PREVENTION IN CANCER. Massimo Bonucci M.D. SURGICAL PATHOLOGIST MEDICAL ONCOLOGIST SAN FELICIANO HOSPITAL- ROMA
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1 AMWC ANTI-AGING MEDICINE WORLD CONGRESS MONTECARLO- MARCH PREVENTION IN CANCER ORTHOMOLECULAR AND GENE-REPAIRING DR. NIEPER S THERAPY. Massimo Bonucci M.D. SURGICAL PATHOLOGIST MEDICAL ONCOLOGIST SAN FELICIANO HOSPITAL- ROMA
2 AMWC ANTI-AGING MEDICINE WORLD CONGRESS - MONTECARLO- MARCH DR. NIEPER ONCOLOGIST PRESIDENT OF GERMAN SOCIETY OF ONCOLOGY CHIEF DEPARTMENT OF MEDICIN PARACELSUS KLINIK - HANNOVER
3 AMWC ANTI-AGING MEDICINE WORLD CONGRESS MONTECARLO- MARCH DR. NIEPER CARDIOVASCULAR DISEASE CANCER MULTIPLE SCEROSIS
4 AMWC ANTI-AGING MEDICINE WORLD CONGRESS - MONTECARLO- MARCH CANCER INCIDENCE CANCER hit on the world about 9 million peaple every year. In Italy are hit about peaple The cancer hits every age, but is more fequent with proceeding of age
5 AMWC ANTI-AGING MEDICINE WORLD CONGRESS - MONTECARLO- MARCH CANCER EVOLUTION Cancer is a complex multisteps process and requires a lot of cause and stage. INSIDE and OUTSIDE Factors act before damages has been clinically diagnosed. Those Factors acting sequencially or simultaneously It is called CARCINOGEN the agent (chemical, physical, hormonal, environmental, pharmacological) that is able to modify a normal cell into cancer cell. Phases of change: Initiation (quick passage without return- damage on DNA)-Promotion (slow process, repeative, reversible and influenced of environment. Progression(cancer cells acquire biological characters of invasion,metastases, immortality.
6 AMWC ANTI-AGING MEDICINE WORLD CONGRESS - MONTECARLO- MARCH CANCER EVOLUTION The final result is precancerous lesion (hyperplasia, dysplasia). They are circumscribed lesions, they rise also year before the cancer (es. polyp of colon) The destiny is influenced from environmental factors. They can spontaneously to regress, to stay stationary for long times The dramatic event is a malignant evolution
7 CAUSES of CANCER REPLICATION INCREASE CELLULAR PERMEABILITY INCREASE FACTORS OF RECEPTOR EXSPRESSION CHANGE ENZYME/GENS REGOLATORS (p53,p21,mdm2) IMMUNE SYSTEM SUPPRESSION DIRECT ( TO CANCER CELLS) INDIRECT (TO FREE RADICALS) INCREASE IL10 WITH REDUCTION T CELL HELPER
8 IMMUNE SYSTEM Lack of foreign cell antigen Weak immune reply (difect amount) Blocked serum factors produced to cancer Spontaneous cancers are antigen devoid
9 AMWC ANTI-AGING MEDICINE WORLD CONGRESS - MONTECARLO- MARCH VIRUS HPV (16,18,31,33,45,53) HSV CMV EBV
10 AMWC ANTI-AGING MEDICINE WORLD CONGRESS - MONTECARLO -MARCH NEOPLASTIC CHANCE environmental factors DNA damage Immune System Cell precancerous lesion cancer cell
11 Cancer therapy objectives To active body s process defence To reduce cancer cells
12 AMWC ANTI-AGING MEDICINE WORLD CONGRESS - MONTECARLO- MARCH Orthodox chemotherapy: chemical substances synthesized on laboratory Extract of plants (taxanes) Prodocts of bacteria or fungy (antracyclins) Alchilants substances (nitrosuree) Composts of minerals (platinum) Hormons
13 AMWC ANTI-AGING MEDICINE WORLD CONGRESS - MONTECARLO -MARCH ORTHOMOLECULAR THERAPY Natural substances not toxics that work to balance body chemistry, with low stress impact on patient and our immune system Natural substances that need months or years end up illness without side effect to chemotherapies agents.
14 Gene-repairing substances Acetaldehyde Benzaldehyde Didrovaltrate Extracts of Carnivorous plant Iridodials Oncostatins, plombagine Squalen Tumusterons
15 Aldehyds Acetaldehyd, Benzaldehyd, Iridodyal Donors mono or di-aldeyds Re-differentiation genetic Block and arrest tumor growth Enhancing immune system
16 Carnivorous Plant Extracts Enzymes (endo peptidase e nuclease, plombagina, droseron, idrossidroseron) Eliminate injured or cancer cells Efficient on viruses infected cells or injured to radiations Destroy the nuclei acids
17 Squalene (Shark liver oil) Triterpenoid enzyme ( shark extract liver oil) Increase carcinogen-blocking hormone (DHEA) Reduces growth blood vessels Enhancing antiviral resistance
18 Tumosterons Steroids weak, producted to Timosteryn made to activated linfocytes (NK) Go into cancer cells and block replication Repaire or inactive gens Block cellular growth from lipids Actived from timic factors (timosin) and Squalene, Vit. C, Vit. D2, prednison
19 Beta-carotene Antioxidant, reduction free radicals Activation gh. Thymic Activation linfocyts (T Helper) Block T suppressor
20 DHEA Steroid product to gh. adrenal Hight concentration in the blood Inhibit G6PDH Prevent cancerization for oncogens virus Antioxidant, reduce free radicals Blocking genetic action from: tobacco, UV, radiations, heavy metals, alcool, fats
21 AMWC ANTI-AGING MEDICINE WORLD CONGRESS MONTECARLO - MARCH DECREASE OF CANCER INCIDENCE To decrease carcinogens exposition. (primary prevention) To discover genetics damages and precancerous lesion. (secondary prevention)
22 PREVENTION To use natural agents, medicines, foods and supplements to prevent illness (named also chemoprophilaxis) Is better to call of risk reduction instead prevention in respect of cancer Protection is done with substances that to reduce the risk of cancer
23 PROTECTIVE AGENTS ANTIOXIDANTS - BETA-CAROTENE - ALFA-TOCOFEROLS - N. ACETIL CISTEINA - ANTI-INFLAMMATORY (BLOCK OF COX-2) - NIMESULIDE - PIROXICAN
24 PROTECTIVE AGENTS VIT. A - CELLULAR DIFFERENTIATION - TO EXITE IMMUNE SYSTEM - INHIBITION CELLS TRASFORMATION - INHIBITION CANCER PROMOTION - INDUCTION APOPTOSIS (DEATH CELL) VIT. E - CELLULAR DIFFERENTIATION - INHIBITION CANCER GROWTH - INDUCTION APOPTOSIS (DEATH CELL)
25 PROTECTIVE AGENTS BETA-CAROTENE - REMISSION OF PRENEOP. LES. - TO EXITE IMMUNE SYSTEM INOSITOL - REGULATION P53 E WAF1 - ACTIVITY OF T. NAT. KILLER N.A.CISTEINA - RESTORE TO P53 - RIDUCTION ANGIOGENESIS
26 SECONDARY PREVENTION To use rich foods on protective elements BETA-CAROTENE (coloured vegetables) VIT. C; VIT.E; VIT. A (fresch or dried fruit) CO-Enzyme Q10, OMEGA-3 (fish, soie) N.A.C. (dried fruit) INOSITOL (legumes, soie) MINERALS (dried fruit, fish, soie)
27 Cases patients only cancer one/more mts (30%) (70%) The study valued time to progression ( months) and wellness to complementary therapy (Nieper s therapy)
28 Cases patients (no mts at the beginning of the study) Treated with complementary therapy with or without chemotherapy All patients are alive and illnees free
29 Cases patients (with more mts at beginning study) 8-12 (month) (month) > 25 (month) 79 (15%) 154 (45%) 143 (36%) 55 (15%) are today alive
30 Cases All patients submitted to complementary therapy: Improvement of wellness Decrease side effects (nausea, sick, bone pains) Reduction blood stimulating Only 5% delay to chemotherapy
31 Conclusions Complementary therapy increase compliance with reduction side effects Complementary therapy is good for prevention and protection for release Complementary therapy increase expected lifetime
32 Good-bye from ROME
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