Critical care nurses are called
|
|
- Laurence Horn
- 6 years ago
- Views:
Transcription
1 CardiovascularMedicine Hemodynamic Monitoring in High-Risk Obstetrics Patients, II Pregnancy-Induced Hypertension and Preeclampsia Lt Col Elizabeth J. Bridges, USAF NC Capt Shannon Womble, USAF NC Capt Marlene Wallace, USAF NC Capt Jerry McCartney, USAF NC Critical care nurses are called upon to assist with the care of critically ill obstetrics patients. Some of the most complex care is required for patients with pregnancy-induced hypertension or preeclampsia. This article provides an overview of the pathological changes and expected hemodynamic changes associated with pregnancy-induced hypertension. A previous article 1 addressed the changes expected during a normal pregnancy and the changes associated with cardiovascular disease or hemorrhage. The clinical manifestations and medical and nursing management are summarized in a case study. Expected Hemodynamic Changes in Pregnancy Interpreting the pathophysiological changes that occur with pregnancy-induced hypertension requires a review of the hemodynamic changes expected during pregnancy, particularly during the third trimester. In summary (Table 1), increases in systolic and diastolic blood pressure of up to 10% of baseline are expected. 2-4 These increases in blood pressure reflect an increase in stroke volume and cardiac output, despite a decrease in systemic vascular resistance (SVR). Additionally, despite the marked increase in blood volume (as indicated Authors Elizabeth J. Bridges is Deputy Commander of the 59th Clinical Research Squadron and senior nurse researcher at the 59th Medical Wing, Lackland AFB, San Antonio, Tex. Shannon Womble, Marlene Wallace, and Jerry McCartney are staff nurses in the surgical intensive care unit of the 59th Medical Wing at Lackland AFB. To purchase reprints, contact The InnoVision Group, 101 Columbia, Aliso Viejo, CA Phone, (800) or (949) (ext 532); fax, (949) ; , reprints@aacn.org. by an increase in left ventricular enddiastolic volume) and pulmonary blood flow, pulmonary artery pressure and pulmonary artery wedge pressure (PAWP) remain at baseline levels throughout pregnancy. 6,15,16 Recognition of these expected changes is important. In patients with preeclampsia, SVR may increase dramatically, with a resultant decrease in cardiac output, or a hyperdynamic profile (high cardiac output and low SVR) may persist. 8,17-19 Additionally, remembering that pulmonary artery pressure and PAWP generally remain at baseline levels throughout pregnancy is useful in the differential diagnosis of refractory oliguria that may accompany preeclampsia. 20,21 Hemodynamic Alterations Associated With Pregnancy- Induced Hypertension, Preeclampsia, and Eclampsia Pregnancy-induced hypertension is hypertension that develops as a consequence of pregnancy and regresses after delivery. Pregnancy-induced hypertension can be differentiated from chronic hypertension, which appears before 20 weeks gestation or continues for a long period after delivery. 22 Preeclampsia, which is a type of pregnancy-induced hypertension characterized by progressive hypertension and pathological edema, is clinically defined as a blood pressure greater than 140/90 mm Hg after 20 weeks gestation plus proteinuria (300 mg/24 hours or greater than 1+ protein on a dipstick sample of urine collected at random). 23 Eclampsia is the occurrence of con- 52 CRITICALCARENURSE Vol 23, No. 5, OCTOBER 2003
2 Table 1 Hemodynamic changes during pregnancy 2,5-14 Time Parameter Before pregnancy Third trimester,* % change (laboratory value) Labor (during contractions), % change After delivery Blood volume, L % (6.2 L) L (10-20%) Plasma volume, L % ( L) 10% by day 5 Total decrease in blood volume (cells + plasma) = 20% Hemoglobin, g/l % ( g/l) Hematocrit, proportion of % ( ) Stable, then 6% by day 5 Blood pressure, mm Hg Systolic Diastolic % (decreased through week 24) 0-10% (decreased through week 24) Increases Increases Returns to prelabor values at 24 hours Returns to prelabor values at 24 hours Cardiac output, L/min % (7.3 L/min) 11 Returns to prelabor values at 1 hour and to prepregnancy values at days Stroke volume, ml per beat % (79-85 ml per beat) 11 Returns to prelabor values at 24 hours and to prepregnancy values at 12 weeks to 1 year Heart rate, beats per minute % (87-95 beats per minute) 0-20 Returns to prelabor values at 1 hour and to prepregnancy values at 6 to 12 weeks Systemic vascular resistance, dyn s cm % ( 34% to nadir at weeks) Returns to prepregnancy values at 12 weeks to 1 year Central venous pressure, mm Hg 2-6 Pulmonary artery pressures, mm Hg Systolic End-diastolic Wedge Unchanged throughout pregnancy Unchanged throughout pregnancy Unchanged throughout pregnancy Pulmonary vascular resistance, dyn s cm % (175 dyn s cm -5 ) Colloid oncotic pressure, mm Hg 20 *Compared with value before pregnancy. Compared with third trimester/prelabor values. indicates increase;, decrease;, no data. Reprinted from Bridges et al, 1 with permission. vulsions or coma unrelated to other cerebral conditions with signs and symptoms of preeclampsia. Preeclampsia and eclampsia may be complicated by the onset of the HELLP syndrome (hemolysis, elevated liver enzymes, and low platelet count). Patients with HELLP syndrome are a subset of those with severe pregnancy-induced hypertension who are at increased risk for multiple organ system dysfunction. 24,25 Maternal complications associated with HELLP include a coagulopathy (specifically, microangiopathic hemolytic anemia) due to liver failure and thrombocytopenia, acute respiratory distress syndrome, and acute renal failure; all of these may require hemodynamic monitoring to guide therapy. The hemodynamic profile of a patient with preeclampsia varies CRITICALCARENURSE Vol 23, No. 5, OCTOBER
3 CardiovascularMedicine depending on the stage of the disease. During the preclinical or latent phase of preeclampsia, the hemodynamic profile is characterized as hyperdynamic, that is, increased cardiac output with normal vascular resistance. 8,17 With the onset of preeclampsia, the hemodynamic profile varies. In one longitudinal study, 17 women in whom preeclampsia developed crossed over from a high-output state to a highresistance state, with a dramatic decrease in cardiac output (ie, greater than 3 L/min) and an increase in vascular resistance. However, in other studies, 8,18,19 many women had an unchanged profile (high-output or high-resistance states). These differences may be due to variability in treatment among different study populations or to the presence of more than a single hemodynamic profile for preeclampsia. These findings highlight the importance of individualizing the treatment of patients who have preeclampsia. Hemodynamic monitoring may be required for 3 subsets of patients with preeclampsia, specifically patients with refractory oliguria, pulmonary edema, or refractory hypertension. 20,21,26 In patients with preeclampsia, central venous pressures correlate poorly with PAWP; thus if hemodynamic monitoring is needed, a pulmonary artery catheter is generally required. 27 Refractory Oliguria Three different hemodynamic subsets of patients with preeclampsia with persistent oliguria have been described (Table 2). 20,21 Subset 1 (low PAWP, hyperdynamic left ventricular function, and normal or increased SVR) is consistent with intravascular volume depletion. Patients in this subset respond to volume resuscitation. Subset 2, which is characterized by normal or increased PAWP, normal cardiac output, and normal SVR, is thought to be caused by renal arteriospasm. Treatment for patients in this subset is focused on reducing arteriospasm by administration of low-dose dopamine. In subset 3, the hemodynamic profile is consistent with systemic vasoconstriction (increased PAWP, increased SVR, decreased cardiac output). Treatment for patients in this subset is focused on afterload reduction and diuresis to improve ventricular function. Pulmonary Edema The risk of pulmonary edema is increased in patients with pregnancyinduced hypertension and preeclamp- sia because of a decrease in colloid osmotic pressure (COP). 6,28-33 COP, which is approximately 16 to 20 mm Hg in a healthy person lying supine, offsets the pressure (capillary hydrostatic pressure) that forces fluid out of the capillary. If the COP is decreased, net movement of fluid into the interstitium is increased, resulting in pulmonary edema. The risk of pulmonary edema is especially high during the postpartum period when a further decrease in COP occurs. 28,34 The decreased COP is particularly problematic if large volumes of crystalloids are used in resuscitation Other causes of pulmonary edema, including cardiogenic pulmonary edema (eg, iatrogenic volume overload) and alteration in pulmonary capillary permeability must also be ruled out. Refractory Hypertension Placement of a pulmonary artery catheter may be useful in treating patients who are refractory to standard hypertensive therapy (eg, hydralazine or labetolol). 21,38 Monitoring via a pulmonary artery catheter may help differentiate the cause of increased blood pressure, such as increased blood pressure due to increased vascular resistance. In Table 2 Hemodynamic subsets in preeclampsia with oliguria 20,21 Subset Causal mechanism Pulmonary artery wedge pressure Systemic vascular resistance Left ventricular function Treatment 1 Intravascular volume depletion Low or low normal Moderately increased Hyperdynamic cardiac output Volume resuscitation 2 Renal arteriospasm Normal Volume resuscitation Preload reduction Afterload reduction 3 Generalized vasospasm Suppressed Decreased cardiac output Afterload reduction Diuresis 54 CRITICALCARENURSE Vol 23, No. 5, OCTOBER 2003
4 Case Study The following case study describes the hemodynamic changes and medical and nursing care for a patient with complicated preeclampsia. Findings at Admission A 20-year-old, gravida 2, para 0 woman was admitted at 30 weeks gestation because of pregnancy-induced hypertension. She was having irregular contractions. She had no history of hypertension or other medical conditions. Her vital signs were as follows: Heart rate: 108/min Blood pressure: 160/110 mm Hg Respirations: 26/min Temperature: 37.4 C (99.2 F) Arterial oxygen saturation: 94% Nursing Note All blood pressure measurements should be done with the patient in the same position (blood pressure is highest in the sitting position and lowest in the sidelying position) 43 and by using the same technique (ie, do not compare a measurement obtained via an arterial catheter with a measurement obtained by using an automated blood pressure cuff ) The assessment findings were as follows: Renal system: 4+ proteinuria with a urine output of approximately 12 ml/h Generalized peripheral edema Respiratory system: lung sounds clear Fetal heart tones: heart rate = 140/min, with accelerations and occasional variable decelerations. (Variable decelerations occur in approximately 50% of all fetuses during labor, are usually temporary, and may change with the mother s body position. 47 ) Nursing Note Despite clear lung sounds, this patient is at increased risk for pulmonary edema, particularly during the postpartum period. 28,34 Continued close assessment of pulmonary status is warranted. The oliguria may reflect intravascular volume deficit, renal arterial vasospasm, or systemic vasoconstriction. Evaluation of the patient s response to a bolus of fluid and evaluation of her hemodynamic status will aid in the differential diagnosis of the oliguria. 20,21 Treatment The patient was given a 4-g bolus dose of magnesium sulfate and then a maintenance dose of 2 g. Isotonic sodium chloride solution was infused at a rate of 125 ml/h after intravenous administration of a bolus dose of 500 ml. Pitocin was administered to induce labor. Nursing Note Continued close monitoring of neurological status is warranted to detect signs and symptoms of hypoxemia, impending seizure activity, increased intracranial pressure, or toxic effects of magnesium. 48 Calcium gluconate, the antidote for magnesium poisoning, should be kept at the bedside; the usual dose is a 1-g intravenous bolus. 48 Administration of crystalloid solution must be performed with caution because the combination of increased volume resuscitation and decreased COP increases the risk for pulmonary edema Findings 4 Hours After Initial Treatment Four hours after treatment, the patient s vital signs were as follows: Heart rate: 120/min Blood pressure: 169/104 mm Hg Respirations: 28/min Arterial oxygen saturation: 87%, with oxygen given at a rate of 10 L/min Assessment findings were as follows: Renal system: urine output 30 to 50 ml/h Respiratory system: crackles in lower lobes Fetal heart tones: tracing worsened to deceleration with no variability (indicative of fetal compromise) Treatment The patient was delivered of her infant by cesarean birth. The mother had a catheter inserted in the pulmonary artery because of intraoperative bleeding and pulmonary compromise. Her hemodynamic profile was as follows: Blood pressure: 173/118 mm Hg, mean 136 mm Hg Right atrial pressure: 6 mm Hg Pulmonary artery pressures (systolic/diastolic): 32/18 mm Hg PAWP: 16 mm Hg Cardiac output: 12 L/min SVR: 832 dynes s cm -5 COP: 10 mm Hg The rate of administration of intravenous fluids was decreased to 50 ml/h. The patient was given furosemide (Lasix) 10 mg intravenously and labetolol 10 mg intravenously. Outcome Within 24 hours of delivery, the patient s hemodynamic profile returned to normal. Discussion This patient had the classic signs and symptoms of preeclampsia (hypertension, proteinuria, edema) along with oliguria. 48 The oliguria was initially treated with crystalloids. Four hours later, her condition had deteriorated and was consistent with uncontrolled hypertension and pulmonary edema (bibasilar crackles and decreased arterial oxygen saturation). After placement of the pulmonary artery catheter, the hemodynamic profile reflected a hyperdynamic state (increased cardiac output and decreased SVR). The pulmonary edema most likely was due to decreased COP and was exacerbated by the slight increase in PAWP associated with the crystalloid administration. (Note: pulmonary edema does not generally become apparent until the PAWP exceeds 18 mm Hg.) As in this case, the treatment for preeclampsia is delivery if possible. 47 The pulmonary edema was treated in a standard manner with diuretics and reduction of intravenous fluids. CRITICALCARENURSE Vol 23, No. 5, OCTOBER
5 CardiovascularMedicine patients with increased blood pressure due to increased vascular resistance, when the vascular resistance is decreased, the cardiac output increases without a change in blood pressure. The other less common cause of increased blood pressure is increased cardiac output, which can be diagnosed via pulmonary artery pressure monitoring. 21,26,39 Other Clinical Concerns in Preeclampsia Other factors to consider in the care of patients with preeclampsia are that compared with women who have a normal pregnancy, women with severe preeclampsia have decreased oxygen consumption, oxygen delivery, and oxygen extraction ratio. 17,35,39-41 The altered oxygen balance may be particularly important in patients with compromised cardiac output. Additionally, in patients with preeclampsia or eclampsia, the blood volume does not increase as much as in a normal pregnancy, making these patients with complicated preeclampsia less tolerant to peripartum blood loss. 42 Acknowledgments The opinions and assertions contained herein are the private views of the authors and are not to be construed as the official policy or position of the US government, the Department of Defense, or the Department of the Air Force. References 1. Bridges EJ, Womble S, Wallace M, McCartney J. Hemodynamic monitoring in high-risk obstetrics patients, I: expected hemodynamic changes during pregnancy. Crit Care Nurse. August 2003;23: Clapp JF III, Capeless E. Cardiovascular function before, during, and after the first and subsequent pregnancies. Am J Cardiol. 1997;80: Duvekot JJ, Cheriex EC, Pieters FA, Menheere PP, Peeters LH. Early pregnancy changes in hemodynamics and volume homeostasis are consecutive adjustments triggered by a primary fall in systemic vascular tone. Am J Obstet Gynecol. 1993;169: Robson SC, Hunter S, Boys RJ, Dunlop W. Serial study of factors influencing changes in cardiac output during human pregnancy. Am J Physiol. 1989;256(4 pt 2):H1060- H Capeless EL, Clapp JF. When do cardiovascular parameters return to their preconception values? Am J Obstet Gynecol. 1991;165 (4 pt 1): Clark SL, Cotton DB, Lee W, et al. Central hemodynamic assessment of normal term pregnancy. Am J Obstet Gynecol. 1989;161 (6 pt 1): Duvekot JJ, Peeters LL. Maternal cardiovascular hemodynamic adaptation to pregnancy. Obstet Gynecol Surv. 1994;49(12 suppl): S1-S Easterling TR, Benedetti TJ, Schmucker BC, Millard SP. Maternal hemodynamics in normal and preeclamptic pregnancies: a longitudinal study. Obstet Gynecol. 1990;76: Mabie WC, DiSessa TG, Crocker LG, Sibai BM, Arheart KL. A longitudinal study of cardiac output in normal human pregnancy. Am J Obstet Gynecol. 1994;170: Milman N, Byg KE, Agger AO. Hemoglobin and erythrocyte indices during normal pregnancy and postpartum in 206 women with and without iron supplementation. Acta Obstet Gynecol Scand. 2000;79: Robson SC, Boys RJ, Hunter S, Dunlop W. Maternal hemodynamics after normal delivery and delivery complicated by postpartum hemorrhage. Obstet Gynecol. 1989; 74: Thomsen JK, Prien-Larsen JC, Devantier A, Fogh-Andersen N. Low dose iron supplementation does not cover the need for iron during pregnancy. Acta Obstet Gynecol Scand. 1993;72: Thomsen JK, Fogh-Andersen N, Jaszczak P, Giese J. Atrial natriuretic peptide (ANP) decrease during normal pregnancy as related to hemodynamic changes and volume regulation. Acta Obstet Gynecol Scand. 1993;72: van Oppen AC, van der Tweel I, Alsbach GP, Heethaar RM, Bruinse HW. A longitudinal study of maternal hemodynamics during normal pregnancy. Obstet Gynecol. 1996; 88: Gilson GJ, Samaan S, Crawford MH, Qualls CR, Curet LB. Changes in hemodynamics, ventricular remodeling, and ventricular contractility during normal pregnancy: a longitudinal study. Obstet Gynecol. 1997; 89: Robson SC, Hunter S, Boys RJ, Dunlop W. Serial changes in pulmonary haemodynamics during human pregnancy: a non-invasive study using Doppler echocardiography. Clin Sci (Lond). 1991;80: Bosio PM, McKenna PJ, Conroy R, O Herlihy C. Maternal central hemodynamics in hypertensive disorders of pregnancy. Obstet Gynecol. 1999;94: Hjertberg R, Belfrage P, Hagnevik K. Hemodynamic measurements with Swan- Ganz catheter in women with severe proteinuric gestational hypertension (pre-eclampsia). Acta Obstet Gynecol Scand. 1991;70: Penny JA, Anthony J, Shennan AH, De Swiet M, Singer M. A comparison of hemodynamic data derived by pulmonary artery flotation catheter and the esophageal Doppler monitor in preeclampsia. Am J Obstet Gynecol. 2000;183: Clark SL, Greenspoon JS, Aldahl D, Phelan JP. Severe preeclampsia with persistent oliguria: management of hemodynamic subsets. Am J Obstet Gynecol. 1986;154: Clark SL, Cotton DB. Clinical indications for pulmonary artery catheterization in the patient with severe preeclampsia. Am J Obstet Gynecol. 1988;158: Committee on Technical Bulletins of the American College of Obstetricians and Gynecologists. ACOG technical bulletin: hypertension in pregnancy. Number 219 January 1996 (replaces No. 91, February 1986). Int J Gynaecol Obstet. 1996;53: Davey DA, MacGillivray I. The classification and definition of the hypertensive disorders of pregnancy. Am J Obstet Gynecol. 1988;158: Curtin WM, Weinstein L. A review of HELLP syndrome. J Perinatol. 1999;19: Isler CM, Rinehart BK, Terrone DA, Martin RW, Magann EF, Martin JN Jr. Maternal mortality associated with HELLP (hemolysis, elevated liver enzymes, and low platelets) syndrome. Am J Obstet Gynecol. 1999;181: Fox DB, Troiano NH, Graves CR. Use of the pulmonary artery catheter in severe preeclampsia: a review. Obstet Gynecol Surv. 1996;51: Clark SL. Reliance on central venous pressure with regard to fluid management in preeclampsia deemed dangerous. Am J Obstet Gynecol. 1990;162: Benedetti TJ, Kates R, Williams V. Hemodynamic observations in severe preeclampsia complicated by pulmonary edema. Am J Obstet Gynecol. 1985;152: Oian P, Maltau JM. Transcapillary forces in normal pregnant women. Acta Med Scand Suppl. 1985;693: Oian P, Maltau JM. Calculated capillary hydrostatic pressure in normal pregnancy and preeclampsia. Am J Obstet Gynecol. 1987;157: Wu PY, Udani V, Chan L, Miller FC, Henneman CE. Colloid osmotic pressure: variations in normal pregnancy. J Perinat Med. 1983;11: Oian P, Maltau JM, Noddeland H, Fadnes HO. Transcapillary fluid balance in preeclampsia. Br J Obstet Gynaecol. 1986;93: Sibai BM, Mabie BC, Harvey CJ, Gonzalez AR. Pulmonary edema in severe preeclampsia-eclampsia: analysis of thirtyseven consecutive cases. Am J Obstet Gynecol. 1987;156: Nguyen HN, Clark SL, Greenspoon J, Diesfield P, Wu PY. Peripartum colloid osmotic pressures: correlation with serum proteins. Obstet Gynecol. 1986;68: Sibai BM, Mabie WC. Hemodynamics of preeclampsia. Clin Perinatol. 1991;18: Hauch MA, Gaiser RR, Hartwell BL, Datta S. Maternal and fetal colloid osmotic pressure following fluid expansion during cesarean section. Crit Care Med. 1995;23: Park GE, Hauch MA, Curlin F, Datta S, Bader AM. The effects of varying volumes of crystalloid administration before cesarean delivery on maternal hemodynamics and colloid osmotic pressure. Anesth Analg. 1996;83: CRITICALCARENURSE Vol 23, No. 5, OCTOBER 2003
6 38. Repke JT, Robinson JN. The prevention and management of preeclampsia and eclampsia. Int J Gynaecol Obstet. 1998;62: Cotton DB, Lee W, Huhta JC, Dorman KF. Hemodynamic profile of severe pregnancy-induced hypertension. Am J Obstet Gynecol. 1988;158: Belfort MA, Anthony J, Saade GR, et al. The oxygen consumption/oxygen delivery curve in severe preeclampsia: evidence for a fixed oxygen extraction state. Am J Obstet Gynecol. 1993;169: Mabie WC, Ratts TE, Sibai BM. The central hemodynamics of severe preeclampsia. Am J Obstet Gynecol. 1989;161: Visser W, Wallenburg HC. Central hemodynamic observations in untreated preeclamptic patients. Hypertension. 1991;17: Clark SL, Cotton DB, Pivarnik JM, et al. Position change and central hemodynamic profile during normal third-trimester pregnancy and post partum. Am J Obstet Gynecol. 1991;164: Gupta M, Shennan AH, Halligan A, Taylor DJ, de Swiet M. Accuracy of oscillometric blood pressure monitoring in pregnancy and pre-eclampsia. Br J Obstet Gynaecol. 1997;104: Kirshon B, Lee W, Cotton DB, Giebel R. Indirect blood pressure monitoring in the postpartum patient. Obstet Gynecol. 1987;70: Natarajan P, Shennan AH, Penny J, Halligan AW, de Swiet M, Anthony J. Comparison of auscultatory and oscillometric automated blood pressure monitors in the setting of preeclampsia. Am J Obstet Gynecol. 1999;181: Bobak I, Jensen M. Maternity and Gynecologic Care. St Louis, Mo: Mosby; Poole JH. Aggressive management of HELLP syndrome and eclampsia. AACN Clin Issues. 1997;8:
Outcomes of Pregnancies at Risk for Hypertensive Complications Managed Using Impedance Cardiography
Outcomes of Pregnancies at Risk for Hypertensive Complications Managed Using Impedance Cardiography David G. Chaffin, M.D., 1 and Denise G. Webb, RNC, BSN 2 ABSTRACT We assessed the effect of antihypertensive
More informationPolicy REVISED: 6/30/2016 3:30 PM. Applies To: ObGyn Responsible Department: ObGyn Revised: June 30, 2016
Title: Antihypertensive Treatment for Severe Hypertension During Pregnancy Applies To: ObGyn Responsible Department: ObGyn Revised: June 30, 2016 Policy POLICY STATEMENT: Pregnant or postpartum patients
More informationYou admitted a previously healthy nullipara at 36 weeks gestation who presented with new-onset periorbital edema and is found to have blood pressure
Preeclampsia Case report You admitted a previously healthy nullipara at 36 weeks gestation who presented with new-onset periorbital edema and is found to have blood pressure readings of 150/100 to 155/105
More informationMercy San Juan Medical Center. Preeclampsia and Other Hypertensive Disorders of Pregnancy
SUBJECT: Preeclampsia and Other Hypertensive Disorders of Pregnancy DEPARTMENTS: FBC, Emergency Department PURPOSE: To outline the nursing management of inpatients who have preeclampsia or other hypertensive
More informationMagnesium sulfate has an antihypertensive effect on severe pregnancy induced hypertension
Hypertension Research In Pregnancy 11 S. Takenaka et al. ORIGINAL ARTICLE Magnesium sulfate has an antihypertensive effect on severe pregnancy induced hypertension Shin Takenaka 1, Ryu Matsuoka 2, Daisuke
More informationHemodynamic Changes in Obstetric Anesthesia. Sonia Vaida PANA, Hershey, April 2009
Hemodynamic Changes in Obstetric Anesthesia Sonia Vaida PANA, Hershey, April 2009 Pregnancy is a normal healthy condition and is simultaneously the most common altered physiologic state to which human
More informationAWHONN Oregon Section 2014
AWHONN Oregon Section 2014 Carol J Harvey, MS, BSN, RNC-OB, C-EFM, CS Northside Hospital Atlanta Cherokee - Forsyth Hypertensive in Pregnancy Carol J Harvey, MS, RNC-OB, C-EFM Clinical Specialist Northside
More informationCMQCC Preeclampsia Tool Kit: Hypertensive Disorders Across the Lifespan
CMQCC Preeclampsia Tool Kit: Hypertensive Disorders Across the Lifespan Carol J Harvey, MS, BSN, RNC-OB, C-EFM, CS Northside Hospital Atlanta Cherokee - Forsyth New! Improving Health Care Response to Preeclampsia:
More informationStroke in Pregnancy. Stroke in Pregnancy 6/23/13
G5#$#Preven*ng#Maternal#Morbidity#and#Mortality#Via# Expanded#Scope#of#Nursing#Prac*ce#As#First#Responder# in#hypertensive#crisis#of#preeclampsia# The$presenter$reports$no$relevant,$influencing$financial$rela5onships.$
More informationWHAT FLUIDS AND WHEN
WHAT FLUIDS AND WHEN Fluid and Electrolytes Balance Pregnancy Labor Pathological processes during pregnancy Fluid and Electrolytes Balance Non pregnant woman Water homeostasis is a delicate balance between
More informationCHAPTER 12 HYPERTENSION IN SPECIAL GROUPS HYPERTENSION IN PREGNANCY
CHAPTER 12 HYPERTENSION IN SPECIAL GROUPS HYPERTENSION IN PREGNANCY v Mild preeclampsia is managed by close observation of the mother and fetus preferably in hospital. If the diastolic blood pressure remains
More informationClinical features. Abnormal vasculogenesis and angiogenesis and releasing of antiangiogenic
Clinical features Abnormal vasculogenesis and angiogenesis and releasing of antiangiogenic factors results in Vasospasm Endothelial dysfunction Etiology of various clinical signs and symptoms So, Preeclampsia
More informationAdmission of patient CVICU and hemodynamic monitoring
Admission of patient CVICU and hemodynamic monitoring Prepared by: Rami AL-Khatib King Fahad Medical City Pi Prince Salman Heart tcentre CVICU-RN Admission patient to CVICU Introduction All the patients
More informationHST 071. IN SUMMARY PREGNANCY INDUCED HYPERTENSION Classification of Hypertensive Disorders of Pregnancy
Harvard-MIT Division of Health Sciences and Technology HST.071: Human Reproductive Biology Course Director: Professor Henry Klapholz IN SUMMARY Classification of Hypertensive Disorders of Pregnancy HST
More informationValve Disease in the Pregnant Patient
Valve Disease in the Pregnant Patient Julie B. Damp, MD December 6, 2012 VanderbiltHeart.com If single, do not allow marriage. If fertile, do not allow pregnancy. If pregnant, do not allow delivery. If
More informationAcute Postpartum Pulmonary Edema: A Case Report
Postpartum pulmonary edema 157 Acute Postpartum Pulmonary Edema: A Case Report Min-Po Ho 1, Wing-Keung Cheung 2, Kaung-Chau Tsai 1 Acute pulmonary edema after pregnancy is rare. Pulmonary emobolism, pneumonia,
More informationPreeclampsia, defined by increased blood pressure and proteinuria,
Cardiovascular Changes in Preeclampsia Judith U. Hibbard, Sanjeev G. Shroff, and Roberto M. Lang The cardiovascular system undergoes a host of changes in association with the development of preeclampsia,
More informationPreeclampsia. &Eclampsia. Hypertensive Disorders of Pregnancy. What we need to know about. Hypertensive Disorders of Pregnancy.
Preeclampsia &Eclampsia Hypertensive Disorders of Pregnancy Barbara Koop MS, RNC-OB What we need to know about Hypertensive Disorders of Pregnancy Define clinical criteria for: Gestational hypertension,
More informationClinical Focus Paper Part I: Preeclampsia. Molly Boyle. Northwest University
Running head: CLINICAL FOCUS PAPER PART II: PREECLAMPSIA 1 Clinical Focus Paper Part I: Preeclampsia Molly Boyle Northwest University Author Note Molly A. Boyle, Buntain School of Nursing, Northwest University.
More informationPre-eclampsia: key issues. Robin Russell Nuffield Department of Anaesthetics John Radcliffe Hospital Oxford
Robin Russell Nuffield Department of Anaesthetics John Radcliffe Hospital Oxford Antenatal Issues Labour Analgesia Anaesthesia for Delivery High Dependency Care Hypertension systolic >140 mmhg or diastolic
More informationLEARNING OBJECTIVES 2/20/2017
HYPERTENSION IN PREGNANCY: PREVENTING SEVERE MATERNAL MORBIDITY & MORTALITY THROUGH THE IMPLEMENTATION OF EVIDENCED BASED PROTOCOLS Laura Senn, RN, PhD, CNS Sutter Medical Center, Sacramento LEARNING OBJECTIVES
More informationSHOCK AETIOLOGY OF SHOCK (1) Inadequate circulating blood volume ) Loss of Autonomic control of the vasculature (3) Impaired cardiac function
SHOCK Shock is a condition in which the metabolic needs of the body are not met because of an inadequate cardiac output. If tissue perfusion can be restored in an expeditious fashion, cellular injury may
More informationMaternal And Fetal Outcome In Pregnancies Complicated With Maternal Cardiac Diseases: Experience At A Tertiary Care Hospital
ISPUB.COM The Internet Journal of Gynecology and Obstetrics Volume 19 Number 1 Maternal And Fetal Outcome In Pregnancies Complicated With Maternal Cardiac Diseases: Experience At A Tertiary Care Hospital
More informationHYPOVOLEMIA AND HEMORRHAGE UPDATE ON VOLUME RESUSCITATION HEMORRHAGE AND HYPOVOLEMIA DISTRIBUTION OF BODY FLUIDS 11/7/2015
UPDATE ON VOLUME RESUSCITATION HYPOVOLEMIA AND HEMORRHAGE HUMAN CIRCULATORY SYSTEM OPERATES WITH A SMALL VOLUME AND A VERY EFFICIENT VOLUME RESPONSIVE PUMP. HOWEVER THIS PUMP FAILS QUICKLY WITH VOLUME
More informationTocolytics. Tocolytics (terbutaline, magnesium sulfate injection) Description
Federal Employee Program 1310 G Street, N.W. Washington, D.C. 20005 202.942.1000 Fax 202.942.1125 5.30.06 Subject: Tocolytics Page: 1 of 5 Last Review Date: September 15, 2016 Tocolytics Description Tocolytics
More informationTopics to be Covered. Cardiac Measurements. Distribution of Blood Volume. Distribution of Pulmonary Ventilation & Blood Flow
Topics to be Covered MODULE F HEMODYNAMIC MONITORING Cardiac Output Determinants of Stroke Volume Hemodynamic Measurements Pulmonary Artery Catheterization Control of Blood Pressure Heart Failure Cardiac
More informationMaternal and Fetal Physiology
Background Maternal and Fetal Physiology Anderson Lo, DO Fellow, Maternal-Fetal Medicine Wayne State University School of Medicine SEMCME Fetal Assessment Course July 20, 2018 Oxygen pathway Mother Placenta
More informationFUNDAMENTALS OF HEMODYNAMICS, VASOACTIVE DRUGS AND IABP IN THE FAILING HEART
FUNDAMENTALS OF HEMODYNAMICS, VASOACTIVE DRUGS AND IABP IN THE FAILING HEART CINDY BITHER, MSN, ANP, ANP, AACC, CHFN CHIEF NP, ADV HF PROGRAM MEDSTAR WASHINGTON HOSPITAL CENTER CONFLICTS OF INTEREST NONE
More informationHOW LOW CAN YOU GO? HYPOTENSION AND THE ANESTHETIZED PATIENT.
HOW LOW CAN YOU GO? HYPOTENSION AND THE ANESTHETIZED PATIENT. Donna M. Sisak, CVT, LVT, VTS (Anesthesia/Analgesia) Seattle Veterinary Specialists Kirkland, WA dsisak@svsvet.com THE ANESTHETIZED PATIENT
More informationSESSION D5. The Heart of the Matter: Cardiac Disease in Pregnancy Brad M. Dolinsky, MD, MFM
37th Annual Advanced Practice in Primary and Acute Care Conference: October 9-11, 2014 2:45 SESSION D5 Session Description: The Heart of the Matter: Cardiac Disease in Pregnancy Brad M. Dolinsky, MD, MFM
More informationGuideline for Management of Severe or Fulminating Pre-Eclampsia
Guideline for Management of Severe or Fulminating Pre-Eclampsia Originator: Labour Ward Forum, Maternity Services Date Approved: September 2011 Approved by: W&CH Quality & Safety Group Reviewed and ratified
More informationMagnesium Sulfate Prophylaxis in Preeclampsia: Evidence From Randomized Trials
CLINICAL OBSTETRICS AND GYNECOLOGY Volume 48, Number 2, 478 488 Ó 2005, Lippincott Williams & Wilkins Magnesium Sulfate Prophylaxis in Preeclampsia: Evidence From Randomized Trials BAHA M. SIBAI, MD Department
More informationRETIRED: REVIEWED/Revised: 12/14; 10/15; 1/16; 9/16, 10/16, 5/17, 5/18, 9/18
PAGE: 1 of 9 Scope Louisiana Healthcare Connections (LHCC) Medical Department Purpose To provide medical necessity criteria for obstetrical Home Health programs offered by vendors such as Optum Obstetrical
More informationPostpartum hypertension, preeclampsia and eclampsia. Arun Jeyabalan, MD MS University of Pittsburgh
Postpartum hypertension, preeclampsia and eclampsia Arun Jeyabalan, MD MS University of Pittsburgh Confusing concept Preeclampsia only occurs in pregnancy placenta is required Delivery cures preeclampsia
More informationHypertensives Emergency and Urgency
Hypertensives Emergency and Urgency Budi Yuli Setianto Cardiology Divisision Department of Internal Medicine Faculty of Medicine UGM Sardjito Hospital Yogyakarta Background USA: Hypertension is 30% of
More informationThe Role of the Anaesthesiologist in the Perioperative Management of Preeclampsia. RA Dyer Interlaken 2017
The Role of the Anaesthesiologist in the Perioperative Management of Preeclampsia RA Dyer Interlaken 2017 6 In preeclampsia - Understanding of pathophysiology Assessment of disease severity Prediction
More informationHemodynamic Monitoring and Circulatory Assist Devices
Hemodynamic Monitoring and Circulatory Assist Devices Speaker: Jana Ogden Learning Unit 2: Hemodynamic Monitoring and Circulatory Assist Devices Hemodynamic monitoring refers to the measurement of pressure,
More informationShock is defined as a state of cellular and tissue hypoxia due to : reduced oxygen delivery and/or increased oxygen consumption or inadequate oxygen
Shock is defined as a state of cellular and tissue hypoxia due to : reduced oxygen delivery and/or increased oxygen consumption or inadequate oxygen utilization The effects of shock are initially reversible
More informationImages in Cardiovascular Medicine
Images in Cardiovascular Medicine Management of Severe Mitral Stenosis During Pregnancy Rebecca S. Norrad, MBBS; Omid Salehian, MSc, MD, FRCPC, FACC, FAHA A 37-year-old woman originally from Iraq was referred
More informationEQUIPMENT: Nitrous Oxygen Delivery System:
Policy: Nitrous Oxide Use in the Intrapartum and Immediate Postpartum Period for Obstetrical Patients in the Family Birth Place Approvers: CEO. CNO, Medical Staff President, Anesthesia Chair, OB Medical
More informationCONTROL OF BLOOD PRESSURE IN PREGNANCY: HOW HIGH IS TOO HIGH? EVELYNE REY, CHU Ste-Justine, Montreal
CONTROL OF BLOOD PRESSURE IN PREGNANCY: HOW HIGH IS TOO HIGH? EVELYNE REY, CHU Ste-Justine, Montreal CONFLICTS OF INTEREST $: None Others: Canadian guidelines, CHIPS CSIM2015 2 LEARNING OBJECTIVES New
More informationMANAGEMENT OF HYPERTENSION IN PREGNANCY, THE ALGORHITHM
MANAGEMENT OF HYPERTENSION IN PREGNANCY, THE ALGORHITHM Are Particular Anti-hypertensives More Effective or Harmful Than Others in Hypertension in Pregnancy? Existing data is inadequate Methyldopa and
More informationACUTE RESPIRATORY DISTRESS SYNDROME (ARDS) Rv
ACUTE RESPIRATORY DISTRESS SYNDROME (ARDS) Rv.8.18.18 ACUTE RESPIRATORY DISTRESS SYNDROME (ARDS) SUDDEN PROGRESSIVE FORM OF ACUTE RESPIRATORY FAILURE ALVEOLAR CAPILLARY MEMBRANE BECOMES DAMAGED AND MORE
More informationIntroduction. Invasive Hemodynamic Monitoring. Determinants of Cardiovascular Function. Cardiovascular System. Hemodynamic Monitoring
Introduction Invasive Hemodynamic Monitoring Audis Bethea, Pharm.D. Assistant Professor Therapeutics IV January 21, 2004 Hemodynamic monitoring is necessary to assess and manage shock Information obtained
More informationMagnesium Sulphate - Management of Hypertensive Disorders of Pregnancy
1. Purpose Magnesium sulphate is the anticonvulsant of choice for pre-eclampsia prophylaxis and treatment. This clinical guideline outlines the indications, contraindications, administration and monitoring
More informationFLUIDS AND SOLUTIONS IN THE CRITICALLY ILL. Daniel De Backer Department of Intensive Care Erasme University Hospital Brussels, Belgium
FLUIDS AND SOLUTIONS IN THE CRITICALLY ILL Daniel De Backer Department of Intensive Care Erasme University Hospital Brussels, Belgium Why do we want to administer fluids? To correct hypovolemia? To increase
More informationWhat would be the response of the sympathetic system to this patient s decrease in arterial pressure?
CASE 51 A 62-year-old man undergoes surgery to correct a herniated disc in his spine. The patient is thought to have an uncomplicated surgery until he complains of extreme abdominal distention and pain
More informationIndex. Note: Page numbers of article titles are in boldface type.
Index Note: Page numbers of article titles are in boldface type. A Acid base balance pregnancy-related changes to, 640 Acquired heart disease, 731 Acute fatty liver of pregnancy (AFLP), 618 Acute kidney
More informationHypertensive Disorders of Pregnancy Guideline
Hypertensive Disorders of Pregnancy Guideline Approved by: Senior Director of Operations, Women s & Child Health, GNCH/MCH Facility Chief, Obstetrics/Gynecology, GNCH Facility Chief, Obstetrics/Gynecology,
More informationState of Florida Hypothermia Protocol. Michael D. Weiss, M.D. Associate Professor of Pediatrics Division of Neonatology
State of Florida Hypothermia Protocol Michael D. Weiss, M.D. Associate Professor of Pediatrics Division of Neonatology I. Entry Criteria 1. Gestational Age greater than or equal to 35 weeks gestation
More informationCounseling and Long-term Follow up After Gestational Disorders
Counseling and Long-term Follow up After Gestational Disorders Tanya Melnik, MD Assistant Professor, University of Minnesota Sarina Martini, MD Ob/Gyn Resident, PGY4 University of Minnesota Counseling
More informationRelax and Learn At the Farm 2012
Relax and Learn At the Farm Session 9: Invasive Hemodynamic Assessment and What to Do with the Data Carol Jacobson RN, MN Cardiovascular Nursing Education Associates Function of CV system is to deliver
More informationHow to maintain optimal perfusion during Cardiopulmonary By-pass. Herdono Poernomo, MD
How to maintain optimal perfusion during Cardiopulmonary By-pass Herdono Poernomo, MD Cardiopulmonary By-pass Target Physiologic condition as a healthy person Everything is in Normal Limit How to maintain
More informationDepartment of Intensive Care Medicine UNDERSTANDING CIRCULATORY FAILURE IN SEPSIS
Department of Intensive Care Medicine UNDERSTANDING CIRCULATORY FAILURE IN SEPSIS UNDERSTANDING CIRCULATORY FAILURE IN SEPSIS a mismatch between tissue perfusion and metabolic demands the heart, the vasculature
More informationMaternal Cardiac Disease In Pregnancy. August 25, 2017 PREGNANCY ECHO CONFERENCE
Maternal Cardiac Disease In Pregnancy August 25, 2017 PREGNANCY ECHO CONFERENCE Maternal Physiology Cardiac Output = HR x SV Non-pregnant: 4.5 L/min Pregnant: 6.0 L/min Increase most acute in first 10
More informationMore History. Organization. Maternal Cardiac Disease: a historical perspective. The Parturient with Cardiac Disease 9/21/2012
The Parturient with Cardiac Disease Pamela Flood M.D. Professor of Anesthesia and Perioperative Care Obstetrics, Gynecology and Reproductive Sciences University of California, San Francisco Maternal Cardiac
More informationSEVERE PRE-ECLAMPSIA
DEFINITIONS SEVERE PRE-ECLAMPSIA Pre-eclampsia: pregnancy induced hypertension with significant proteinuria +/- oedema affecting virtually any organ system in the body Severe pre-eclampsia: Diastolic blood
More informationCardiac Output MCQ. Professor of Cardiovascular Physiology. Cairo University 2007
Cardiac Output MCQ Abdel Moniem Ibrahim Ahmed, MD Professor of Cardiovascular Physiology Cairo University 2007 90- Guided by Ohm's law when : a- Cardiac output = 5.6 L/min. b- Systolic and diastolic BP
More informationHAEMODYNAMIC MONITORING in SEVERE PREECLAMPSIA. Eldrid Langesæter MD PhD Consultant Anaesthesiologist Oslo University Hospital Rikshospitalet
HAEMODYNAMIC MONITORING in SEVERE PREECLAMPSIA Eldrid Langesæter MD PhD Consultant Anaesthesiologist Oslo University Hospital Rikshospitalet Stockholm May 2011 Rikshospitalet Haemodynamics in severe preeclamspia
More informationEVALUATION OF PREGNANT PATIENTS WITH HEART DISEASE. Karen Stout, MD University of Washington Seattle Children s Seattle, WA
EVALUATION OF PREGNANT PATIENTS WITH HEART DISEASE Karen Stout, MD University of Washington Seattle Children s Seattle, WA CASE PRESENTATION 24 year old woman with aortic regurgitation referred for evaluation
More informationPregnancy and Heart Disease Sharon L. Roble, MD Echo Hawaii 2016
1 Pregnancy and Heart Disease Sharon L. Roble, MD Echo Hawaii 2016 DISCLOSURES I have no disclosures relevant to today s talk 2 Cardiovascular Effects of Pregnancy Anatomic Ventricular muscle mass increases
More informationPlasma colloid osmotic pressure, Briones index and ascites in preeclampsia-eclampsia
Cir Cir 2010;78:133-139 Plasma colloid osmotic pressure, Briones index and ascites in preeclampsia-eclampsia Juan Gustavo Vázquez-Rodríguez Abstract Background: Capillary leak in preeclampsia-eclampsia
More informationNeonatal Fluid Therapy Not my mother s physiology!!
Neonatal Fluid Therapy Not my mother s physiology!! Physiologic Approach to Neonatal Fluid Therapy General principles of fluid balance Fetal physiology of fluid balance Neonatal physiology of fluid balance
More informationA Challenging Case: Von Willebrand Disease and Pulmonary Hypertension in Pregnancy
A Challenging Case: Von Willebrand Disease and Pulmonary Hypertension in Pregnancy Diana S. Wolfe, MD, MPH Assistant Professor Department of Obstetrics & Gynecology and Women s Health Associate Fellowship
More informationIV Fluids. I.V. Fluid Osmolarity Composition 0.9% NaCL (Normal Saline Solution, NSS) Uses/Clinical Considerations
IV Fluids When administering IV fluids, the type and amount of fluid may influence patient outcomes. Make sure to understand the differences between fluid products and their effects. Crystalloids Crystalloid
More information39 th Annual Perinatal Conference Vanderbilt University December 6, 2013 IUGR. Diagnosis and Management
39 th Annual Perinatal Conference Vanderbilt University December 6, 2013 IUGR Diagnosis and Management Giancarlo Mari, M.D., M.B.A. Professor and Chair Department of Obstetrics and Gynecology University
More informationFAILURE IN PATIENTS WITH MYOCARDIAL INFARCTION
Br. J. clin. Pharmac. (1982), 14, 187S-19lS BENEFICIAL EFFECTS OF CAPTOPRIL IN LEFT VENTRICULAR FAILURE IN PATIENTS WITH MYOCARDIAL INFARCTION J.P. BOUNHOURE, J.G. KAYANAKIS, J.M. FAUVEL & J. PUEL Departments
More informationHemodynamic Monitoring
Perform Procedure And Interpret Results Hemodynamic Monitoring Tracheal Tube Cuff Pressure Dean R. Hess PhD RRT FAARC Hemodynamic Monitoring Cardiac Rate and Rhythm Arterial Blood Pressure Central Venous
More informationCASE 5 - Toy et al. CASE FILES: Obstetrics & Gynecology
z CASE 5 - Toy et al. CASE FILES: Obstetrics & Gynecology A 28-year-old woman is brought into the emergency room with a blood pressure of 60/40. The patient s husband states that she had 2 days of nausea
More informationBased on 2014 SOGC Guidelines
Based on 2014 SOGC Guidelines 22nd Edition 2015 1 ICH + gestational hypertension by far the biggest cause of direct maternal deaths New stats coming in 2013 OCR 22nd Edition 2015 2 Diastolic 90 mmhg is
More informationThe Fetus: Five Top Do Not Miss Diagnoses. Doppler Ultrasound
The Fetus: Five Top Do Not Miss Diagnoses Doppler Ultrasound Giancarlo Mari, MD, MBA Professor and Chair Department of Obstetrics and Gynecology University of Tennessee Health Science Center Memphis, TN
More informationAlbumina nel paziente critico. Savona 18 aprile 2007
Albumina nel paziente critico Savona 18 aprile 2007 What Is Unique About Critical Care RCTs patients eligibility is primarily defined by location of care in the ICU rather than by the presence of a specific
More informationHow and why I give IV fluid Disclosures SCA Fluids and public health 4/1/15. Andrew Shaw MB FRCA FCCM FFICM
How and why I give IV fluid Andrew Shaw MB FRCA FCCM FFICM Professor and Chief Cardiothoracic Anesthesiology Vanderbilt University Medical Center 2015 Disclosures Consultant for Grifols manufacturer of
More informationTACO Est ce que cette complication transfusionnelle peut être prédite et prévenue?
TACO Est ce que cette complication transfusionnelle peut être prédite et prévenue? Jeannie Callum, BA, MD, FRCPC, CTBS En vertu des règles de divulgation, je suis tenu de vous Nothing dire que je suis
More informationThe New England Journal of Medicine
The New England Journal of Medicine Copyright 2001 by the Massachusetts Medical Society VOLUME 344 M AY 24, 2001 NUMBER 21 MATERNAL AND FETAL OUTCOMES OF SUBSEQUENT PREGNANCIES IN WOMEN WITH PERIPARTUM
More informationMyocardial Infarction: Left Ventricular Failure
CARDIOVASCULAR PHYSIOLOGY 93 Case 17 Myocardial Infarction: Left Ventricular Failure Marvin Zimmerman is a 52-year-old construction manager who is significantly overweight. Despite his physician's repeated
More informationFluids in Sepsis: How much and what type? John Fowler, MD, FACEP Kent Hospital, İzmir Eisenhower Medical Center, USA American Hospital Dubai, UAE
Fluids in Sepsis: How much and what type? John Fowler, MD, FACEP Kent Hospital, İzmir Eisenhower Medical Center, USA American Hospital Dubai, UAE In critically ill patients: too little fluid Low preload,
More informationSWISS SOCIETY OF NEONATOLOGY. Prolonged arterial hypotension due to propofol used for endotracheal intubation in a newborn infant
SWISS SOCIETY OF NEONATOLOGY Prolonged arterial hypotension due to propofol used for endotracheal intubation in a newborn infant July 2001 2 Wagner B, Intensive Care Unit, University Children s Hospital
More informationReference ranges for tissue Doppler measures of maternal systolic and diastolic left ventricular function
Ultrasound Obstet Gynecol 2007; 29: 414 420 Published online 2 March 2007 in Wiley InterScience (www.interscience.wiley.com). DOI: 10.1002/uog.3966 Reference ranges for tissue Doppler measures of maternal
More informationA study of neonatal and maternal outcomes of asthma during pregnancy
International Journal of Research in Medical Sciences Meena BL et al. Int J Res Med Sci. 2013 Feb;1(1):23-27 www.msjonline.org pissn 2320-6071 eissn 2320-6012 Research Article DOI: 10.5455/2320-6012.ijrms20130206
More informationPREGESTATIONAL DIABETES (TYPE 1 AND 2)
PREGESTATIONAL DIABETES (TYPE 1 AND 2) Women with diabetes prior to pregnancy need to evaluate and optimize their baseline to assure the healthiest pregnancy possible.[1] The overall prevalence of pregnant
More informationMcHenry Western Lake County EMS System Paramedic, EMT-B and PHRN Optional Continuing Education 2018 #12 Understanding Preload and Afterload
McHenry Western Lake County EMS System Paramedic, EMT-B and PHRN Optional Continuing Education 2018 #12 Understanding Preload and Afterload Cardiac output (CO) represents the volume of blood that is delivered
More informationHEART FAILURE PHARMACOLOGY. University of Hawai i Hilo Pre- Nursing Program NURS 203 General Pharmacology Danita Narciso Pharm D
HEART FAILURE PHARMACOLOGY University of Hawai i Hilo Pre- Nursing Program NURS 203 General Pharmacology Danita Narciso Pharm D 1 LEARNING OBJECTIVES Understand the effects of heart failure in the body
More informationSHOCK. Emergency pediatric PICU division Pediatric Department Medical Faculty, University of Sumatera Utara H. Adam Malik Hospital
SHOCK Emergency pediatric PICU division Pediatric Department Medical Faculty, University of Sumatera Utara H. Adam Malik Hospital 1 Definition Shock is an acute, complex state of circulatory dysfunction
More informationCritical Care in Obstetrics: An Innovative and Integrated Model for Learning the Essentials
Critical Care in Obstetrics: An Innovative and Integrated Model for Learning the Essentials Respiratory Distress Syndrome and Pulmonary Edema Sonya S. Abdel-Razeq, MD Maternal-Fetal Medicine Surgical Critical
More informationCASE DISCUSSION. Dr JAYASREE VEERABOINA 2nd yr PG MS OBG
CASE DISCUSSION Dr JAYASREE VEERABOINA 2nd yr PG MS OBG Normal Cardiovascular changes in Pregnancy CARDIAC OUTPUT 5 th wk -- starts 12 wks -- 30-35% 30-32 wks -- 40% During labour -- 50% After delivery
More informationNothing to Disclose. Severe Pulmonary Hypertension
Severe Ronald Pearl, MD, PhD Professor and Chair Department of Anesthesiology Stanford University Rpearl@stanford.edu Nothing to Disclose 65 year old female Elective knee surgery NYHA Class 3 Aortic stenosis
More informationClinical Outcomes of Women with Peripartum Cardiomyopathy With and Without Preeclampsia: a Population-based Study
Clinical Outcomes of Women with Peripartum Cardiomyopathy With and Without Preeclampsia: a Population-based Study Isabelle Malhamé, MSc Candidate in Epidemiology, McGill University Obstetric Medicine Fellow,
More informationMedical Management of Acute Heart Failure
Critical Care Medicine and Trauma Medical Management of Acute Heart Failure Mary O. Gray, MD, FAHA Associate Professor of Medicine University of California, San Francisco Staff Cardiologist and Training
More informationHypothermia in Neonates with HIE TARA JENDZIO, DNP(C), RN, RNC-NIC
Hypothermia in Neonates with HIE TARA JENDZIO, DNP(C), RN, RNC-NIC Objectives 1. Define Hypoxic-Ischemic Encephalopathy (HIE) 2. Identify the criteria used to determine if an infant qualifies for therapeutic
More informationTOPIC : Cardiogenic Shock
University of Ferrara Department of Morphology, Surgery and Experimental Medicine. Section of Anaesthesia and Intensive Care Medicine TOPIC : Cardiogenic Shock What is shock? Shock is a condition of inadequate
More informationPeripartum kardiomyopati
Fall 3 Peripartum kardiomyopati Roman A roch 2017 01 26 2 3 Dg. PPCM Symptoms ECG, pro-bnp echocardiography 4 Peripartum cardiomyopathy: a systematic literature review Acta Obstetricia et Gynecologica
More informationCONTEMPORARY APPROACH PULMONARY HYPERTENSION IN PREGNANCY
CONTEMPORARY APPROACH PULMONARY HYPERTENSION IN PREGNANCY Dianne L. Zwicke, MD Clinical Adjunct Professor of Medicine University of Wisconsin School of Medicine and Public Health Medical Director, Pulmonary
More informationIs massive proteinuria associated with maternal and fetal morbidities in preeclampsia?
Original Article Obstet Gynecol Sci 2017;60(3):260-265 https://doi.org/10.5468/ogs.2017.60.3.260 pissn 2287-8572 eissn 2287-8580 Is massive proteinuria associated with maternal and fetal morbidities in
More informationPregnancy outcomes in women after an arterial switch operation for transposition of the great arteries
Pregnancy outcomes in women after an arterial switch operation for transposition of the great arteries Department of Obstetrics and Gynecology 1) Department of Pediatrics 2) National Cerebral and Cardiovascular
More informationACUTE KIDNEY INJURY FOCUS ON OBSTETRICS DONNA HIGGINS, CLINICAL NURSE EDUCATOR, NORTHERN LINCOLNSHIRE HOSPITALS NHS FOUNDATION TRUST
ACUTE KIDNEY INJURY FOCUS ON OBSTETRICS DONNA HIGGINS, CLINICAL NURSE EDUCATOR, NORTHERN LINCOLNSHIRE HOSPITALS NHS FOUNDATION TRUST AIMS & OBJECTIVES Review the functions of the kidney Identify renal
More informationIsolated proteinuria in Chinese pregnant women with pre-eclampsia: Results of retrospective observational study
Biomedical Research 2017; 28 (11): 5162-5166 ISSN 0970-938X www.biomedres.info Isolated proteinuria in Chinese pregnant women with pre-eclampsia: Results of retrospective observational study Jing Cai 1,
More informationA pregnant patient with a prosthetic valve Giacomo Boccuzzi, MD, FESC
A pregnant patient with a prosthetic valve Giacomo Boccuzzi, MD, FESC Department of Invasive Cardiology, Ospedale San Giovanni Bosco, Turin, Italy *C.V. was born the 24th May 1980 Rheumatic fever during
More informationHypovolemic Shock: Regulation of Blood Pressure
CARDIOVASCULAR PHYSIOLOGY 81 Case 15 Hypovolemic Shock: Regulation of Blood Pressure Mavis Byrne is a 78-year-old widow who was brought to the emergency room one evening by her sister. Early in the day,
More informationNurse Driven Fluid Optimization Using Dynamic Assessments
Nurse Driven Fluid Optimization Using Dynamic Assessments 2016 1 WHAT WE BELIEVE We believe that clinicians make vital fluid and drug decisions every day with limited and inconclusive information Cheetah
More information