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24-Month Analysis of Change From Baseline In Clinical Dementia Rating Scale Cognitive and Functional Domains in PRIME, A Randomized Phase b Study of the Anti-Amyloid Beta Monoclonal Antibody Aducanumab Philipp von Rosenstiel, MD, Carmen Castrillo-Viguera, MD, Sarah Gheuens, MD, PhD, Tianle Chen, PhD, John O Gorman, PhD, Ping Chiao, PhD 2, Guanfang Wang, PhD 3, Christian von Hehn, MD, PhD, LeAnne Skordos, PharmD, Christoph Hock, MD 4, Roger M Nitsch, MD 4, Samantha Budd Haeberlein, PhD, Alfred Sandrock, MD, PhD Biogen, Cambridge, Massachusetts, USA; 2 Former employee of Biogen, Cambridge, Massachusetts, USA; 3 Cytel, Cambridge, Massachusetts, USA; 4 Neurimmune, Schlieren-Zurich, and University of Zurich, Switzerland AAIC 28, Chicago, Illinois, US July 22, 28

Disclaimer The following materials, which have been posted in the Medical and Scientific Conferences Presentations IR section of the Biogen website, are provided for informational purposes to the investor community only and may include information about investigational compounds that have not been approved by the applicable regulatory authorities as safe and effective. 2

PHOTO, VIDEO AND AUDIO POLICY Photo, video and audio recordings are prohibited in this presentation.

Disclosures This study is funded by Biogen a SBH, CCV, SG, TC, JO, LS, CvH, PvR, and AS are employees and shareholders of Biogen PC was an employee and shareholder of Biogen at the time of this work GW is an employee of Cytel CH and RMN are employees and shareholders of Neurimmune Biogen licensed the worldwide rights to aducanumab from Neurimmune Holding AG in 27 and is responsible for its development and the commercialization As of October 22, 27, Biogen and Eisai are collaborating on the development and commercialization of aducanumab globally a Medical writing support and editing for this presentation was funded by Biogen and was provided by Nucleus Global. 4

Overview Aducanumab is a human monoclonal antibody that binds to both soluble and insoluble aggregated forms of Aβ, including oligomers, protofibrils, and fibrils PRIME is an ongoing Phase b study assessing the safety, tolerability, PK and PD of aducanumab in patients with prodromal or mild Alzheimer s disease In PRIME, treatment with aducanumab resulted in a time and dose dependent removal of plaques from the brain as shown by positron emission tomography (PET) standardized uptake value ratio Patients with early Alzheimer s disease enrolled in PRIME experienced a sustained delay in the disease progression as measured by exploratory clinical endpoints at 2, 24 and 36 months -3 The primary endpoint in the PRIME LTE is safety/tolerability Exploratory endpoints include: o o Changes in amyloid PET Measures of clinical decline on the CDR-SB and MMSE A post hoc analysis was conducted on the fixed-dose and titration cohorts that assessed cognitive and functional domains of the CDR scale over 24 months, including 2 months from the placebo-controlled period and 2 months from the LTE. Sevigny et al. Nature. 26;537:5 56; 2. Viglietta V, et al. J Prev Alz Dis 26;3:284. Data presented at CTAD 26; 3. Budd Haeberlein S, et al. J Prev Alz Dis 27;4:33. Data presented at CTAD 27. CDR, Clinical Dementia Rating scale; CDR-SB, Clinical Dementia Rating-Sum of Boxes; MMSE, Mini-Mental State Examination; PD, pharmacodynamics; PET, positron emission tomography; PK, pharmacokinetics; LTE, long-term extension. 5

PRIME Study Design: Placebo-Controlled and LTE Periods Placebo-controlled period mg/kg 3 mg/kg Placebo mg/kg Placebo 6 mg/kg Placebo LTE period All patients receive aducanumab 3 mg/kg 3 mg/kg Aducanumab 3 mg/kg or titration (3 6 mg/kg) a mg/kg Aducanumab titration (3 6 mg/kg) a 6 mg/kg Aducanumab titration (3 6 mg/kg) a Titration (ApoE ε4 carriers; mg/kg) Placebo (ApoE ε4 carriers) mg/kg Aducanumab titration (ApoE ε4 carriers; mg/kg) b Randomization: 3: active: placebo within cohorts, fixed-dose cohorts stratified by ApoE ε4 status Patients randomized to placebo in the placebo-controlled period were switched to aducanumab 3 mg/kg or a titration regimen in the LTE ( placebo switchers ). Patients randomized to aducanumab 3, 6, or mg/kg or titration in the placebo-controlled period were assigned to continue in the same dose group in the LTE ( continuers ) a Titration denotes 2 doses of 3 mg/kg followed by subsequent doses of 6 mg/kg. b Titration denotes 2 doses of mg/kg, 4 doses of 3 mg/kg, 5 doses of 6 mg/kg followed by subsequent doses of mg/kg. ApoE ε4, Apolipoprotein E ε4; LTE, long-term extension. 6

Titration Dosing Regimen Placebo-controlled period (Months -2) LTE period (Months 2-24) Study week: 4 8 2 6 2 24 28 32 36 4 44 48 52 Titration mg/kg Dose (mg/kg): Placebo 3 3 3 3 6 6 6 6 6 mg/kg mg/kg 3 mg/kg 6 mg/kg mg/kg By Week 24 average expected dose = 2.9 mg/kg By Week 52 average expected dose = 5.3 mg/kg By Week average expected dose = 7.6 mg/kg 7

Timeline of PET and Clinical Assessments in PRIME IV infusions every 4 weeks for 52 weeks (4 total) active or placebo IV infusions every 4 weeks for up to 6 years All patients received active treatment Week Screening Placebo controlled period LTE (Year ) LTE (Year 2) 24-26 52-54 76-78 8-32-34 64-66 LTE Years 3-6 Amyloid PET Clinical Tests PET, positron emission tomography. 8

Patient Disposition at 24 Months 97 randomized (96 dosed) Dosed in placebocontrolled period Completed treatment in the placebo-controlled period Pooled placebo 48 mg/kg 3 3 mg/kg 32 6 mg/kg 3 38 24 26 25 2 Switchers mg/kg 32 Continuers Titration 23 9 Dosed in the LTE 37 9 26 24 9 8 Discontinued treatment in the first year of the LTE AE Death Disease progression Other 8 7 4 4 8 2 6 2 4 3 2 Analysis of data up to Month 24. AE, adverse event; LTE, long-term extension. 9

Baseline Disease Characteristics Aducanumab Placebo (n=48) mg/kg (n=3) 3 mg/kg (n=32) 6 mg/kg (n=3) mg/kg (n=32) Titration (n=23) Age in years, mean ± SD 73.3 ± 6.8 72.6 ± 7.8 7.5 ± 8.2 73.3 ± 9.3 73.7 ± 8.3 73. ± 7.8 ApoE ε4, n (%) Carriers Non-carriers 34 (7) 4 (29) 9 (6) 2 (39) 2 (66) (34) 2 (7) 9 (3) 2 (63) 2 (38) 23 () Clinical stage, n (%) Prodromal Mild 22 (46) 26 (54) (32) 2 (68) 4 (44) 8 (56) 2 (4) 8 (6) 3 (4) 9 (59) 3 (57) (43) MMSE, mean ± SD 24.7 ± 3.6 23.6 ± 3.3 23.2 ± 4.2 24.4 ± 2.9 24.8 ± 3. 24.7 ± 3. CDR Global Score, n (%).5 4 (83) 8 (7) 22 (7) 9 (29) 22 (69) (3) 25 (83) 5 (7) 24 (75) 8 (25) 8 (78) 5 (22) CDR-SB, mean ± SD 2.69 ±.54 3.4 ±.76 3.5 ± 2.6 3.32 ±.54 3.4 ±.7 3.24 ±.84 PET SUVR, mean composite.435.44.464.429.44.325 AD medications used, a n (%) 32 (67) 2 (68) 28 (88) 2 (67) 7 (53) 2 (52) a Cholinesterase inhibitors and/or memantine. AD, Alzheimer s disease; ApoE ε4, Apolipoprotein E ε4; CDR-SB, Clinical Dementia Rating Sum of Boxes; MMSE, Mini-Mental State Examination; PET, positron emission tomography; SD, standard deviation; SUVR, standardized uptake value ratio.

MMSE CDR-SB Summary of Previously Presented PRIME 24-Month Interim Results Clinical effect of aducanumab (exploratory endpoints) Amyloid plaque reduction with aducanumab Safety (primary endpoint) Of the 85 patients dosed with aducanumab since the start of the PRIME study, 46 patients experienced ARIA-E Of the 46 patients who experienced ARIA-E, 65% were asymptomatic and 35% were symptomatic; the majority of symptomatic cases experienced symptoms that were mild to moderate in severity 6 patients experienced more than one episode of ARIA The majority of ARIA events occurred early in the course of treatment; they were typically mild, asymptomatic, and resolved or stabilized within 4-2 weeks, with most patients continuing treatment a Placebo switchers received aducanumab 3 mg/kg or titration (3 mg/kg 6 mg/kg or mg/kg mg/kg) in the LTE. Nominal * P<.5; Nominal ** P<.; Nominal *** P<. vs placebo in the placebo-controlled period and vs placebo switchers in the LTE period. MMSE and CDR-SB are exploratory endpoints. Results based on MMRM, fitted with change from baseline as a dependent variable, and included fixed effects for categorical treatment, categorical visit and treatmentby-visit interaction, continuous baseline value, and laboratory ApoE ε4 status (carrier and non-carrier). ARIA-E, amyloid-related imaging abnormalities - vasogenic edema; CDR-SB, Clinical Dementia Rating Sum of Boxes; LTE, long-term extension; MMRM, mixed model for repeated measures; MMSE, Mini Mental State Exam; SE, standard error.

Clinical Dementia Rating (CDR) Scale Domains The CDR-SB is a validated global assessment of Alzheimer s disease disability that is capable of tracking the progression of Alzheimer s disease in the MCI and mild Alzheimer s disease population It assesses a patient s level of impairment on 6 domains using semi-structured interviews by a trained clinician/rater with the patient and an informant Functional domains Community Affairs Home and Hobbies Personal Care Cognitive domains Memory Orientation Judgement and Problem Solving Each impairment level of the cognitive and functional domains (except for Personal Care) is rated on a continuum of five levels, with a scale of to 3 o The five levels of impairment are (none),.5 (questionable), (mild), 2 (moderate), and 3 (severe) o Personal Care uses only 4 levels:,, 2, and 3 Morris JC, et al. Neurology. 997;48:58-. 2

Baseline CDR Global and Domain Scores Aducanumab Placebo (n=48) mg/kg (n=3) 3 mg/kg (n=32) 6 mg/kg (n=3) mg/kg (n=32) Titration (n=23) CDR-SB, mean ± SD 2.69 ±.54 3.4 ±.76 3.5 ± 2.6 3.32 ±.54 3.4 ±.7 3.24 ±.84 CDR cognitive domain score, mean ± SD CDR functional domain score, mean ± SD.79 ±.87 2. ±.94 2.2 ±.2 2.3 ±.67 2.3 ±.3 2.4 ±.94.9 ±.83.4 ±.9.3 ±.2.28 ±.6. ±.9.2 ±.5 CDR-SB, Clinical Dementia Rating Sum of Boxes; SD, standard deviation. 3

Week 54: Effect of Aducanumab on Both CDR Cognitive and Functional Domains CDR Cognitive Domain (Week 54) CDR Functional Domain (Week 54) Aducanumab dose Favors aducanumab vs placebo Aducanumab dose Favors aducanumab vs placebo mg/kg (n=23) mg/kg (n=23) 3 mg/kg (n=27) 3 mg/kg (n=27) 6 mg/kg (n=26) 6 mg/kg (n=26) mg/kg (n=23) mg/kg (n=23) Titration (n=2) Titration (n=2) -2.5-2 -.5 - -.5.5.5-2.5-2 -.5 - -.5.5.5 Difference vs Placebo, Adjusted Mean [95%CI] Difference vs Placebo, Adjusted Mean [95%CI] Results based on MMRM, fitted with change from baseline as a dependent variable, and included fixed effects for categorical treatment, categorical visit and treatment-by-visit interaction, continuous baseline value, and laboratory ApoE ε4 status (carrier and non-carrier). CDR, Clinical Dementia Rating; CI, confidence interval; MMRM, mixed model for repeated measures. 4

Week : Effect of Aducanumab on Both CDR Cognitive and Functional Domains CDR Cognitive Domain (Week ) CDR Functional Domain (Week ) Aducanumab dose Favors aducanumab vs placebo switchers Aducanumab dose Favors aducanumab vs placebo switchers 3 mg/kg (n=4) 3 mg/kg (n=4) 3 mg/kg (n=6) 3 mg/kg (n=6) 6 mg/kg (n=23) 6 mg/kg (n=23) mg/kg (n=5) mg/kg (n=5) Titration (n=6) Titration (n=6) -2.5-2 -.5 - -.5.5.5-2.5-2 -.5 - -.5.5.5 Difference vs Placebo Switchers a, Adjusted Mean [95%CI] Difference vs Placebo Switchers a, Adjusted Mean [95%CI] a Placebo switchers received aducanumab 3 mg/kg or titration (3 mg/kg 6 mg/kg or mg/kg mg/kg) in the LTE. Results based on MMRM, fitted with change from baseline as a dependent variable, and included fixed effects for categorical treatment, categorical visit and treatment-by-visit interaction, continuous baseline value, and laboratory ApoE ε4 status (carrier and non-carrier). CDR, Clinical Dementia Rating; CI, confidence interval; MMRM, mixed model for repeated measures. 5

Summary Analyses of exploratory clinical endpoint CDR-SB suggest a continued benefit on the rate of clinical decline over 24 months Post hoc analysis suggests a beneficial effect of aducanumab on progression of both cognitive and functional CDR domain scores over 24 months These data continue to support further investigation of the clinical efficacy and safety of aducanumab in patients with early Alzheimer s disease in the ENGAGE and EMERGE Phase 3 trials CDR-SB, Clinical Dementia Rating Sum of Boxes; LTE, long-term extension; MMSE, Mini-Mental State Examination. 6

Acknowledgments We thank all the patients and their family members participating in the aducanumab studies, as well as the investigators and their staff conducting these studies. 7