DR SHRENIK M SHAH SHREY HOSPITAL AHMEDABAD
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1 DR SHRENIK M SHAH SHREY HOSPITAL AHMEDABAD
2 Surgical anatomy Physiology of healing Classification Pre-operative evaluation OVERVIEW
3 Ultrastructure of the nerve
4 Fragile handle with care Damaged by pressure, stretching or cutting can stop signals to and from the brain Motor loss Sensory loss Neuromas Phantom limb CRPS I & II
5 Pathophysiology demyelination and axonal degeneration motor and sensory loss. Recovery of function remyelination axonal regeneration 1 mm/day or 1 inch/month monitored with an advancing Tinel sign. reinnervation of the sensory receptors, muscle end plates, or both.
6 Neural elements fail first to stretch and epineurium last
7 Seddon classification 1943 Neuropraxia Axonotemesis Neurotemesis
8 Sunderland classification of nerve injury
9 Sunderland classification 1951 First-degree nerve injury- neuropraxia, recovery-upto 12 weeks Second-degree nerve injury: axonotmesis causes wallerian degeneration distal to the level of injury and proximal axonal degeneration to at least the next node of Ranvier EMG/NCV denervation changes in the affected muscles reinnervation motor unit potentials (MUPs) + Endoneurial tubes are intact recovery is complete with axons reinnervating their original motor and sensory targets
10 Sunderland classification-3* More severe- endoneurial tubes are not intact regenerating axons therefore may not reinnervate their original motor and sensory targets The pattern of recovery is mixed and incomplete
11 Sunderland classification-4 large area of scar at the site of nerve injury precludes any axons from advancing distal to the level of nerve injury. EMG-denervation changes in the affected muscles, and no MUPs Tinel s sign doesnot advance No improvement in function Surgery
12 Sunderland classification-5 & 6 5th-degree injury is a complete transection of the nerve 6 th degree:(mackinnon ) a mixed nerve injury that combines the other degrees of injury. some fascicles of the nerve are working normally while other fascicles may be recovering, and other fascicles may require surgical intervention to permit axonal regeneration.
13 Sensory loss with nerve injury
14 Treatment indications of surgery Closed nerve injury: With no evidence of recovery either clinically /EMG at 3 months following injury Open nerve injury : All lacerations with a reported loss of sensation or motor weakness should be surgically explored asap. Crush nerve injury: Surgery delayed for as long as several weeks. after 3 months explore if no evidence of reinnervation electrically or clinically-repair or graft
15 Splints, slings Medical therapy Physical therapy- prevent stiffness, exercises and biofeedback strategies to increase strength no studies have shown that external stimulation will stop total degeneration of the muscle fibers and/or neuromuscular junction direct current stimulation risk of a thermal burn.
16 Surgical therapy Laceration: explore asap- repair if clean cut, nerve ends approximated if untidy wound/ crush and delayed repair at 3 weeks. Gunshot wounds associated with neurologic deficit have good potential for neurologic recovery. managed conservatively and monitored with frequent clinical examinations Closed injury- traction injuries- At 3 months if no recovery- explore
17 Pre operative assessment Sensory evaluation: 2 point discrimination Light touch- Ten test(patient compares the sensation on a scale of 0-10) Motor assessment: Pinch and grip strength assessment evaluation of individual muscle strength using the Medical Research Council (MRC) 0-5 grading scale
18 Intra opearative details Magnification- 4.3 loupes/ microscope Tourniquett Nerve coaptations are performed with 9-0 microsuture so that tension at the repair site is avoided. Nerve graft if tension sural nerve anterior branch of the medial antebrachial cutaneous (MABC) nerve - shorter nerve gaps-minimal scar and sensory loss Vs lat antebrachial cutaneous nerve
19 Post op regime Immobilization: n graft d, n repair- 3 weeks Hand therapist: splint and exercises Motor and sensory reeducation monitored every few months to evaluate for evidence of reinnervation. With nerve regeneration, a Tinel sign progresses distally along the nerve distal injuries respond more quickly than proximal brachial plexus injuries, which respond for 2-3 years following surgery
20
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