7 1.3.7 1.8 3.9 Abdominal pain.3 1.4. 7.1.7 3.6 1.2 AE=adverse event; Vilda=vildagliptin; Met=metformin; Rosi=rosiglitazone; Pio=pioglitazone; PBO=placebo Preferred terms are sorted by descending order of incidence in the vildagliptin 5 mg bid group. Note: A patient with multiple occurrences of an AE under one treatment is counted only once in the AE category for that treatment. Data on file, Novartis. 39

Vildagliptin Monotherapy: Lower Incidence Rate of Edema vs Rosiglitazone Preferred Term Any AE Headache Diarrhea Nausea Vomiting Myalgia 42327- Data on file, Novartis Peripheral Edema Hypertension Paraesthesia Upper respiratory infections ECG abnormalities Vilda N=515 (%) 316 (61.4) 11 (2.1) 26 (5) 7 (1.4) 18 (3.5) 11 (2.1) 11 (2.1) 1 (2) 23 (4.5) 6(1.2) 51 (9.9) Rosi N=267(%) 171 (64.) 11 (4.1) 14 (5.2) 7 (2.6) 2 (.7) () 5 (1.9) 1 (3.7) 8 (3) 3 (1.1) 32 (12)

Vildagliptin Hypoglycemic Events in Monotherapy Vilda 5 mg qd (N=323) Vilda 5 mg bid (N=1292) Vilda 1 mg qd (N=238) Met up to 1 g bid (N=252) Rosi 8 mg qd (N=267) Pio 3 mg qd (N=55) PBO (N=255) % of Pts with > 1 hypoglycemic event.6.3.8.4.4 % of Pts who disc. due to hypo Total no of hypos 2/323 4/1292 3/238 1/252 1/267 /55 /255 Severity Grade 1 2 4 3 1 1 Grade 2 Suspected Grade 2 Hypoglycemic events are defined as: Grade 1: Symptoms suggestive of hypoglycemia, where the patient is able to initiate self-treatment and plasma glucose measurement is <3.1 mmol/l; Grade 2: Symptoms suggestive of hypoglycemia, where the patient is unable to initiate self-treatment and plasma glucose measurement is <3.1 mmol/l, Suspected Grade 2: Symptoms suggestive of hypoglycemia, where the patient is unable to initiate self-treatment and no plasma glucose measurement is available Data on file, Novartis 41

General Summary: Vildagliptin in Monotherapy Produces significant reductions in HbA1c levels (up to 1.8% in monotherapy) Sustains meaningful HbA1c reductions out to 1 year Has neutral effect on body weight Convenience of use: Simple oral dosing, no need for titration, very low potential for drug-drug interaction Superior GI tolerability vs metformin Overall incidence of AEs comparable to placebo No increased risk of hypoglycemia at any dose Potential for disease modification based on islet-cell effects and animal data 42

Vildagliptin in Combotherapy

Vildagliptin Shows Consistent and Clinically Relevant Reductions of HbA1c in Add-on Combination Therapy Add-on to Met Add-on to SU Add-on to Pio Add-on to Insulin Change From Baseline in HbA1c (%).4 233 (24 wks) Baseline ~ 8.4%.2. -.2 -.4 -.6 -.8-1. -1.2 -.51 -.88.23 235 (24 wks) Baseline ~ 8.5% -.58 -.63.7 Vilda-vildagliptin, Met=metformin; SU=sulfonylurea; Pio=Pioglitazone PBO=placebo Primary efficacy ITT population 44 P<.1, # P=.22 (vs PBO) Data on file, Novartis Pharmaceuticals 234 (24 wks) Baseline ~ 8.7% -.76 -.97 -.3 2311 (24 wks) Baseline ~ 8.5% -.51 # -.24 Vilda 5 mg qd + Comparator Vilda 5 mg bid + Comparator PBO + Comparator

FPG Reductions in Add-on Combination Therapy Add-on to Met Add-on to SU Add-on to Pio Add-on to Insulin Change From Baseline in FPG (mmol/l) 1..5. -.5-1. -1.5 233 (24 wks) BL ~ 9.9 -.16 -.99.67 235 (24 wks) BL ~ 1.5 -.32 -.44.18 Vilda-vildagliptin, Met=metformin; SU=sulfonylurea; Pio=Pioglitazone PBO=placebo Primary efficacy ITT population 45 P<.5, vs PBO+Comparator Data on file, Novartis Pharmaceuticals 234 (24 wks) BL ~ 1.1 -.82-1.13 -.48 -.5 2311 (24 wks) BL ~ 9.3 -.25 Vilda 5 mg qd + Comparator Vilda 5 mg bid + Comparator PBO + Comparator

Add-on to Metformin: Improved Glycemic Control With Fewer GI Side Effects HbA 1c Reduction From Baseline % GI Disorders Change in HbA1c (%).2. -.2 -.4 -.6 -.8-1. -.51 -.88.23 % in GI Disorders 2 15 1 5 9.6 14.8 18.2 Vilda 5 mg qd + Met (n=143) Vilda 5 mg bid + Met (n=143) PBO + Met (n=13) 46

Add-on to SU: Hypoglycemic Events Event Vilda 5 mg qd + Glim (n=17) Vilda 5 mg bid + Glim (n=169) PBO + Glim (n=176) % Pts with >1 hypo 1.2 3.6.6 % Pts who D/C due to hypo Total no. of hypo 3 11 1 Severity Grade 1 (n) Suspected Grade 2 (n) 3 11 1 Vilda=vildagliptin; PBO=placebo; D/C=discontinued Safety population, 6 events occurred in the same patient Data on file, Novartis Pharmaceuticals, LAF237A235 47

Add-On to Pioglitazone: Hypoglycemic Events Event Vilda 5mg qd + Pio 45mg N=146 Vilda 5mg bid + Pio 45mg N=158 Placebo + Pio 45mg N=158 % Pts with >1 hypo.6 1.9 % Pts who D/C due to hypo Total no. of hypo 2 3 Severity Grade 1 (n) 2 3 Vilda=vildagliptin; PBO=placebo; D/C=discontinued; Pio=pioglitazone; Pts=patients; Hypo=hypoglycemic event Safety population 48 Data on file, Novartis Pharmaceuticals, LAF237A234

No Additional Weight Gain in Initial Combination with Pioglitazone Mean body weight change from baseline Change in body weight (kg) 3. 2.5 2. 1.5 1..5. Vilda 1 mg qd (n=15) Vilda 1 mg qd + Pio 3 mg qd (n=146) Pio 3 mg qd (n=157) Vilda 5 mg qd + Pio 15 mg qd (n=139) NS 2.1 1.5 1.4.2 BL (kg) 82 81 8 82 Vilda=vildagliptin; Pio=pioglitazone ITT population (intention-to-treat) NS: Weight change not statistically significant vs Pio 3 mg Data 49on file, Novartis Pharmaceuticals, LAF237A2355

Add-on to Insulin: Less Hypoglycemic Events No. of Patients No. of Events No. of Severe Events Vilda + insulin 5 2 PBO + insulin Number of patients 4 3 2 1 33 45 Number of events 16 12 8 4 113 185 Number of severe events 1 8 6 4 2 6 P<.1; P<.5 between groups Incidence of hypoglycemia: fewer events (113 vs 185, P <.1) and fewer patients affected (22.9% vs 29.6%) Severe, grade 2 events : none with vildagliptin add-on to insulin vs 6 events with PBO add-on to insulin (P <.1) 5

Conclusion The inhibition of DPP-4 enhances GLP-1 activities ; elevating insulin levels suppressing glucagon levels A DPP-4 inhibitor, Vildagliptin, normalizes hyperglycemic conditions without inducing AE and hypoglycemia. Combination of Vildagliptin with other anti-diabetic agents shows synergistic effects to further normalize hyperglycemia with high safety and efficacy. 51

Abbreviations CNS = central nervous system DPP-4 = dipeptidyl peptidase-4 GI = gastrointestinal GIP = glucose-dependent insulinotropic peptide GLP-1 = glucagon-like peptide-1 HbA 1c = hemoglobin A 1c IV = intravenous m/z = mass:charge ratio OGTT = oral glucose tolerance test qd = once daily t 1/2 = half-life T2DM = type 2 diabetes mellitus 52