6/3/2018 9:37:00AM 6/3/2018 9:39:05AM 6/3/2018 1:44:56PM A/c Status. Test Name Results Units Bio. Ref. Interval Bilirubin Direct 0.

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1 LL - LL-ROHINI (NATIONAL REFERENCE Age 45 Years Gender Male 6/3/ AM 6/3/ AM 6/3/ M Ref By Final Swasth lus Tax Saver anel 1 LIVER & KIDNEY ANEL, SERUM (Spectrophotometry, Indirect ISE) Bilirubin Total Bilirubin Direct 0.25 <0.20 Bilirubin Indirect 0.93 <1.10 AST (SGOT) 58 U/L <50 ALT (SGT) 46 U/L <50 GGT 208 U/L <55 Alkaline hosphatase (AL) 107 U/L Total rotein 7.70 g/dl Albumin 4.56 g/dl A G Ratio Urea Creatinine Uric Acid Calcium, Total hosphorus Sodium meq/l otassium 3.97 meq/l Chloride meq/l THYROID ROFILE,TOTAL, SERUM (CLIA) T3, Total 1.15 ng/ml T4, Total 6.10 ug/dl TSH 3.36 uiu/ml Note 1. TSH levels are subject to circadian variation, reaching peak levels between 2-4.a.m. and at a minimum between 6-10 pm. The variation is of the order of 50, hence time of the day has influence on the measured serum TSH concentrations. 2. Recommended test for T3 and T4 is unbound fraction or free levels as it is metabolically active. 3. hysiological rise in Total T3 / T4 levels is seen in pregnancy and in patients on steroid therapy. Clinical Use atientreportscsuperanel.general_anel_analyte_sc (Version 6) age 1 of 8

2 LL - LL-ROHINI (NATIONAL REFERENCE Age 45 Years Gender Male 6/3/ AM 6/3/ AM 6/3/ M Ref By Final rimary Hypothyroidism Hyperthyroidism Hypothalamic - ituitary hypothyroidism Inappropriate TSH secretion Nonthyroidal illness Autoimmune thyroid disease regnancy associated thyroid disorders Thyroid dysfunction in infancy and early childhood atientreportscsuperanel.general_anel_analyte_sc (Version 6) age 2 of 8

3 LL - LL-ROHINI (NATIONAL REFERENCE Age 45 Years Gender Male 6/3/ AM 6/3/ AM 6/3/ M Ref By Final URINE EXAMINATION, ROUTINE; URINE, R/E (Automated Strip Test, Microscopy) hysical Colour Specific Gravity ph Chemical roteins Glucose Ketones Bilirubin Urobilinogen Leucocyte Esterase Nitrite Microscopy R.B.C. us Cells Epithelial Cells Casts Crystals Light Yellow <= Normal Few ale yellow Normal 0-5 WBC / hpf Few /lpf Others - atientreportscsuperanel.urine_examination_sc (Version 6) age 3 of 8

4 LL - LL-ROHINI (NATIONAL REFERENCE Age 45 Years Gender Male 6/3/ AM 6/3/ AM 6/3/ M Ref By Final HbA1c (GLYCOSYLATED HEMOGLOBIN), BLOOD (HLC, NGS certified) 5.9 Interpretation As per American Diabetes Association (ADA) Reference Group HbA1c in Non diabetic adults >=18 years < At risk (rediabetes) Diagnosing Diabetes >= Therapeutic goals for glycemic Age > 19 years control. Goal of therapy < 7.0. Action suggested > 8.0 Age < 19 years. Goal of therapy < Note 1. Since HbA1c reflects long term fluctuations in the blood glucose concentration, a diabetic patient who is recently under good control may still have a high concentration of HbA1c. Converse is true for a diabetic previously under good control but now poorly controlled. 2. Target goals of < 7.0 may be beneficial in patients with short duration of diabetes, long life expectancy and no significant cardiovascular disease. In patients with significant complications of diabetes, limited life expectancy or extensive co-morbid conditions, targeting a goal of < 7.0 may not be appropriate. Comments HbA1c provides an index of average blood glucose levels over the past 8-12 weeks and is a much better indicator of long term glycemic control as compared to blood and urinary glucose determinations. ADA criteria for correlation between HbA1c & Mean plasma glucose levels HbA1c() Mean lasma Glucose () atientreportscsuperanel.hbelectro_sc (Version 7) age 4 of 8

5 LL - LL-ROHINI (NATIONAL REFERENCE Age 45 Years Gender Male 6/3/ AM 6/3/ AM 6/3/ M Ref By Final atientreportscsuperanel.hbelectro_sc (Version 7) age 5 of 8

6 LL - LL-ROHINI (NATIONAL REFERENCE Age 45 Years Gender Male 6/3/ AM 6/3/ AM 6/3/ M Ref By Final HEMOGRAM (Electrical Impendance & VCS, Capillary photometry,hotometry) Hemoglobin g/dl acked Cell Volume (CV) RBC Count mill/mm3 MCV fl MCH pg MCHC g/dl Red Cell Distribution Width (RDW) Total Leukocyte Count (TLC) Differential Leucocyte Count (DLC) Segmented Neutrophils Lymphocytes Monocytes 8.60 Eosinophils 1.20 Basophils 0.30 Absolute Leucocyte Count Neutrophils Lymphocytes Monocytes Eosinophils Basophils latelet Count ESR mm/hr Note 1. As per the recommendation of International council for Standardization in Hematology, the differential leucocyte counts are additionally being reported as absolute numbers of each cell in per unit volume of blood < Test conducted on EDTA whole blood age 6 of 8

7 LL - LL-ROHINI (NATIONAL REFERENCE 6/3/ AM 6/3/ AM Age 45 Years Gender Male 6/3/ M Ref By Final GLUCOSE, FASTING (F), LASMA (Hexokinase) LIID SCREEN, SERUM (Spectrophotometry) Cholesterol, Total < Triglycerides < HDL Cholesterol >40.00 LDL Cholesterol, Calculated < VLDL Cholesterol,Calculated <30.00 Interpretation REMARKS TOTAL TRIGLYCERIDE LDL CHOLESTEROL CHOLESTEROL in in in Optimal <200 <150 <100 Above Optimal Borderline High High >= Very High - >=500 >= Note 1. Measurements in the same patient can show physiological & analytical variations. Three serial samples 1 week apart are recommended for Total Cholesterol, Triglycerides, HDL & LDL Cholesterol. 2. AT III recommends a complete lipoprotein profile as the initial test for evaluating cholesterol. 3. Friedewald equation to calculate LDL cholesterol is most accurate when Triglyceride level is < 400. Measurement of Direct LDL cholesterol is recommended when Triglyceride level is > 400. IRON STUDIES, SERUM (Spectrophotometry) atientreportscsuperanel.s_general_temlate01_sc (Version 7) age 7 of 8

8 LL - LL-ROHINI (NATIONAL REFERENCE Age 45 Years Gender Male 6/3/ AM 6/3/ AM 6/3/ M Ref By Final Iron µg/dl Total Iron Binding Capacity µg/dl Transferrin Saturation Comments Iron is an essential trace mineral element which forms an important component of hemoglobin, metallocompounds and Vitamin A. Deficiency of iron, leads to microcytic hypochromic anemia. The toxic effects of iron are deposition of iron in various organs of the body and hemochromatosis. Total Iron Binding capacity (TIBC) is a direct measure of the protein Transferrin which transports iron from the gut to storage sites in the bone marrow. In iron deficiency anemia, serum iron is reduced and TIBC increases. Transferrin Saturation occurs in Idiopathic hemochromatosis and Transfusional hemosiderosis where no unsaturated iron binding capacity is available for iron mobilization. Similar condition is seen in congenital deficiency of Transferrin. Dr. Anil Arora MD (athology) HOD Hemat & Imm - NRL Dr Biswadip Hazarika MD (athology) Sr. Consultant athologist - NRL Dr Himangshu Mazumdar MD (Biochemistry) Consultant Biochemist - NRL Dr. Nimmi Kansal MD (Biochemistry) HOD Biochem & IA - NRL Dr. Shalabh Malik MD (Microbiology) National Head - Microbiology & Serology - NRL End of report atientreportscsuperanel.s_general_temlate01_sc (Version 7) age 8 of 8