NutraHacker. Complete Gene Mutation Report for Customer: 1428d57e bc-8bfa-fa3c85dda19b. Instructions:
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1 NutraHacker Complete Gene Mutation Report for Customer: 1428d57e bc-8bfa-fa3c85dda19b Instructions: NutraHacker reports mutations (single nucleotide polymorphisms) in this uploaded genome. Genes not reported in this report are either normal, not actionable, or not currently detected by NutraHacker. The expected allele is the one seen in a normally functioning gene. The high risk alleles reported are the ones measured from the uploaded genome. NutraHacker reports the effects of these mutations as discovered by published empirical data and suggests nutritional supplements that can mitigate potential issues caused by these mutations. This report is meant to serve as a guide for nutritional supplementation for the owner of the genome and is not applicable to any other individual. Supplement quantities and dosages are not included as they are indicated on the purchased product. Multiple recommendations for the same supplement does not mean that the dosage should be multiplied. In the case of a conflict (such as a particular vitamin being both encouraged and discouraged), the owner of the genome should assess his/her own personal biology to decide whether to include or discard that particular supplement. NOTICE: State law allows any person to provide nutritional advice or give advice concerning proper nutrition--which is the giving of advice as to the role of food and food ingredients, including dietary supplements. This state law does NOT confer authority to practice medicine or to undertake the diagnosis, prevention, treatment, or cure of any disease, pain, deformity, injury, or physical or mental condition and specifically does not authorize any person other than one who is a licensed health practitioner to state that any product might cure any disease, disorder, or condition. NutraHacker reports are for scientific, educational and nutritional information only and are not intended to diagnose, cure, treat or prevent any disease, disorder or condition. Thank you for using NutraHacker. To your health! Gender of customer: Female A total of 56 mutations were detected at this time for your genome out of the 265 polymorphisms assessed. There were 23 homozygous mutations. There were 33 heterozygous mutations.
2 Please continue to the next page to begin your discovery process.
3 Detoxification rs CYP1A1 C AC: 1/ % Phase I xenobiotic metabolism, Reduced function of enzyme, Diindolylmethane PAH's, metabolize E2 to 2-hydroxyestradiol effects vary with race Detoxification rs CYP1A2 C AA: 2/2 N/A Hydroxylation or dealkylation of xenobiotics, Phase I, metabolize E2 to 2-hydroxyestradiol CYP1A2*11 allele with approximately 5% activity of that of the CYP1A2 wild type Induce with broccoli, Cabbage, Diindolylmethane, Glucarate, NAC, Cardamom, Sulforaphane Detoxification rs CYP1A2 A AC: 1/ % Hydroxylation or dealkylation of Slow to metabolize caffeine, Main Induce with broccoli, xenobiotics, Phase I, metabolize liver pathway Cabbage, E2 to 2-hydroxyestradiol Diindolylmethane, Glucarate, NAC, Cardamom, Sulforaphane Detoxification rs CYP1B1 A CT: 1/ % 4-hydroxylation of estrogen Probable increased enzyme Diindolylmethane function, increased deleterious estrogen metabolism and activation of pro-carcinogens Detoxification rs CYP2D6 C AG: 1/ % Detoxifies 20% of prescription Poor metabolizer drugs Detoxification rs CYP2D6 G CT: 1/ % Detoxifies 20% of prescription CYP2D6*4 - nonfunctioning drugs variant; the most common variant Detoxification rs1208 NAT2 A AG: 1/ % This gene encodes an enzyme Fast metabolizer NAC, Vitamin B2, that functions to both activate and Vitamin B3, Vitamin deactivate arylamine and B5, Molybdenum hydrazine drugs and carcinogens. Detoxification rs NAT2 C CT: 1/2 N/A This gene encodes an enzyme Decreased activity NAC, Vitamin B2, that functions to both activate and Vitamin B3, Vitamin deactivate arylamine and B5, Molybdenum hydrazine drugs and carcinogens. Curcumin, Cumin, Grapefruit Curcumin, Cumin, Grapefruit Substrates of this enzyme Substrates of this enzyme
4 Detoxification rs NAT2 T CT: 1/ % This gene encodes an enzyme that functions to both activate and deactivate arylamine and hydrazine drugs and carcinogens. Neurotransmitter rs CHRNA5 T GG: 2/ % Neuronal acetylcholine receptor subunit alpha-5 Neurotransmitter rs4633 COMT C TT: 2/ % Degrades catecholamines, Phase II, inactivates hydroxy-estrogens Neurotransmitter rs COMT G TT: 2/ % Degrades catecholamines, Phase II, inactivates hydroxy-estrogens Neurotransmitter rs4680 COMT G AA: 2/ % Degrades catecholamines, Phase II, inactivates hydroxy-estrogens Neurotransmitter rs GAD1 A GG: 2/ % Catalyzes production of GABA from glutamate Neurotransmitter rs GAD1 A CC: 2/ % Catalyzes production of GABA from glutamate Neurotransmitter rs GAD1 C TT: 2/ % Catalyzes production of GABA from glutamate Neurotransmitter rs GAD1 C TT: 2/ % Catalyzes production of GABA from glutamate Neurotransmitter rs MAO-A T CC: 2/2 N/A Oxidizes serotonin, dopamine, epinephrine, norepinephrine Neurotransmitter rs6323 MAO-A G TT: 2/2 N/A Oxidizes serotonin, dopamine, epinephrine, norepinephrine Neurotransmitter rs MAO-B A CT: 1/2 N/A Oxidizes phenethylamine, benzethylamine, dopamine Neurotransmitter rs NTRK2 C TT: 2/ % Neurotrophic tyrosine kinase receptor type 2 Decreased activity NAC, Vitamin B2, Vitamin B3, Vitamin B5, Molybdenum Increased nicotine intake Nicotine Same amino acid sequence, lower expression of gene, less breakdown of catecholamines Decreased COMT activity Slower breakdown dopamine, oestrogen, worrier, prone to anxiety, more sensitive to green tea High glutamate, low GABA High glutamate, low GABA High glutamate, low GABA High glutamate, low GABA Increased expression MAO-A Hydroxy B12 Methyl B12, Methyl (hydroxycobalamin) donors Hydroxy B12 Methyl B12, Methyl (hydroxycobalamin) donors Hydroxy B12 Methyl B12, Methyl (hydroxycobalamin) donors, Cannabis Taurine, Theanine, MSG NAC,Glycine, Vitamin B3 Taurine, Theanine, MSG NAC,Glycine Taurine, Theanine, MSG NAC,Glycine Taurine, Theanine, MSG NAC,Glycine Curcumin Lower expression of MAO A Progesterone Curcumin, Estrogens, Androgens Decreased activity MAO B enzyme Decreased BDNF Theanine, Curcumin, Beta-alanine, Lithium orotate, Phosphatidylserine Quercetin, Other MAOB inhibitors
5 Neurotransmitter rs TH T GG: 2/ % Tyrosine hydroxylase, produces Low dopamine N-acetyl-tyrosine, dopamine from tyrosine Mucuna pruriens (with caution) rs ACAT1 G AG: 1/ % Forms cholesterol esters from 3-ketothiolase deficiency, Probiotics, Vitamin Cholesterol cholesterol depletes B12 B12, Low fat diet rs BHMT08 T CT: 1/ % Methylates homocysteine to Downregulation Phosphatidylcholine, methionine TMG, Phosphatidylserine, Zinc rs CBS G AG: 1/ % Adds l-serine to homocysteine to Upregulation, high taurine, high Ornithine/Arginine, Methyl donors, produce l-cystathionine ammonia, high sulfates, decrease Manganese, Vitamin B6 (P-5-P in glutatione synthesis Molybdenum, Zinc, form ok), Taurine, SAMe inhibits, CoQ10 Sulfates, BCAA rs CBS G AG: 1/ % Adds l-serine to homocysteine to Increased responsiveness to Vitamin B6 produce l-cystathionine homocysteine-lowering effects of folic acid. Marginally increased disposal of homocysteine. rs DHFR T CT: 1/ % Reduces dihydrofolate to Decreased function of enzyme Reduced forms of Green tea, EGCG tetrahydrofolate folate, Glycine rs GGH C AG: 1/ % This gene catalyzes the hydrolysis May have functional relevance of folylpoly-gamma-glutamates and result in alterations in plasma and homocysteine levels. antifolylpoly-gamma-glutamates by the removal of gamma-linked polyglutamates and glutamate rs MTHFR C AA: 2/ % Converts folic acid to When homozygous it's functioning L-methylfolate, Folinic acid, Folate 5-methyltetrahydrofolate at about 30% of normal, leads to Vitamin B12, high homocysteine, folate Riboflavin for high concentrations lower. blood pressure, Ribo-5-phosphate
6 rs MTRR A AG: 1/ % Methylates, recycles vitamin b12 Poor methylation of Vitamin B12 leading to higher homocysteine Methyl B12, L-methylfolate levels. rs7946 PEMT C CT: 1/ % Converts Fatty liver due to low choline Phosphatidylcholine phosphatidylethanolamine to phosphatidylcholine HPA axis / Endocrine rs ADIPOQ C TT: 2/ % Important adipokine involved in the control of fat metabolism and insulin sensitivity, with direct anti-diabetic, anti-atherogenic and Decreased adiponectin Omega-3 fatty acids like fish oil, Coffee, Leucine, Magnesium, Fiber, Exercise anti-inflammatory activities. HPA axis / Endocrine rs CaSR A AG: 1/ % Calcium sensitive receptor Arg990Gly polymorphism was associated with nephrolithiasis and hypercalciuria in different populations Vitamin K, Magnesium HPA axis / Endocrine rs ESR1 T CC: 2/ % Estrogen receptor alpha Female health affected Diindolylmethane HPA axis / Endocrine rs ESR1 A AG: 1/ % Estrogen receptor alpha Female health affected Diindolylmethane HPA axis / Endocrine rs G6PC2 T CC: 2/ % This gene encodes an enzyme Fasting blood glucose level Chromium, Vanadium belonging to the higher. This is actually the more glucose-6-phosphatase catalytic common form subunit family. These enzymes are part of a multicomponent integral membrane system that catalyzes the hydrolysis of glucose-6-phosphate, the terminal step in gluconeogenic and glycogenolytic pathways, allowing the release of glucose into the bloodstream. The family member encoded by this gene is found in pancreatic islets. HPA axis / Endocrine rs NR3C1 G AA: 2/ % Glucocorticoid receptor Mutation associated with Phosphatidylserine, generalized glucocorticoid Possibly ketogenic resistance, high cortisol, CFS diet Calcium High carb diets
7 HPA axis / Endocrine rs NR3C1 G AA: 2/ % Glucocorticoid receptor Mutation associated with generalized glucocorticoid resistance, high cortisol, CFS Phosphatidylserine, Possibly ketogenic diet HPA axis / Endocrine rs VDR G CT: 1/ % Vitamin D Receptor Downregulated Vitamin D receptor Vitamin D3, Sage, Rosemary Cardiovascular rs ALPL C CT: 1/ % alkaline phosphatase Lower concentration b6 Vitamin B6 Cardiovascular rs5882 CETP G AA: 2/ % Cholesterol ester transfer protein Cholesterol levels affected Low fat diet Cardiovascular rs5275 COX2 A AG: 1/ % Involved in the conversion of Increased response to fish oil Omega-3 fatty acids arachidonic acid to prostaglandin like fish oil H2, an important precursor of prostacyclin and thromboxane A2, among others. Methyl donors
8 Cardiovascular rs FADS1 G GT: 1/ % Fatty Acid Desaturase 1 Regulation of saturation of fatty acids, significantly associated with lower concentrations of long-chain PUFA. At baseline, men with the rs174537t allele had lower arachidonic acid (AA) and AA/linoleic acid (LA), and higher interleukin (IL)-6 levels than rs174537gg counterparts. After 3 years, rs174537gg men had significantly increased AA (P = 0.022), AA/dihomo-gamma-linolenic acid (DGLA) (P = 0.007), docosapentaenoic acid (DPA), low-density lipoprotein (LDL) cholesterol, and oxidized LDL (ox-ldl), but decreased eicosatrienoic acid. The rs174537t group showed significantly increased gamma-linolenic acid and ox-ldl, and decreased eicosadienoic acid, eicosapentaenoic acid (EPA)/alpha-linolenic acid (ALA), and IL-6. Cardiovascular rs662 PON1 A CT: 1/ % Major antiatherosclerotic component of HDL Glutamine high activity, arginine low activity, position 192, Low serum PON1 activity in NIDDM may be related to an increased tendency to lipid peroxidation and may also increase susceptibility to toxicity from organophosphate exposure. Omega-3 fatty acids like fish oil, Plant oils Omega-3 fatty acids like fish oil, Fat soluble antioxidants, Vitamin K High fat diet
9 Cardiovascular rs USF1 G TT: 2/ % Upstream Stimulatory Factor 1 Cholesterol levels affected Fiber High fat diet Cardiovascular rs VKORC1 C TT: 2/ % Reduces vitamin K 2,3-epoxide to the enzymatically activated form. Related to vitamin K recycling. Vitamin K Digestion / Elimination rs BCMO1 G TT: 2/ % Key enzyme in beta-carotene reduced catalytic activity by 48% Vitamin A metabolism to vitamin A. Digestion / Elimination rs BCMO1 G AG: 1/ % Key enzyme in beta-carotene reduced catalytic activity by 51% Vitamin A metabolism to vitamin A. Digestion / Elimination rs BCMO1 A AG: 1/ % Key enzyme in beta-carotene metabolism to vitamin A. reduced catalytic activity by 59% Vitamin A Digestion / Elimination rs FUT2 T AG: 1/ % Fucosyltransferase 2 enzyme which determines 'secretor status' Digestion / Elimination rs FUT2 G AG: 1/ % Fucosyltransferase 2 enzyme which determines 'secretor status' Reduced intestinal microbiota Probiotics diversity but higher vitamin B12 levels Reduced intestinal microbiota Probiotics diversity, non secretor Digestion / Elimination rs FUT2 G AG: 1/ % Fucosyltransferase 2 enzyme which determines 'secretor status' Digestion / Elimination rs IL-23R C AC: 1/ % Important part of the inflammatory response against infection. It promotes upregulation of the matrix metalloprotease MMP9, increases angiogenesis and reduces CD8+ T-cell infiltration. Reduced intestinal microbiota diversity. Interferes with absorption of B12. Individuals on vegetarian diet with GG (homozygous major genotype) have significantly lower levels of vitamin B(12). Affects intestinal health Probiotics Probiotics, Omega-3 fatty acids like fish oil, Vitamin D3
10 Energy / Oxidation rs10370 SOD2 G GT: 1/2 N/A Mitochondrial Superoxide Dismutase 2 Decreased gene function. Noise induced hearing loss, rs10370 'TT', rs4880 'GG' diplo-genotype (diplotype) was associated with more gray matter shrinkage in 76 individuals who report chronic high levels of alcohol consumption. Manganese, Vitamin E in tocotrienol form Energy / Oxidation rs SOD2 T CT: 1/ % Manganese superoxide dismutase Decreased gene function Vitamin E in tocotrienol form, Manganese Energy / Oxidation rs4880 SOD2 A AG: 1/ % Mitochondrial Superoxide Decreased gene function. Noise Manganese, Vitamin Dismutase 2 induced hearing loss, rs10370 E in tocotrienol form 'TT', rs4880 'GG' diplo-genotype (diplotype) was associated with more gray matter shrinkage in 76 individuals who report chronic high levels of alcohol consumption. Alcohol, Noise (greater chance for hearing loss) Alcohol, Noise (greater chance for hearing loss)
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