Case Number 5 Anton s Syndrome
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1 Case Number 5 Anton s Syndrome Luke Portelli Reviewed by: Dr. Pierre Ellul Case Summary: Demographic details: Mr. AS, male, Nadur Referred from: Gozo General Hospital 57-year-old male admitted to ITU with a severe Community Acquired Pneumonia. Stayed in ITU for about three weeks and during this period suffered an episode of deterioration which was attributed to multiple venous infarcts of the brain. This caused the patient to become blind. Presenting complaint: Sudden onset blindness. History of presenting complaint: The blindness occurred suddenly and with no warning. Patient became increasingly disoriented and was clearly blind although adamantly kept saying that he was able to see normally. At the time the patient had been in ITU for about three weeks. Past medical and surgical history: Past medical history: Severe Community Acquired Pneumonia lasting three weeks, exactly preceding the blindness. Patient had to be admitted to ITU due to the severity of the pneumonia. Mild Hypertension. Past surgical history: Patient did not have any major operations. Drug history: Drug Dosage Frequency Type Reason Tazocin Piperacillin 4g TDS Antibiotic Treatment for pneumonia Piperacillin/Tazobactam Tazobactam 0.5g Levofloxacin 500mg Dly Antibiotic Treatment for pneumonia Doxycycline 100mg BD Antibiotic Treatment for pneumonia Antibiotics were later switched due to the possibility of a viral pneumonia. 34
2 Drug Dosage Frequency Type Reason Ganciclovir 300mg - IV BD Antiviral Treatment for CMV pneumonia Patient received IV ganciclovir for 12 days and was later switched to oral valganciclovir. Family history: The patient did not have any major illnesses in the family. His father had hypertension, as did his uncle but other than that nil of note. Social history: Patient used to smoke cigarettes and drink alcohol on occasions. Patient did not divulge the actual amount of cigarettes and alcohol he consumed. Systemic inquiry: General Health: patient looks very thin and showed cachexia due to the long stay in ITU. Cardiovascular System: relatively normal except for mild hypertension. Respiratory System: patient was still short of breath even after the pneumonia was resolving. Patient also had a cough and was on occasions bringing up sputum. Gastrointestinal System: relatively normal. Patient had a NG tube. Genitourinary System: urine output was normal and not painful. Central Nervous System: normal. Musculoskeletal System: very weak due to the cachexia. Patient was unable to stand by himself. Endocrine System: normal. Others: pupillary light reflex and fundoscopy were normal. Current therapy: After the period in ITU and the occurrence of the blindness, the patient recovered considerably. However he was still given the antivirals for complete elimination of the CMV infection. Discussion of results of general and specific examination: The pupillary light reflex of the patient was normal, suggesting that his anterior visual pathway was intact and functioning normally. This indicated that the blindness was arising from a problem in the posterior visual pathway or maybe the occipital lobe. Patient was clearly blind however he insisted that he was seeing things normally. After some days he began to recognise some shades of color but other than that he remained blind. General examination was normal. Chest was clear suggesting that the patient was recovering from the pneumonia. Differential diagnosis: Regarding the pneumonia, at first it was thought that it was bacterial in nature, however diagnosis changed to CMV infection since CMV was detected by PCR. The blindness was probably caused by an injury to the brain during the patient s stay at ITU. Due to the complexity of the symptoms and their relative rarity, the diagnosis was still unknown. 35
3 Diagnostic procedures: Laboratory Exams: Test: Cell pathology. Justification: Exclusion of malignancies. Result: Scattered bronchial epithelial cells, many of which exhibited hyperplastic changes and few macrophages are present on a background of erythrocytes. Conclusion: No malignant cells seen. Instrumental Exams: Test: CT Thorax. Justification: Detection of pneumonia. Results: Inflammatory changes of the lung are noted bilaterally basally and in the left upper lobe. Some pleural effusion is noted but there is no abscess formation. Lymphadenopathy is seen but there was no evidence of malignancy. Conclusion: Pneumonia is confirmed. Test: CT Brain. Justification: Finding the cause of blindness. Result: Multiple hypo dense zones of the brain are noted bilaterally. Conclusion: These changes are most probably due to a post-infarctive process. Test: MRI Head. Justification: Finding the cause of blindness. Result: Multiple bilateral abnormal cerebral foci. These appearances are suggestive of multifocal haemorrhages infarcts with associated haemorrhages changes, worse in the occipital regions bilaterally. Normal appearance of the Circle of Willis. No evidence of aneurysm and no evidence of venous sinus thrombosis. Conclusion: Multifocal bilateral haemorrhagic infarcts. Therapy: Drugs: Drug Name Dosage Frequency Type Reason Lactulose 10mls Dly Synthetic, non-digestible sugar Treatment of constipation Omeprazole 40mg Dly PPI Upset stomach Patient received the last dose of oral anti-viral on the day of discharge. Diagnosis: Due to the pupillary light reflex being normal, it was concluded that the problem did not arise from the anterior visual pathway, as most cases of blindness do. The CT scans and MRI showed infarcts in the occipital lobe and this suggested that the problem was arising from the occipital cortex itself. Cortical blindness is defined as visual inability in the presence of normal light reflexes and normal fundoscopy 1. This observation led to the diagnosis of Anton s syndrome, which is a very rare case of blindness. This syndrome is characterised by a phenomenon called confabulation 1. Patients suffering from Anton s syndrome adamantly say that they can see when they obviously cannot and also speak as if they had normal vision 2. This denial of vision problems is called visual anosognosia 2. 36
4 Cortical blindness was first described in detail by Gabriel Anton, who was an Austrian neurologist. This is why this syndrome now bears his name 3. Final Treatment and Follow ups: The treatment in this case is very limited. Follow-ups included visits from speech language pathologists, physiotherapy teams and occupational therapists. The patient was suggested to have a repeat CT Thorax, two weeks after the first one and to be regularly followed up at the stroke clinic. 37
5 Fact Box 5: Title: Anton s Syndrome Anton s Syndrome is a very rare type of brain damage that occurs in the occipital lobe. People with the condition suffer from cortical blindness. Risk Factors: Since Anton s Syndrome is technically a stroke, its risk factors include: Hypertension Diabetes Smoking Alcohol consumption Prolonged periods of inactivity Symptoms: Blindness (although the patient denies his blindness) Headaches Confusion. Signs: A very specific sign of Anton s Syndrome is confabulation. Confabulation is a way by which the patient denies his blindness, walking and trying to move as he still sees normally. Confabulation is made very clear when the patient stars to describe objects and people in his vicinity that are in fact not there. Prevention: This consists mainly in preventing cerebrovascular disease which include a healthy life style, controlled blood pressure, no smoking and alcohol consumption and others. References: 1. Rickards C and Shepherd DI. Cortical blindness in a 35-year-old man. Postgrad Med J. 1996; 72(846): Maddula M, Lutton S and Keegan B. Anton s syndrome due to cerebrovascular disease: a case report. Journal of Medical Case Reports. 2009; 3: Kondziella D and Frahm-Falkenberg S. Anton s Syndrome and Eugenics. J Clin Neurol, 2011; 7:
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Dear You are scheduled for an appointment with Dr. Manoj Kohli at Christie Clinic in the Department of Rheumatology on at. Please check in on the first floor. The office is located on the 2 nd floor of
Notto Chiropractic Health Center Patient Information
Notto Chiropractic Health Center Patient Information Acct #: Name: Preferred Name: Address: City: State: Zip: Home Phone: ( ) - _. Work Phone: ( ) -. Who Referred You? In Case of Emergency: Phone Number:
HEADACHES THE RED FLAGS
HEADACHES THE RED FLAGS FAYYAZ AHMED CONSULTANT NEUROLOGIST HON. SENIOR LECTURER HULL YORK MEDICAL SCHOOL SECONDARY VS PRIMARY HEADACHES COMMON SECONDARY HEADACHES UNCOMMON BUT SERIOUS SECONDARY HEADACHES
Laser Vein Center Thomas Wright MD Page 1 of 4
Demographics Laser Vein Center Thomas Wright MD Page 1 of 4 Patient Name: Address: City, St, Zip Primary Phone: Alternate: DOB: Social Security #: Insurance Information Primary Insurance ID# Group# Subscriber
DOB: / / Please list the names and telephone numbers of the other physicians involved in your care: Name Specialty Phone Address Receive Report (Y/N)
Medical History: Patient: DOB: / / Please list the names and telephone numbers of the other physicians involved in your care: Name Specialty Phone Address Receive Report (Y/N) List the names of prescription
MEDICAL ASSESSMENT PART 1 - SOCIAL HISTORY
Smoking history Alcohol history Never Quit Never Quit PART 2 - MEDICAL HISTORY Date of last colonoscopy? Date of last mammogram? Date of last pap smear? Date of last flu vaccine? Date of last pneumonia
Cell Phone #: Home Phone #: ** Address (prefer your forever address):
NEW PATIENT QUESTIONNAIRE * Some of this information is required by the CMS (Centers for Medicare and Medicaid Services). Your demographic answers will never affect your care. Today s Date: **Date of Birth:
Past Surgical History
Name: DOB: Check All That Apply Past Medical History o Anemia o Aneurysm o Asthma o Bipolar o Bleeding Disorder o Blood Clot o Brain Tumor o Bronchitis o Cancer o Crohn s Disease/Ulcerative Colitis o Depression
NEW PATIENT VISIT QUESTIONNAIRE
HeartHealth A Program of the Dalio Institute of Cardiovascular Imaging NEW PATIENT VISIT QUESTIONNAIRE Name: Date of Birth: / / Address: City: State: Zip: Home Phone #: Work Phone #: Cell #: Email: Preferred